Temporal changes in bile acid levels and 12α-hydroxylation after Roux-en-Y gastric bypass surgery in type 2 diabetes

[1]  M. Hayden,et al.  Loss of Cyp8b1 Improves Glucose Homeostasis by Increasing GLP-1 , 2014, Diabetes.

[2]  M. Bellon,et al.  Rapid gastric and intestinal transit is a major determinant of changes in blood glucose, intestinal hormones, glucose absorption and postprandial symptoms after gastric bypass , 2014, Obesity.

[3]  Deepak L. Bhatt,et al.  Bariatric surgery versus intensive medical therapy for diabetes--3-year outcomes. , 2014, The New England journal of medicine.

[4]  C. le Roux,et al.  Urine Bile Acids Relate to Glucose Control in Patients with Type 2 Diabetes Mellitus and a Body Mass Index Below 30 kg/m2 , 2014, PloS one.

[5]  V. Tremaroli,et al.  FXR is a molecular target for the effects of vertical sleeve gastrectomy , 2014, Nature.

[6]  M. Patti,et al.  Fasting serum taurine-conjugated bile acids are elevated in type 2 diabetes and do not change with intensification of insulin. , 2014, The Journal of clinical endocrinology and metabolism.

[7]  C. Klaassen,et al.  Increased bile acids in enterohepatic circulation by short-term calorie restriction in male mice. , 2013, Toxicology and applied pharmacology.

[8]  C. Beglinger,et al.  Bile acids and gut peptide secretion after bariatric surgery: A 1‐year prospective randomized pilot trial , 2013, Obesity.

[9]  D. Accili,et al.  Human Insulin Resistance Is Associated With Increased Plasma Levels of 12α-Hydroxylated Bile Acids , 2013, Diabetes.

[10]  C. L. le Roux,et al.  Enhanced fasting and post-prandial plasma bile acid responses after Roux-en-Y gastric bypass surgery , 2013, Scandinavian journal of gastroenterology.

[11]  Jimmy D Bell,et al.  Obese patients after gastric bypass surgery have lower brain-hedonic responses to food than after gastric banding , 2013, Gut.

[12]  R. Sherwood,et al.  Higher circulating bile acid concentrations in obese patients with type 2 diabetes , 2013, Annals of clinical biochemistry.

[13]  J. Tap,et al.  Gut microbiota after gastric bypass in human obesity: increased richness and associations of bacterial genera with adipose tissue genes. , 2013, The American journal of clinical nutrition.

[14]  W. Strodel,et al.  A Role for Fibroblast Growth Factor 19 and Bile Acids in Diabetes Remission After Roux-en-Y Gastric Bypass , 2013, Diabetes Care.

[15]  P. Mattei,et al.  Systemic bile acid sensing by G protein‐coupled bile acid receptor 1 (GPBAR1) promotes PYY and GLP‐1 release , 2013, British journal of pharmacology.

[16]  M. Horowitz,et al.  Effects of rectal administration of taurocholic acid on glucagon‐like peptide‐1 and peptide YY secretion in healthy humans , 2013, Diabetes, obesity & metabolism.

[17]  L. Kaplan,et al.  Roux-en-Y gastric bypass normalizes the blunted postprandial bile acid excursion associated with obesity , 2013, International Journal of Obesity.

[18]  S. Madsbad,et al.  Fast pouch emptying, delayed small intestinal transit, and exaggerated gut hormone responses after Roux‐en‐Y gastric bypass , 2013, Neurogastroenterology and motility : the official journal of the European Gastrointestinal Motility Society.

[19]  Lee M. Kaplan,et al.  Conserved Shifts in the Gut Microbiota Due to Gastric Bypass Reduce Host Weight and Adiposity , 2013, Science Translational Medicine.

[20]  C. Cobelli,et al.  The Effect of a Bile Acid Sequestrant on Glucose Metabolism in Subjects With Type 2 Diabetes , 2013, Diabetes.

[21]  S. Klein,et al.  Weight loss induced by Roux-en-Y gastric bypass but not laparoscopic adjustable gastric banding increases circulating bile acids. , 2013, The Journal of clinical endocrinology and metabolism.

[22]  S. Woods,et al.  Fibroblast growth factor-19 action in the brain reduces food intake and body weight and improves glucose tolerance in male rats. , 2013, Endocrinology.

[23]  Barbara Gross,et al.  Bile acid receptors as targets for the treatment of dyslipidemia and cardiovascular disease , 2012, Journal of Lipid Research.

[24]  N. Nagelkerke,et al.  Rectal taurocholate increases L cell and insulin secretion, and decreases blood glucose and food intake in obese type 2 diabetic volunteers , 2012, Diabetologia.

[25]  J. H. Bekker,et al.  The role of bile after Roux-en-Y gastric bypass in promoting weight loss and improving glycaemic control. , 2012, Endocrinology.

[26]  M. Laakso,et al.  Conjugated Bile Acids Associate with Altered Rates of Glucose and Lipid Oxidation after Roux-en-Y Gastric Bypass , 2012, Obesity Surgery.

[27]  Yaniv Harel,et al.  Accelerated Gastric Emptying but No Carbohydrate Malabsorption 1 Year After Gastric Bypass Surgery (GBP) , 2012, Obesity Surgery.

[28]  D. Accili,et al.  Impaired generation of 12-hydroxylated bile acids links hepatic insulin signaling with dyslipidemia. , 2012, Cell metabolism.

[29]  S. Haneuse,et al.  A Multisite Study of Long-term Remission and Relapse of Type 2 Diabetes Mellitus Following Gastric Bypass , 2012, Obesity Surgery.

[30]  B. Angelin,et al.  Pronounced variation in bile acid synthesis in humans is related to gender, hypertriglyceridaemia and circulating levels of fibroblast growth factor 19 , 2011, Journal of internal medicine.

[31]  A. von Eckardstein,et al.  Bile Acid Metabolites in Serum: Intraindividual Variation and Associations with Coronary Heart Disease, Metabolic Syndrome and Diabetes Mellitus , 2011, PloS one.

[32]  M. Krempf,et al.  Fasting plasma chenodeoxycholic acid and cholic acid concentrations are inversely correlated with insulin sensitivity in adults , 2011, Nutrition & metabolism.

[33]  R. Straka,et al.  Profile of serum bile acids in non-cholestatic volunteers: gender-related differences in response to fenofibrate , 2011, Clinical pharmacology and therapeutics.

[34]  J. Stoker,et al.  Alterations of Hormonally Active Fibroblast Growth Factors after Roux-en-Y Gastric Bypass Surgery , 2011, Digestive Diseases.

[35]  B. Staels,et al.  Bile Acid Metabolism and the Pathogenesis of Type 2 Diabetes , 2011, Current diabetes reports.

[36]  R. Sherwood,et al.  The relationship between postprandial bile acid concentration, GLP‐1, PYY and ghrelin , 2011, Clinical endocrinology.

[37]  F. Kuipers,et al.  Improved glycemic control with colesevelam treatment in patients with type 2 diabetes is not directly associated with changes in bile acid metabolism , 2010, Hepatology.

[38]  J. Tap,et al.  Differential Adaptation of Human Gut Microbiota to Bariatric Surgery–Induced Weight Loss , 2010, Diabetes.

[39]  D. Pournaras,et al.  Postprandial plasma bile acid responses in normal weight and obese subjects , 2010, Annals of clinical biochemistry.

[40]  R. Goodlad,et al.  Gut Hypertrophy After Gastric Bypass Is Associated With Increased Glucagon-Like Peptide 2 and Intestinal Crypt Cell Proliferation , 2010, Annals of surgery.

[41]  J. Chiang,et al.  Bile acids: regulation of synthesis , 2009, Journal of Lipid Research.

[42]  J. Auwerx,et al.  TGR5-mediated bile acid sensing controls glucose homeostasis. , 2009, Cell metabolism.

[43]  J. Holst,et al.  Serum Bile Acids Are Higher in Humans With Prior Gastric Bypass: Potential Contribution to Improved Glucose and Lipid Metabolism , 2009, Obesity.

[44]  M. Crowell,et al.  Human gut microbiota in obesity and after gastric bypass , 2009, Proceedings of the National Academy of Sciences.

[45]  S. Strom,et al.  Bile acids activate fibroblast growth factor 19 signaling in human hepatocytes to inhibit cholesterol 7α‐hydroxylase gene expression , 2009, Hepatology.

[46]  A. M. Habib,et al.  Glucose Sensing in L Cells: A Primary Cell Study , 2008, Cell metabolism.

[47]  J. Auwerx,et al.  Anti-hyperglycemic activity of a TGR5 agonist isolated from Olea europaea. , 2007, Biochemical and biophysical research communications.

[48]  T. Lundåsen,et al.  Circulating intestinal fibroblast growth factor 19 has a pronounced diurnal variation and modulates hepatic bile acid synthesis in man , 2006, Journal of internal medicine.

[49]  Josep Vidal,et al.  Glucagon-like peptide-1, peptide YY, hunger, and satiety after gastric bypass surgery in morbidly obese subjects. , 2006, The Journal of clinical endocrinology and metabolism.

[50]  Folkert Kuipers,et al.  The Farnesoid X Receptor Modulates Adiposity and Peripheral Insulin Sensitivity in Mice* , 2006, Journal of Biological Chemistry.

[51]  Ke Ma,et al.  Farnesoid X receptor is essential for normal glucose homeostasis. , 2006, The Journal of clinical investigation.

[52]  Dae-Joong Kang,et al.  Bile salt biotransformations by human intestinal bacteria Published, JLR Papers in Press, November 18, 2005. , 2006, Journal of Lipid Research.

[53]  J. Auwerx,et al.  Bile acids induce energy expenditure by promoting intracellular thyroid hormone activation , 2006, Nature.

[54]  Timothy M Willson,et al.  Activation of the nuclear receptor FXR improves hyperglycemia and hyperlipidemia in diabetic mice. , 2006, Proceedings of the National Academy of Sciences of the United States of America.

[55]  G. Tsujimoto,et al.  Bile acids promote glucagon-like peptide-1 secretion through TGR5 in a murine enteroendocrine cell line STC-1. , 2005, Biochemical and biophysical research communications.

[56]  D. Pertsemlidis,et al.  Composition of biliary lipids and kinetics of bile acids after cholecystectomy in man , 1976, The American Journal of Digestive Diseases.

[57]  R. Soriano,et al.  Fibroblast growth factor 19 increases metabolic rate and reverses dietary and leptin-deficient diabetes. , 2004, Endocrinology.

[58]  B. Casetta,et al.  Quantitative Analysis of Bile Acids in Human Plasma by Liquid Chromatography-Electrospray Tandem Mass Spectrometry: A Simple and Rapid One-Step Method , 2003, Clinical chemistry and laboratory medicine.

[59]  D. French,et al.  Printed in U.S.A. Copyright © 2002 by The Endocrine Society Transgenic Mice Expressing Human Fibroblast Growth Factor-19 Display Increased Metabolic Rate and Decreased Adiposity , 2022 .

[60]  Masahiro Tohkin,et al.  Targeted Disruption of the Nuclear Receptor FXR/BAR Impairs Bile Acid and Lipid Homeostasis , 2000, Cell.

[61]  G. Kullak-Ublick,et al.  Long‐term effects of cholecystectomy on bile acid metabolism , 1995, Hepatology.

[62]  L. Magee,et al.  R 2 Measures Based on Wald and Likelihood Ratio Joint Significance Tests , 1990 .

[63]  F. Berr,et al.  Effects of cholecystectomy on the kinetics of primary and secondary bile acids. , 1989, The Journal of clinical investigation.

[64]  T. Akiyoshi,et al.  Altered Bile Acid Metabolism in Nonobese, Spontaneously Diabetic (NOD) Mice , 1985, Diabetes.

[65]  K. Einarsson,et al.  Hepatic uptake of bile acids in man. Fasting and postprandial concentrations of individual bile acids in portal venous and systemic blood serum. , 1982, The Journal of clinical investigation.

[66]  D. H. Gregory,et al.  Bile acid pools, kinetics and biliary lipid composition before and after cholecystectomy. , 1973, New England Journal of Medicine.