Staphylococcus aureus alpha-toxin-induced pores: Channel-like behavior in lipid bilayers and patch clamped cells
暂无分享,去创建一个
G. M. Alder | P. Usherwood | Y. Korchev | A. Bakhramov | E. Sviderskaya | C. L. Bashford | P. N. R. Usherwood | G. Alder | Y. E. Korchev | A. Bakhramov | B. S. Joomun | E. V. Sviderskaya | C. A. Pasternak | C. Pasternak | C. Bashford
[1] J. Graham,et al. Ion modulation of membrane permeability: Effect of cations on intact cells and on cells and phospholipid bilayers treated with pore-forming agents , 1988, The Journal of Membrane Biology.
[2] J. Dankert,et al. Recovery of human neutrophils from complement attack: removal of the membrane attack complex by endocytosis and exocytosis. , 1987, Journal of immunology.
[3] C. Pasternak,et al. Permeability changes resulting from virus-cell fusion: temperature-dependence of the contributing processes , 1985, Molecular and Cellular Biochemistry.
[4] O. Krasilnikov,et al. The structure of Staphylococcus aureus alpha-toxin-induced ionic channel. , 1988, General physiology and biophysics.
[5] Wilfred D. Stein,et al. Transport and Diffusion Across Cell Membranes , 1986 .
[6] J. Graham,et al. Tissue-culture cell fractionation. Fractionation of cellular membranes from 125I/lactoperoxidase-labelled Lettrée cells homogenized by bicarbonate-induced lysis: resolution of membranes by zonal centrifugation and in sucrose and metrizamide gradients. , 1979, The Biochemical journal.
[7] M. Thelestam. Membrane damage by staphylococcal alpha-toxin to different types of cultured mammalian cell. , 1983, Biochimica et biophysica acta.
[8] G. Menestrina,et al. Membrane damage by hemolytic viruses, toxins, complement, and other cytotoxic agents. A common mechanism blocked by divalent cations. , 1986, The Journal of biological chemistry.
[9] Ion transport through channels formed in lipid bilayers by Staphylococcus aureus alpha-toxin. , 1989 .
[10] T. Bolton,et al. Calcium‐activated potassium channels in single smooth muscle cells of rabbit jejunum and guinea‐pig mesenteric artery. , 1986, The Journal of physiology.
[11] S. Schein,et al. Colicin K acts by forming voltage-dependent channels in phospholipid bilayer membranes , 1978, Nature.
[12] R. Möllby,et al. Interaction of streptolysin O from Streptococcus pyogenes and theta-toxin from Clostridium perfringens with human fibroblasts , 1980, Infection and immunity.
[13] C. Pasternak,et al. Sequential onset of permeability changes in mouse ascites cells induced by Sendai virus. , 1985, Biochimica et biophysica acta.
[14] S. Bhakdi,et al. Damage to mammalian cells by proteins that form transmembrane pores. , 1987, Reviews of physiology, biochemistry and pharmacology.
[15] Gianfranco Menestrina,et al. Ionic channels formed byStaphylococcus aureus alpha-toxin: Voltage-dependent inhibition by divalent and trivalent cations , 2005, The Journal of Membrane Biology.
[16] G. Menestrina,et al. Pore-forming toxins: Experiments with S. aureus α-toxin, C. perfringens θ-toxin and E. coli haemolysin in lipid bilayers, liposomes and intact cells , 1990 .
[17] W. Seeger,et al. Staphylococcal alpha-toxin-induced PGI2 production in endothelial cells: role of calcium. , 1985, The American journal of physiology.
[18] C. Gitler,et al. Cytopathogenicity of Entamoeba histolytica. , 1984, Philosophical transactions of the Royal Society of London. Series B, Biological sciences.
[19] G. Menestrina,et al. Staphylococcal alpha-toxin increases the permeability of lipid vesicles by cholesterol- and pH-dependent assembly of oligomeric channels. , 1989, European journal of biochemistry.
[20] P. Usherwood,et al. Single channel kinetics of a glutamate receptor. , 1986, Biophysical journal.
[21] C. Granzow,et al. Sublines of the ehrlich-lettré mouse ascites tumour a new tool for experimental cell research , 1972, Naturwissenschaften.
[22] O. Krasilnikov,et al. A simple method for the determination of the pore radius of ion channels in planar lipid bilayer membranes. , 1992, FEMS microbiology immunology.
[23] G. Menestrina,et al. Pore-forming toxins: experiments with S. aureus alpha-toxin, C. perfringens theta-toxin and E. coli haemolysin in lipid bilayers, liposomes and intact cells. , 1990, Toxicon : official journal of the International Society on Toxinology.
[24] B. Drasar. Staphylococci and Staphylococcal Infections: Vols 1 and 2 , 1985 .
[25] C. Pasternak. Effect of pore formers on intracellular calcium. , 1986, Cell calcium.
[26] C. L. Bashford,et al. Plasma membrane potential of Lettré cells does not depend on cation gradients but on pumps , 2005, The Journal of Membrane Biology.
[27] M. Shin,et al. Elimination of complement channels from the plasma membranes of U937, a nucleated mammalian cell line: temperature dependence of the elimination rate. , 1983, Journal of immunology.
[28] R. Johnstone,et al. Influence of (DL)-propranolol and Ca2+ on membrane potential and amino acid transport in Ehrlich ascites tumor cells. , 1978, Biochimica et biophysica acta.
[29] G. Menestrina,et al. Mechanisms of attack and defence at the cell surface: The use of phospholipid bilayers as models for cell membrane , 1989, Bioscience reports.
[30] M. Montal. Formation of bimolecular membranes from lipid monolayers. , 1974, Methods in enzymology.
[31] J. Jeljaszewicz,et al. Bacterial toxins and cell membranes , 1978 .
[32] P. Henkart,et al. Cell damage by cytolysin. Spontaneous recovery and reversible inhibition by divalent cations. , 1988, Journal of immunology.
[33] Y. Korchev,et al. Membrane damage: common mechanisms of induction and prevention. , 1992, FEMS microbiology immunology.
[34] M. Thelestam,et al. Staphylococcal alpha toxin--recent advances. , 1988, Toxicon : official journal of the International Society on Toxinology.
[35] M Montal,et al. Formation of bimolecular membranes from lipid monolayers and a study of their electrical properties. , 1972, Proceedings of the National Academy of Sciences of the United States of America.
[36] S Bhakdi,et al. Alpha-toxin of Staphylococcus aureus. , 1991, Microbiological reviews.
[37] S. Bhakdi,et al. Quantitative analysis of the binding and oligomerization of staphylococcal alpha-toxin in target erythrocyte membranes , 1987, Infection and immunity.
[38] H. Bussey. K1 killer toxin, a pore‐forming protein from yeast , 1991, Molecular microbiology.
[39] H Schindler,et al. Formation of planar bilayers from artificial or native membrane vesicles , 1980, FEBS letters.
[40] M. Thelestam. Membrane damage by staphylococcal α-toxin to different types of cultured mammalian cell , 1983 .
[41] G. Alder,et al. Common action of certain viruses, toxins, and activated complement: pore formation and its prevention by extracellular Ca2+ , 1984, Bioscience reports.