The CC chemokine eotaxin/CCL11 has a selective profibrogenic effect on human lung fibroblasts.
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[1] D. Ribatti,et al. Human peripheral blood eosinophils induce angiogenesis. , 2005, The international journal of biochemistry & cell biology.
[2] M. Burdick,et al. The Role of the Th2 CC Chemokine Ligand CCL17 in Pulmonary Fibrosis1 , 2004, The Journal of Immunology.
[3] S. Orkin,et al. A Critical Role for Eosinophils in Allergic Airways Remodeling , 2004, Science.
[4] F. Levi-Schaffer,et al. Reduced eosinophil pro‐fibrogenic effect in severe childhood asthma compared to mild disease: An effect of corticosteroids? , 2004, Pediatric pulmonology.
[5] F. Martinez,et al. Enhanced monocyte chemoattractant protein-3/CC chemokine ligand-7 in usual interstitial pneumonia. , 2004, American journal of respiratory and critical care medicine.
[6] M. Keane. Chemokine profiling in pulmonary fibrosis: ready for prime time? , 2004, American journal of respiratory and critical care medicine.
[7] Christine M. Miller,et al. CCL11 (Eotaxin) Induces CCR3-Dependent Smooth Muscle Cell Migration , 2004, Arteriosclerosis, thrombosis, and vascular biology.
[8] Lauren Cohn,et al. Asthma: mechanisms of disease persistence and progression. , 2004, Annual review of immunology.
[9] S. Jimenez,et al. Pulmonary and activation-regulated chemokine stimulates collagen production in lung fibroblasts. , 2003, American journal of respiratory cell and molecular biology.
[10] D. Friend,et al. CC chemokine receptor 3 mobilizes to the surface of human mast cells and potentiates immunoglobulin E-dependent generation of interleukin 13. , 2003, American journal of respiratory cell and molecular biology.
[11] S. Holgate,et al. Airway remodeling in asthma: new insights. , 2003, The Journal of allergy and clinical immunology.
[12] Q. Hamid,et al. The CCR3 Receptor Is Involved in Eosinophil Differentiation and Is Up-Regulated by Th2 Cytokines in CD34+ Progenitor Cells1 , 2003, The Journal of Immunology.
[13] S. Wenzel,et al. TGF-β and IL-13 Synergistically Increase Eotaxin-1 Production in Human Airway Fibroblasts1 , 2002, The Journal of Immunology.
[14] Yoshiyuki Yamada,et al. The role of mitogen-activated protein kinases in eotaxin-induced cytokine production from bronchial epithelial cells. , 2002, American journal of respiratory cell and molecular biology.
[15] S. Kunkel,et al. Airway hyperresponsiveness, but not airway remodeling, is attenuated during chronic pulmonary allergic responses to Aspergillus in CCR4‐/‐ mice , 2002, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.
[16] K. Sekizawa,et al. Increases in collagen type I synthesis in asthma: the role of eosinophils and transforming growth factor‐bβ , 2002, Clinical and experimental allergy : journal of the British Society for Allergy and Clinical Immunology.
[17] B. Hinz,et al. Myofibroblasts and mechano-regulation of connective tissue remodelling , 2002, Nature Reviews Molecular Cell Biology.
[18] Z. Werb,et al. Decreased allergic lung inflammatory cell egression and increased susceptibility to asphyxiation in MMP2-deficiency , 2002, Nature Immunology.
[19] C. Hogaboam,et al. The role of CC chemokine receptor 5 (CCR5) and RANTES/CCL5 during chronic fungal asthma in mice1 , 2002, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.
[20] J. Ward,et al. Eotaxin (CCL11) Induces In Vivo Angiogenic Responses by Human CCR3+ Endothelial Cells1 , 2001, The Journal of Immunology.
[21] R. Breuer,et al. Eotaxin-3 but not eotaxin gene expression is upregulated in asthmatics 24 hours after allergen challenge. , 2001, American journal of respiratory cell and molecular biology.
[22] A. Kapp,et al. Characterization of the CC chemokine receptor 3 on human keratinocytes. , 2001, The Journal of investigative dermatology.
[23] Y. Kato,et al. Chemokines induce eosinophil degranulation through CCR-3. , 2000, The Journal of allergy and clinical immunology.
[24] T. Standiford,et al. Enhanced Pulmonary Allergic Responses to Aspergillus in CCR2−/− Mice1 , 2000, The Journal of Immunology.
[25] S. Kunkel,et al. Chemokines and their role in airway hyper-reactivity , 2000, Respiratory research.
[26] Toshiyuki Yamamoto,et al. Monocyte Chemoattractant Protein-1 Enhances Gene Expression and Synthesis of Matrix Metalloproteinase-1 in Human Fibroblasts by an Autocrine IL-1α Loop1 , 2000, The Journal of Immunology.
[27] S. Stafford,et al. Eotaxin induces degranulation and chemotaxis of eosinophils through the activation of ERK2 and p38 mitogen-activated protein kinases. , 2000, Blood.
[28] R. Homer,et al. Airway remodeling in asthma. , 1999, The Journal of clinical investigation.
[29] A. Nagler,et al. Human Eosinophils Regulate Human Lung- and Skin-Derived Fibroblast Properties in vitro: A Role for Transforming Growth Factor β (TGF-β) , 1999 .
[30] C. Mackay,et al. Enhanced expression of eotaxin and CCR3 mRNA and protein in atopic asthma. Association with airway hyperresponsiveness and predominant co‐localization of eotaxin mRNA to bronchial epithelial and endothelial cells , 1997, European journal of immunology.
[31] S. Holgate,et al. Release of RANTES, MIP-1 alpha, and MCP-1 into asthmatic airways following endobronchial allergen challenge. , 1997, American journal of respiratory and critical care medicine.
[32] P. Kuna,et al. Monocyte chemotactic and activating factor/monocyte chemoattractant protein in bronchoalveolar lavage fluid from patients with atopic asthma and chronic bronchitis. Relationship to lung function tests, bronchial hyper-responsiveness and cytology of bronchoalveolar lavage fluid. , 1997, Immunology letters.
[33] S. Phan,et al. Costimulation of Fibroblast Collagen and Transforming Growth Factor β1 Gene Expression by Monocyte Chemoattractant Protein-1 via Specific Receptors* , 1996, The Journal of Biological Chemistry.
[34] P. Leder,et al. Human eotaxin is a specific chemoattractant for eosinophil cells and provides a new mechanism to explain tissue eosinophilia , 1996, Nature Medicine.
[35] W Newman,et al. Cloning of the human eosinophil chemoattractant, eotaxin. Expression, receptor binding, and functional properties suggest a mechanism for the selective recruitment of eosinophils. , 1996, The Journal of clinical investigation.
[36] T. Williams,et al. Cooperation between interleukin-5 and the chemokine eotaxin to induce eosinophil accumulation in vivo , 1995, The Journal of experimental medicine.
[37] P. Leder,et al. Murine eotaxin: an eosinophil chemoattractant inducible in endothelial cells and in interleukin 4-induced tumor suppression. , 1995, Proceedings of the National Academy of Sciences of the United States of America.
[38] R. Reich,et al. Role of phospholipase D in laminin-induced production of gelatinase A (MMP-2) in metastatic cells , 1995, Clinical & Experimental Metastasis.
[39] S. Phan,et al. Lung monocyte chemoattractant protein-1 gene expression in bleomycin-induced pulmonary fibrosis. , 1994, Journal of immunology.
[40] T. Springer,et al. Monocyte chemoattractant protein 1 acts as a T-lymphocyte chemoattractant. , 1994, Proceedings of the National Academy of Sciences of the United States of America.
[41] J. Hsuan,et al. Eotaxin: a potent eosinophil chemoattractant cytokine detected in a guinea pig model of allergic airways inflammation , 1994, The Journal of experimental medicine.
[42] A. J. Valente,et al. Expression of monocyte chemoattractant protein 1 mRNA in human idiopathic pulmonary fibrosis. , 1992, Proceedings of the National Academy of Sciences of the United States of America.
[43] S. Hurd,et al. Investigative use of bronchoscopy, lavage and bronchial biopsies in asthma and other airways diseases. , 1992, The European respiratory journal.
[44] Y Iwamoto,et al. A rapid in vitro assay for quantitating the invasive potential of tumor cells. , 1987, Cancer research.
[45] F. Levi-Schaffer,et al. Mast cells and eosinophils: the hallmark of asthma. , 2004, Paediatric respiratory reviews.