Cognitive and hippocampal effects of adult male mice exposure to environmentally relevant doses of phthalates.

[1]  S. Radovick,et al.  Deletion of neural estrogen receptor alpha induces sex differential effects on reproductive behavior in mice , 2022, Communications Biology.

[2]  Qu-nan Wang,et al.  Maternal exposure to bis(2-ethylhexyl) phthalate during the thyroid hormone-dependent stage induces persistent emotional and cognitive impairment in middle-aged offspring mice. , 2022, Food and chemical toxicology : an international journal published for the British Industrial Biological Research Association.

[3]  Xiaomei Wu,et al.  Association between mixed exposure of phthalates and cognitive function among the U.S. elderly from NHANES 2011-2014: Three statistical models. , 2022, The Science of the total environment.

[4]  S. Mhaouty-Kodja,et al.  Effects and underlying cellular pathway involved in the impairment of the neurovascular unit following exposure of adult male mice to low doses of di(2-ethylhexyl) phthalate alone or in an environmental phthalate mixture. , 2021, Environmental research.

[5]  D. Sabatini,et al.  Limited survival and impaired hepatic fasting metabolism in mice with constitutive Rag GTPase signaling , 2021, Nature communications.

[6]  M. Ghasemi-Kasman,et al.  Prenatal and breastfeeding exposure to low dose of diethylhexyl phthalate induces behavioral deficits and exacerbates oxidative stress in rat hippocampus. , 2021, Food and chemical toxicology : an international journal published for the British Industrial Biological Research Association.

[7]  S. Mhaouty-Kodja,et al.  Disruption of the blood-brain barrier and its close environment following adult exposure to low doses of di(2-ethylhexyl)phthalate alone or in an environmental phthalate mixture in male mice. , 2021, Chemosphere.

[8]  Xu Yang,et al.  Continuous artificial light at night exacerbates diisononyl phthalate-induced learning and memory impairment in mice: Toxicological evidence. , 2021, Food and chemical toxicology : an international journal published for the British Industrial Biological Research Association.

[9]  F. Villalta,et al.  Activation of proline biosynthesis is critical to maintain glutamate homeostasis during acute methamphetamine exposure , 2021, Scientific reports.

[10]  S. Mhaouty-Kodja,et al.  Exposure of Adult Female Mice to Low Doses of di(2-ethylhexyl) Phthalate Alone or in an Environmental Phthalate Mixture: Evaluation of Reproductive Behavior and Underlying Neural Mechanisms , 2021, Environmental health perspectives.

[11]  R. Campbell,et al.  The Role of Amino Acids in Neurotransmission and Fluorescent Tools for Their Detection , 2020, International journal of molecular sciences.

[12]  Carlos Dombret,et al.  Effects of neural estrogen receptor beta deletion on social and mood-related behaviors and underlying mechanisms in male mice , 2020, Scientific Reports.

[13]  Paige L. Williams,et al.  Association of urinary concentrations of phthalate metabolites with quinolinic acid among women: A potential link to neurological disorders. , 2020, Environment international.

[14]  N. Montazeri-Najafabady,et al.  Anti-androgenic effect of 6-formylindolo[3,2-b]carbazole (FICZ) in LNCaP cells is mediated by the aryl hydrocarbon-androgen receptors cross-talk , 2020, Steroids.

[15]  José Manuel Barat Baviera,et al.  Update of the risk assessment of di‐butylphthalate (DBP), butyl‐benzyl‐phthalate (BBP), bis(2‐ethylhexyl)phthalate (DEHP), di‐isononylphthalate (DINP) and di‐isodecylphthalate (DIDP) for use in food contact materials , 2019, EFSA journal. European Food Safety Authority.

[16]  John F. Wesseling,et al.  Elevated synaptic vesicle release probability in synaptophysin/gyrin family quadruple knockouts , 2019, eLife.

[17]  B. Coull,et al.  High phthalate exposure increased urinary concentrations of quinolinic acid, implicated in the pathogenesis of neurological disorders: Is this a potential missing link? , 2019, Environmental research.

[18]  Ø. Øverli,et al.  Tryptophan Metabolic Pathways and Brain Serotonergic Activity: A Comparative Review , 2019, Front. Endocrinol..

[19]  M. Holahan,et al.  Preadolescent Phthalate (DEHP) Exposure Is Associated With Elevated Locomotor Activity and Reward-Related Behavior and a Reduced Number of Tyrosine Hydroxylase Positive Neurons in Post-Adolescent Male and Female Rats , 2018, Toxicological sciences : an official journal of the Society of Toxicology.

[20]  Maryam Ghotbaddini,et al.  Constitutive Aryl Hydrocarbon Receptor Signaling in Prostate Cancer Progression , 2018, Journal of cancer treatment & diagnosis.

[21]  J. Flaws,et al.  Prenatal Exposure to DEHP Induces Neuronal Degeneration and Neurobehavioral Abnormalities in Adult Male Mice , 2018, Toxicological sciences : an official journal of the Society of Toxicology.

[22]  Daniel G Kougias,et al.  Perinatal Exposure to an Environmentally Relevant Mixture of Phthalates Results in a Lower Number of Neurons and Synapses in the Medial Prefrontal Cortex and Decreased Cognitive Flexibility in Adult Male and Female Rats , 2018, The Journal of Neuroscience.

[23]  C. Ríos,et al.  Relevance of Alternative Routes of Kynurenic Acid Production in the Brain , 2018, Oxidative medicine and cellular longevity.

[24]  Neha Singh,et al.  Structure based mimicking of Phthalic acid esters (PAEs) and inhibition of hACMSD, an important enzyme of the tryptophan kynurenine metabolism pathway. , 2018, International journal of biological macromolecules.

[25]  Daphné Capela,et al.  Adult male mice exposure to nonylphenol alters courtship vocalizations and mating , 2018, Scientific Reports.

[26]  S. Mhaouty-Kodja,et al.  Revisiting medial preoptic area plasticity induced in male mice by sexual experience , 2017, Scientific Reports.

[27]  Daphné Capela,et al.  Neural Mechanisms Underlying the Disruption of Male Courtship Behavior by Adult Exposure to Di(2-ethylhexyl) Phthalate in Mice , 2017, Environmental health perspectives.

[28]  Aiqing Li,et al.  Di-n-butyl phthalate exposure negatively influences structural and functional neuroplasticity via Rho-GTPase signaling pathways. , 2017, Food and chemical toxicology : an international journal published for the British Industrial Biological Research Association.

[29]  R. Schwarcz,et al.  The kynurenine pathway and the brain: Challenges, controversies and promises , 2017, Neuropharmacology.

[30]  F. Kobarfard,et al.  Determination of dibutyl phthalate neurobehavioral toxicity in mice. , 2016, Food and chemical toxicology : an international journal published for the British Industrial Biological Research Association.

[31]  J. Billard,et al.  Neural Androgen Receptor Deletion Impairs the Temporal Processing of Objects and Hippocampal CA1-Dependent Mechanisms , 2016, PloS one.

[32]  Ling Peng Mice Brain Tissue Injury Induced by Diisononyl Phthalate Exposure and the Protective Application of Vitamin E , 2015, Journal of biochemical and molecular toxicology.

[33]  T. Dinan,et al.  Serotonin, tryptophan metabolism and the brain-gut-microbiome axis , 2015, Behavioural Brain Research.

[34]  Ni Zhang,et al.  Perinatal exposure to di‐(2‐ethylhexyl)‐phthalate leads to cognitive dysfunction and phospho‐tau level increase in aged rats , 2014, Environmental toxicology.

[35]  F. Tronche,et al.  Conditional Inactivation of Androgen Receptor Gene in the Nervous System: Effects on Male Behavioral and Neuroendocrine Responses , 2009, The Journal of Neuroscience.

[36]  M. Day,et al.  Activation of estrogen receptor-β regulates hippocampal synaptic plasticity and improves memory , 2008, Nature Neuroscience.

[37]  M. Fernstrom,et al.  Tyrosine, phenylalanine, and catecholamine synthesis and function in the brain. , 2007, The Journal of nutrition.

[38]  R. Sasaki,et al.  Effects of dietary di(2-ethylhexyl)phthalate, a putative endocrine disrupter, on enzyme activities involved in the metabolism of tryptophan to niacin in rats. , 2004, Biochimica et biophysica acta.

[39]  K. Shibata,et al.  Identification of a Toxic Mechanism of the Plasticizers, Phtahlic Acid Esters, which are Putative Endocrine Disrupters: Time-dependent Increase in Quinolinic Acid and Its Metabolites in… , 2002, Bioscience, biotechnology, and biochemistry.

[40]  K. Shibata,et al.  Elucidation of the Toxic Mechanism of the Plasticizers, Phthalic Acid Esters, Putative Endocrine Disrupters: Effects of Dietary Di(2-ethylhexyl)phthalate on the Metabolism of Tryptophan to… , 2002, Bioscience, biotechnology, and biochemistry.

[41]  M. Packard,et al.  Posttraining intrahippocampal estradiol injections enhance spatial memory in male rats: interaction with cholinergic systems. , 1996, Behavioral neuroscience.

[42]  KM Harris,et al.  Three-dimensional structure of dendritic spines and synapses in rat hippocampus (CA1) at postnatal day 15 and adult ages: implications for the maturation of synaptic physiology and long-term potentiation [published erratum appears in J Neurosci 1992 Aug;12(8):following table of contents] , 1992, The Journal of neuroscience : the official journal of the Society for Neuroscience.

[43]  L. Swanson,et al.  Distribution of androgen and estrogen receptor mRNA‐containing cells in the rat brain: An in situ hybridization study , 1990, The Journal of comparative neurology.