Hypernatremia secondary to post-stroke hypodipsia: just add water!

Disorders in water metabolism may occur in stroke patients. When hypernatremia arises in this setting, it is usually secondary to the development of central diabetes insipidus or it is the result of neurologic lesions that prevent patients from having free access to water. Much rarer are the cases of post-stroke hypernatremia caused by hypodipsia secondary to lesions of the thirst center. We report the case of a patient with severe hypernatremia, probably secondary to post-hemorrhagic stroke hypodipsia. The hypernatremia seen in this case was corrected by scheduling the patient’s water intake.

[1]  A. Johnson,et al.  The physiological regulation of thirst and fluid intake. , 2004, News in physiological sciences : an international journal of physiology produced jointly by the International Union of Physiological Sciences and the American Physiological Society.

[2]  B. Cha,et al.  Hypodipsic hypernatremia with intact AVP response to non-osmotic stimuli induced by hypothalamic tumor: a case report. , 2001, Journal of Korean medical science.

[3]  A. Cowley,et al.  Humoral control of water and electrolyte excretion during water restriction. , 1986, Kidney international.

[4]  G. Robertson,et al.  Abnormalities of thirst regulation. , 1984, Kidney international.

[5]  P. Miller,et al.  Hypodipsia in geriatric patients. , 1982, The American journal of medicine.

[6]  S. Spiro,et al.  Adipsia and hypothermia after subarachnoid hemorrhage. , 1971, British medical journal.

[7]  B. Brenner,et al.  Brenner & Rector's the Kidney , 1996 .

[8]  C. Ronco,et al.  Fluctuation of vasopressin secretion in chronic hypernatremia. , 1990, Nephron.