cGMP/Protein Kinase G Signaling Suppresses Inositol 1,4,5-Trisphosphate Receptor Phosphorylation and Promotes Endoplasmic Reticulum Stress in Photoreceptors of Cyclic Nucleotide-gated Channel-deficient Mice*
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Hongwei Ma | M. Biel | S. Michalakis | W. Baehr | Xi-Qin Ding | Fan Yang | Michael R. Butler | Arjun Thapa | Josh Belcher
[1] S. Beck,et al. Mutations in the unfolded protein response regulator ATF6 cause the cone dysfunction disorder achromatopsia , 2015, Nature Genetics.
[2] Cheng-Ying Wu,et al. Receptor interacting protein 140 attenuates endoplasmic reticulum stress in neurons and protects against cell death , 2014, Nature Communications.
[3] Hongwei Ma,et al. Loss of cone cyclic nucleotide-gated channel leads to alterations in light response modulating system and cellular stress response pathways: a gene expression profiling study. , 2013, Human molecular genetics.
[4] A. Dizhoor,et al. cGMP Accumulation Causes Photoreceptor Degeneration in CNG Channel Deficiency: Evidence of cGMP Cytotoxicity Independently of Enhanced CNG Channel Function , 2013, The Journal of Neuroscience.
[5] P. Pinton,et al. Selective modulation of subtype III IP3R by Akt regulates ER Ca2+ release and apoptosis , 2012, Cell Death and Disease.
[6] Hongwei Ma,et al. Endoplasmic Reticulum Stress-associated Cone Photoreceptor Degeneration in Cyclic Nucleotide-gated Channel Deficiency* , 2012, The Journal of Biological Chemistry.
[7] Yun-Ru Chen,et al. Ca2+ store depletion and endoplasmic reticulum stress are involved in P2X7 receptor‐mediated neurotoxicity in differentiated NG108‐15 cells , 2012, Journal of cellular biochemistry.
[8] A. Dubra,et al. Photoreceptor structure and function in patients with congenital achromatopsia. , 2011, Investigative ophthalmology & visual science.
[9] M. Aghaei,et al. Cyclic GMP induced apoptosis via protein kinase G in oestrogen receptor‐positive and ‐negative breast cancer cell lines , 2011, The FEBS journal.
[10] S. Tsang,et al. shRNA knockdown of guanylate cyclase 2e or cyclic nucleotide gated channel alpha 1 increases photoreceptor survival in a cGMP phosphodiesterase mouse model of retinitis pigmentosa , 2011, Journal of cellular and molecular medicine.
[11] D. Mekahli,et al. Endoplasmic-reticulum calcium depletion and disease. , 2011, Cold Spring Harbor perspectives in biology.
[12] Jianhua Xu,et al. Early-onset, slow progression of cone photoreceptor dysfunction and degeneration in CNG channel subunit CNGB3 deficiency. , 2011, Investigative ophthalmology & visual science.
[13] Livia S. Carvalho,et al. Long-term and age-dependent restoration of visual function in a mouse model of CNGB3-associated achromatopsia following gene therapy , 2011, Human molecular genetics.
[14] N. Tanimoto,et al. A key role for cyclic nucleotide gated (CNG) channels in cGMP-related retinitis pigmentosa. , 2011, Human molecular genetics.
[15] A. Eskin,et al. PKG-mediated MAPK signaling is necessary for long-term operant memory in Aplysia. , 2011, Learning & memory.
[16] C. Klaver,et al. Progressive loss of cones in achromatopsia: an imaging study using spectral-domain optical coherence tomography. , 2010, Investigative ophthalmology & visual science.
[17] N. Tanimoto,et al. Restoration of cone vision in the CNGA3-/- mouse model of congenital complete lack of cone photoreceptor function. , 2010, Molecular therapy : the journal of the American Society of Gene Therapy.
[18] Jianhua Xu,et al. The disease-causing mutations in the carboxyl terminus of the cone cyclic nucleotide-gated channel CNGA3 subunit alter the local secondary structure and interfere with the channel active conformational change. , 2010, Biochemistry.
[19] R. Barlow,et al. Impaired cone function and cone degeneration resulting from CNGB3 deficiency: down-regulation of CNGA3 biosynthesis as a potential mechanism. , 2009, Human molecular genetics.
[20] M. Ueffing,et al. PKG activity causes photoreceptor cell death in two retinitis pigmentosa models , 2009, Journal of neurochemistry.
[21] James D. Johnson,et al. Roles of IP3R and RyR Ca2+ Channels in Endoplasmic Reticulum Stress and β-Cell Death , 2009, Diabetes.
[22] John Calvin Reed,et al. Cell death and endoplasmic reticulum stress: disease relevance and therapeutic opportunities , 2008, Nature Reviews Drug Discovery.
[23] P. Barabas,et al. Depletion of calcium stores regulates calcium influx and signal transmission in rod photoreceptors , 2008, The Journal of physiology.
[24] M. Bootman,et al. Phosphorylation of inositol 1,4,5-trisphosphate receptors by protein kinase B/Akt inhibits Ca2+ release and apoptosis , 2008, Proceedings of the National Academy of Sciences.
[25] G. Guillemette,et al. Protein kinase C decreases the apparent affinity of the inositol 1,4,5-trisphosphate receptor type 3 in RINm5F cells. , 2007, Cell calcium.
[26] P. Sieving,et al. Constitutive Excitation by Gly90Asp Rhodopsin Rescues Rods from Degeneration Caused by Elevated Production of cGMP in the Dark , 2007, The Journal of Neuroscience.
[27] G. Somfai,et al. Optical coherence tomography of the macula in congenital achromatopsia. , 2007, Investigative ophthalmology & visual science.
[28] G. Guillemette,et al. Protein kinase C phosphorylates the inositol 1,4,5-trisphosphate receptor type 2 and decreases the mobilization of Ca2+in pancreatoma AR4-2J cells. , 2007, The Journal of endocrinology.
[29] Roberta Tammaro,et al. Apoptosis in retinal degeneration involves cross-talk between apoptosis-inducing factor (AIF) and caspase-12 and is blocked by calpain inhibitors , 2006, Proceedings of the National Academy of Sciences.
[30] Xiuying Huang,et al. Inositol 1,4,5-trisphosphate receptor type 1 phosphorylation and regulation by extracellular signal-regulated kinase. , 2006, Biochemical and biophysical research communications.
[31] Afshin Samali,et al. Mediators of endoplasmic reticulum stress‐induced apoptosis , 2006, EMBO reports.
[32] V. Marigo,et al. Cross-talk between two apoptotic pathways activated by endoplasmic reticulum stress: differential contribution of caspase-12 and AIF , 2006, Apoptosis.
[33] D. Yule,et al. Akt Kinase Phosphorylation of Inositol 1,4,5-Trisphosphate Receptors* , 2006, Journal of Biological Chemistry.
[34] R. Wojcikiewicz,et al. The type III inositol 1,4,5-trisphosphate receptor is phosphorylated by cAMP-dependent protein kinase at three sites. , 2005, The Biochemical journal.
[35] R. Bartenschlager,et al. Hepatitis C virus core triggers apoptosis in liver cells by inducing ER stress and ER calcium depletion , 2005, Oncogene.
[36] S. Haverkamp,et al. Impaired opsin targeting and cone photoreceptor migration in the retina of mice lacking the cyclic nucleotide-gated channel CNGA3. , 2005, Investigative ophthalmology & visual science.
[37] M. Sandberg,et al. Cone cGMP‐gated channel mutations and clinical findings in patients with achromatopsia, macular degeneration, and other hereditary cone diseases , 2005, Human mutation.
[38] D. Kass,et al. Chronic inhibition of cyclic GMP phosphodiesterase 5A prevents and reverses cardiac hypertrophy , 2005, Nature Medicine.
[39] F. Hofmann,et al. Reduced inflammatory hyperalgesia with preservation of acute thermal nociception in mice lacking cGMP-dependent protein kinase I. , 2004, Proceedings of the National Academy of Sciences of the United States of America.
[40] K. Murthy,et al. Selective phosphorylation of the IP3R-I in vivo by cGMP-dependent protein kinase in smooth muscle. , 2003, American journal of physiology. Gastrointestinal and liver physiology.
[41] U. Kaupp,et al. Cyclic nucleotide-gated ion channels. , 2002, Physiological reviews.
[42] Mineo Kondo,et al. Nrl is required for rod photoreceptor development , 2001, Nature Genetics.
[43] S. Jacobson,et al. CNGA3 mutations in hereditary cone photoreceptor disorders. , 2001, American journal of human genetics.
[44] A. Reichenbach,et al. Role of Muller cells in retinal degenerations. , 2001, Frontiers in bioscience : a journal and virtual library.
[45] Masataka Mori,et al. Nitric oxide-induced apoptosis in pancreatic β cells is mediated by the endoplasmic reticulum stress pathway , 2001, Proceedings of the National Academy of Sciences of the United States of America.
[46] P. Sieving,et al. Mutations in the CNGB3 gene encoding the beta-subunit of the cone photoreceptor cGMP-gated channel are responsible for achromatopsia (ACHM3) linked to chromosome 8q21. , 2000, Human molecular genetics.
[47] K. Yau,et al. Disruption of a Retinal Guanylyl Cyclase Gene Leads to Cone-Specific Dystrophy and Paradoxical Rod Behavior , 1999, The Journal of Neuroscience.
[48] M. Seeliger,et al. Selective loss of cone function in mice lacking the cyclic nucleotide-gated channel CNG3. , 1999, Proceedings of the National Academy of Sciences of the United States of America.
[49] V. Jensen,et al. Phosphorylation of the Inositol 1,4,5-Trisphosphate Receptor by Cyclic Nucleotide-dependent Kinases in Vitroand in Rat Cerebellar Slices in Situ * , 1999, The Journal of Biological Chemistry.
[50] S. Barnes,et al. Calcium-sensitive calcium influx in photoreceptor inner segments. , 1998, Journal of neurophysiology.
[51] K. Palczewski,et al. Turned on by Ca2+! The physiology and pathology of Ca2+-binding proteins in the retina , 1996, Trends in Neurosciences.
[52] L. Missiaen,et al. Characterization of a Cytosolic and a Luminal Ca2+ Binding Site in the Type I Inositol 1,4,5-Trisphosphate Receptor* , 1996, The Journal of Biological Chemistry.
[53] C. Remé,et al. Light damage in the rat retina: glial fibrillary acidic protein accumulates in Müller cells in correlation with photoreceptor damage. , 1996, Ophthalmic research.
[54] S. M. Goldin,et al. Calcium as a coagonist of inositol 1,4,5-trisphosphate-induced calcium release. , 1991, Science.
[55] 宮脇 敦史. Expressed Cerebellar-Type Inositol 1, 4, 5-Trisphosphate Receptor, P400 ; Has Calcium Release Activity in a Fibroblast L Cell Line , 1991 .
[56] Z. Yin,et al. Activation of Müller cells occurs during retinal degeneration in RCS rats. , 2010, Advances in experimental medicine and biology.