L3T4 (CD4+) cells that mediate contact sensitivity to trinitrochlorobenzene express I-A determinants.

[1]  S. Gautam,et al.  Oral administration of the contact sensitizer trinitrochlorobenzene: initial sensitization and subsequent appearance of a suppressor population. , 1990, Cellular immunology.

[2]  T. Mosmann,et al.  The role of IFN-gamma in delayed-type hypersensitivity mediated by Th1 clones. , 1989, Journal of immunology.

[3]  S. Gautam,et al.  Feeding trinitrochlorobenzene inhibits development of hapten-specific cytotoxic T lymphocytes by interfering with helper T-cell function. , 1989, Regional immunology.

[4]  J. M. Stoltz,et al.  The recruitment of lymphocytes into the skin by T cell lymphokines: the role of gamma-interferon. , 1988, Clinical and experimental immunology.

[5]  H. Kolb,et al.  IL-1 and IFN-gamma increase vascular permeability. , 1988, Immunology.

[6]  J. M. Stoltz,et al.  Lymphocyte recruitment in delayed-type hypersensitivity. The role of IFN-gamma. , 1988, Journal of immunology.

[7]  G. Asherson,et al.  Nonspecific T suppressor factor (nsTsF) cascade in contact sensitivity: nsTsF-1 causes an Ly-1+2- I-A+ immune T cell to produce a second, genetically restricted, nsTsF-2. , 1986, Journal of immunology.

[8]  G. Asherson,et al.  An overview of T-suppressor cell circuits. , 1986, Annual review of immunology.

[9]  B. Birshtein,et al.  Structural requirements of immunoglobulin G for binding to the Fc gamma receptors of the human tumor cell lines U937, HL-60, ML-1, and K562. , 1985, Journal of immunology.

[10]  D. Green,et al.  Characterization of two different Ly-1+ T cell populations that mediate delayed-type hypersensitivity. , 1984, Journal of immunology.

[11]  G. Asherson,et al.  T helper factor in contact sensitivity: antigen-specific I-A+ helper factor is made by an Lyt-1+2-, I-A+, I-J- T cell. , 1984, Immunology.

[12]  P. Askenase,et al.  Delayed-type hypersensitivity: activation of mast cells by antigen-specific T-cell factors initiates the cascade of cellular interactions. , 1983, Immunology today.

[13]  L. Butler,et al.  T cell responses induced by the parenteral injection of antigen-modified syngeneic cells. I. Induction, characterization, and regulation of antigen-specific T helper cells involved in delayed-type hypersensitivity responses. , 1983, Journal of immunology.

[14]  S. Gautam,et al.  Two distinct suppressor T cells acting in concert cause suppression of cytolytic T lymphocyte (CTL) response to hapten-altered self in vivo. , 1983, Journal of immunology.

[15]  G. Pantaleo,et al.  Direct demonstration of the clonogenic potential of every human peripheral blood T cell. Clonal analysis of HLA-DR expression and cytolytic activity , 1983, The Journal of experimental medicine.

[16]  B. Benacerraf,et al.  Hapten-specific T cell responses to 4-hydroxy-3-nitrophenyl acetyl. VIII. Suppressor cell pathways in cutaneous sensitivity responses , 1981, The Journal of experimental medicine.

[17]  G. Asherson,et al.  Differences in the ability of T cells to suppress the induction and expression of contact sensitivity. , 1981, Immunology.

[18]  B. Benacerraf,et al.  Hapten-specific T cell responses to 4-hydroxy-3-nitrophenyl acetyl. VI. Evidence for different T cell receptors in cells that mediate H-21- restricted and H-2D-restricted cutaneous sensitivity responses , 1980, The Journal of experimental medicine.

[19]  M. A. Bray,et al.  Leukotriene B: a potential mediator of inflammation , 1980, The Journal of pharmacy and pharmacology.

[20]  S. Miller,et al.  Suppressive Mechanisms Involving Sensitization and Tolerance in Contact Allergy , 1980, Immunological reviews.

[21]  G. Toews,et al.  Epidermal Langerhans cell density determines whether contact hypersensitivity or unresponsiveness follows skin painting with DNFB. , 1980, Journal of immunology.

[22]  M. Sy,et al.  Regulation of cell mediated immunity by antibodies: possible role of anti-receptor antibodies in the regulation of contact sensitivity to DNFB in mice. , 1979, Journal of immunology.

[23]  R. Winchester,et al.  Ia determinants on stimulated human T lymphocytes. Occurrence on mitogen- and antigen-activated T cells , 1979, The Journal of experimental medicine.

[24]  J. Moorhead.  Tolerance and contact sensitivity to DNFA in mice. VIII. Identification of distinct T cell subpopulations that mediate in vivo and in vitro manifestations of delayed hypersensitivity. , 1978, Journal of immunology.

[25]  J. Gamble,et al.  Ly and Ia antigen phenotypes of T cells involved in delayed-type hypersensitivity and in suppression , 1976, The Journal of experimental medicine.

[26]  O. Devinsky,et al.  Cell-mediated immunity: delayed-type hypersensitivity and cytotoxic responses are mediated by different T-cell subclasses , 1976, The Journal of experimental medicine.

[27]  W. Paul,et al.  T-lymphocyte-enriched murine peritoneal exudate cells. II. Genetic control of antigen-induced T-lymphocyte proliferation , 1976, The Journal of experimental medicine.

[28]  H. Claman,et al.  Tolerance and contact sensitivity to DNFB in mice. 3. Transfer of tolerance with "suppressor T cells". , 1974, Journal of immunology.

[29]  G. Asherson,et al.  The role of t cells in the passive transfer of contact sensitivity and their occurrence in the bone marrow , 1973, European journal of immunology.

[30]  G. Asherson,et al.  Contact sensitivity in the mouse. XII. The use of DNA synthesis in vivo to determine the anatomical location of immunological unresponsiveness to picryl chloride. , 1973, Immunology.

[31]  G. Asherson,et al.  Anatomical location of cells which mediate contact sensitivity in the lympho nodes and bone marrow. , 1973, Nature: New biology.

[32]  P. Nettesheim,et al.  Malignant Tumours of Liver and Lung in Rats fed Aminopyrine or Heptamethyleneimine together with Nitrite , 1973, Nature.

[33]  G. Asherson,et al.  The mechanism of immunological unresponsiveness to picryl chloride and the possible role of antibody mediated depression. , 1971, Clinical and experimental immunology.

[34]  G. Asherson,et al.  Contact and delayed hypersensitivity in the mouse. I. Active sensitization and passive transfer. , 1968, Immunology.

[35]  B. Bloom,et al.  Dual Immunological Unresponsiveness Induced by Cell Membrane Coupled Hapten or Antigen* , 1966, Nature.

[36]  K. Landsteiner,et al.  STUDIES ON THE SENSITIZATION OF ANIMALS WITH SIMPLE CHEMICAL COMPOUNDS , 1935, The Journal of experimental medicine.