Clinical efficacy and optimal dose of ST-630 (falecalcitriol) in secondary hyperparathyroidism in patients undergoing hemodialysis. Late phase II multicenter trial.
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S. Fujimi | Y. Kawaguchi | Masashi Suzuki | K. Kurokawa | H. Morii | S. Koshikawa | Yoshio Suzuki | Y. Ogura | N. Mimura | K. Maeda | Seishi Inoue | Y. Nishizawà
[1] 吉武 彬,et al. 活性型ビタミンD_3のフッ素誘導体ST-630の体内動態(第2報): ラットにおける^3H-ST-630の代謝 , 1996 .
[2] J. Russell,et al. Regulation by vitamin D metabolites of messenger ribonucleic acid for preproparathyroid hormone in isolated bovine parathyroid cells. , 1985, Proceedings of the National Academy of Sciences of the United States of America.
[3] H. Harter,et al. Marked suppression of secondary hyperparathyroidism by intravenous administration of 1,25-dihydroxy-cholecalciferol in uremic patients. , 1984, The Journal of clinical investigation.
[4] S. Ott,et al. Use of the deferoxamine infusion test in the diagnosis of aluminum-related osteodystrophy. , 1984, Annals of internal medicine.
[5] H. DeLuca,et al. 26,26,26,27,27,27-hexafluoro-1,25-dihydroxyvitamin D3: a highly potent, long-lasting analog of 1,25-dihydroxyvitamin D3. , 1984, Archives of biochemistry and biophysics.
[6] T. Inoue,et al. Abnormal calcium metabolism in hemodialyzed patients with diabetic nephropathy. , 1984, Nephron.