Hyperoxic Reperfusion After Global Ischemia Decreases Hippocampal Energy Metabolism
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Gary Fiskum | Robert E. Rosenthal | R. Rosenthal | G. Fiskum | M. McKenna | Mary C. McKenna | Erica M. Richards | Irene Hopkins | I. Hopkins | E. M. Richards
[1] Ursula Sonnewald,et al. GABA alters the metabolic fate of [U‐13C]glutamate in cultured cortical astrocytes , 2005, Journal of neuroscience research.
[2] R. Rosenthal,et al. Normoxic ventilation after cardiac arrest reduces oxidation of brain lipids and improves neurological outcome. , 1998, Stroke.
[3] R. Rosenthal,et al. Postischemic inhibition of cerebral cortex pyruvate dehydrogenase. , 1994, Free radical biology & medicine.
[4] Geirmund Unsgård,et al. Differences in Neurotransmitter Synthesis and Intermediary Metabolism between Glutamatergic and GABAergic Neurons during 4 Hours of Middle Cerebral Artery Occlusion in the Rat: The Role of Astrocytes in Neuronal Survival , 2001, Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism.
[5] P. Hof,et al. Neuronal Subclass-Selective Loss of Pyruvate Dehydrogenase Immunoreactivity Following Canine Cardiac Arrest and Resuscitation , 2000, Experimental Neurology.
[6] S. Sakoda,et al. N-Acetylaspartate to Total Creatine Ratio in the Hippocampal CA1 Sector after Transient Cerebral Ischemia in Gerbils: Influence of Neuronal Elements, Reactive Gliosis, and Tissue Atrophy , 2003, Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism.
[7] S. Whitesall,et al. Cardiopulmonary-cerebral resuscitation with 100% oxygen exacerbates neurological dysfunction following nine minutes of normothermic cardiac arrest in dogs. , 1994, Resuscitation.
[8] M. McKenna,et al. Metabolic Compartmentation in Cortical Synaptosomes: Influence of Glucose and Preferential Incorporation of Endogenous Glutamate into GABA , 2002, Neurochemical Research.
[9] R. Rosenthal,et al. Prevention of Postischemic Canine Neurological Injury Through Potentiation of Brain Energy Metabolism by Acetyl‐L‐Carnitine , 1992, Stroke.
[10] N. Sims,et al. Ischemia in Rats , 2002 .
[11] R. Shulman,et al. In vivo13C NMR measurement of neurotransmitter glutamate cycling, anaplerosis and TCA cycle flux in rat brain during [2‐13C]glucose infusion , 2001, Journal of neurochemistry.
[12] P. Hof,et al. Normoxic Resuscitation after Cardiac Arrest Protects against Hippocampal Oxidative Stress, Metabolic Dysfunction, and Neuronal Death , 2005, Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism.
[13] R. Shank,et al. Pyruvate car☐ylase: an astrocyte-specific enzyme implicated in the replenishment of amino acid neurotransmitter pools , 1985, Brain Research.
[14] F. Plum,et al. Delayed hippocampal damage in humans following cardiorespiratory arrest , 1987, Neurology.
[15] J. Pascual,et al. Glutamate, glutamine, and GABA as substrates for the neuronal and glial compartments after focal cerebral ischemia in rats. , 1998, Stroke.
[16] R. Rosenthal,et al. Postischemic hyperoxia reduces hippocampal pyruvate dehydrogenase activity. , 2006, Free radical biology & medicine.
[17] A. Håberg,et al. Glutamate and GABA metabolism in transient and permanent middle cerebral artery occlusion in rat: Importance of astrocytes for neuronal survival , 2006, Neurochemistry International.
[18] T. Wieloch,et al. Pyruvate Dehydrogenase Activity in the Rat Cerebral Cortex following Cerebral Ischemia , 1989, Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism.
[19] B. Kristal,et al. Metabolic Impairment Induces Oxidative Stress, Compromises Inflammatory Responses, and Inactivates a Key Mitochondrial Enzyme in Microglia , 1999, Journal of neurochemistry.
[20] Fred Plum,et al. Temporal profile of neuronal damage in a model of transient forebrain ischemia , 1982, Annals of neurology.
[21] D. Kondziella,et al. Neuronal glial interaction in different neurological diseases studied by ex vivo 13C NMR spectroscopy , 2003, NMR in biomedicine.
[22] T. Sick,et al. Oxygen sensitivity of mitochondrial redox status and evoked potential recovery early during reperfusion in post-ischemic rat brain. , 1998, Resuscitation.
[23] S. Whitesall,et al. Cardiopulmonary-cerebral resuscitation with 100% oxygen exacerbates neurological dysfunction following nine minutes of normothermic cardiac arrest in dogs (Resuscitation (1994) 27 (159-170)) , 1994 .
[24] Y. Katayama,et al. The effect of duration of cerebral ischemia on brain pyruvate dehydrogenase activity, energy metabolites, and blood flow during reperfusion in gerbil brain , 1998, Brain Research.
[25] R G Shulman,et al. The Flux from Glucose to Glutamate in the Rat Brain in vivo as Determined by 1-Observed, 13C-Edited NMR Spectroscopy , 1990, Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism.
[26] Y. Katayama,et al. Effect of nicardipine, a Ca2+ channel blocker, on pyruvate dehydrogenase activity and energy metabolites during cerebral ischemia and reperfusion in gerbil brain , 1998, Brain Research.
[27] J. Krieglstein,et al. Pharmacology of cerebral ischemia : proceedings of the International Symposium on Pharmacology of Cerebral Ischemia, held in Marburg (FRG) on 16-17 July 1986 , 1986 .
[28] P. Hof,et al. Hyperbaric Oxygen Reduces Neuronal Death and Improves Neurological Outcome After Canine Cardiac Arrest , 2003, Stroke.