Decoding the coupled decision-making of the epithelial-mesenchymal transition and metabolic reprogramming in cancer
暂无分享,去创建一个
[1] D. Hanahan. Hallmarks of Cancer: New Dimensions. , 2022, Cancer discovery.
[2] H. Levine,et al. Quantifying the patterns of metabolic plasticity and heterogeneity along the epithelial-hybrid-mesenchymal spectrum in cancer , 2021, bioRxiv.
[3] I. Rodriguez-Hernandez,et al. The amoeboid state as part of the epithelial-to-mesenchymal transition programme. , 2021, Trends in cell biology.
[4] Shiva Gholizadeh-Ghaleh Aziz,et al. The emerging role of miR-200 family in metastasis: focus on EMT, CSCs, angiogenesis, and anoikis , 2021, Molecular Biology Reports.
[5] Beth K. Martin,et al. Single-cell lineage tracing of metastatic cancer reveals selection of hybrid EMT states. , 2021, Cancer cell.
[6] J. Cheong,et al. Pan-Cancer Analysis Reveals Distinct Metabolic Reprogramming in Different Epithelial–Mesenchymal Transition Activity States , 2021, Cancers.
[7] R. DePinho,et al. Metabolic Codependencies in the Tumor Microenvironment. , 2021, Cancer discovery.
[8] H. Levine,et al. A mechanistic modeling framework reveals the key principles underlying tumor metabolism , 2021, bioRxiv.
[9] E. Morii,et al. Metabolic Reprogramming of Cancer Cells during Tumor Progression and Metastasis , 2021, Metabolites.
[10] J. Onuchic,et al. Towards decoding the coupled decision-making of metabolism and epithelial-to-mesenchymal transition in cancer , 2020, British Journal of Cancer.
[11] Shubham Tripathi,et al. Biological Networks Regulating Cell Fate Choice Are Minimally Frustrated. , 2020, Physical review letters.
[12] A. Rustgi,et al. EMT, MET, Plasticity, and Tumor Metastasis. , 2020, Trends in cell biology.
[13] T. Wen,et al. NRF2, a Transcription Factor for Stress Response and Beyond , 2020, International journal of molecular sciences.
[14] Haitao Ma,et al. Low levels of AMPK promote epithelial‐mesenchymal transition in lung cancer primarily through HDAC4‐ and HDAC5‐mediated metabolic reprogramming , 2020, Journal of cellular and molecular medicine.
[15] M. Jolly,et al. Hypoxia, partial EMT and collective migration: Emerging culprits in metastasis , 2020, Translational oncology.
[16] Jin Wang,et al. Exposing the Underlying Relationship of Cancer Metastasis to Metabolism and Epithelial-Mesenchymal Transitions , 2019, iScience.
[17] Shiwen Xu,et al. Chlorpyrifos induced oxidative stress to promote apoptosis and autophagy through the regulation of miR-19a-AMPK axis in common carp. , 2019, Fish & shellfish immunology.
[18] Jaeseung Yoon,et al. MiR-200c downregulates HIF-1α and inhibits migration of lung cancer cells , 2019, Cellular & Molecular Biology Letters.
[19] J. Onuchic,et al. Quantifying Cancer Epithelial-Mesenchymal Plasticity and its Association with Stemness and Immune Response , 2019, Journal of clinical medicine.
[20] R. Lamont,et al. Streptococcus gordonii programs epithelial cells to resist ZEB2 induction by Porphyromonas gingivalis , 2019, Proceedings of the National Academy of Sciences.
[21] J. Onuchic,et al. Elucidating cancer metabolic plasticity by coupling gene regulation with metabolic pathways , 2019, Proceedings of the National Academy of Sciences.
[22] H. Harada,et al. HIF-1-Dependent Reprogramming of Glucose Metabolic Pathway of Cancer Cells and Its Therapeutic Significance , 2019, International journal of molecular sciences.
[23] N. Kim,et al. Therapeutic implications of cancer epithelial-mesenchymal transition (EMT) , 2019, Archives of Pharmacal Research.
[24] Jingxia Li,et al. Downregulation of miR-200c stabilizes XIAP mRNA and contributes to invasion and lung metastasis of bladder cancer , 2019, Cell adhesion & migration.
[25] E. Ben-Jacob,et al. Toward understanding cancer stem cell heterogeneity in the tumor microenvironment , 2018, Proceedings of the National Academy of Sciences.
[26] L. Jia,et al. Metastatic cancer cells compensate for low energy supplies in hostile microenvironments with bioenergetic adaptation and metabolic reprogramming. , 2018, International journal of oncology.
[27] M. Saitoh. Involvement of partial EMT in cancer progression. , 2018, Journal of biochemistry.
[28] Jason T. George,et al. NRF2 activates a partial epithelial-mesenchymal transition and is maximally present in a hybrid epithelial/mesenchymal phenotype , 2018, bioRxiv.
[29] Michael D. Brooks,et al. Targeting Breast Cancer Stem Cell State Equilibrium through Modulation of Redox Signaling. , 2018, Cell metabolism.
[30] Jason T. George,et al. A mechanism-based computational model to capture the interconnections among epithelial-mesenchymal transition, cancer stem cells and Notch-Jagged signaling , 2018, bioRxiv.
[31] T. Voet,et al. Identification of the tumour transition states occurring during EMT , 2018, Nature.
[32] Michael D. Brooks,et al. Heterogeneity of Human Breast Stem and Progenitor Cells as Revealed by Transcriptional Profiling , 2018, Stem cell reports.
[33] H. Levine,et al. Elucidating the Metabolic Plasticity of Cancer: Mitochondrial Reprogramming and Hybrid Metabolic States , 2018, Cells.
[34] Rafał Bartoszewski,et al. miRNAs regulate the HIF switch during hypoxia: a novel therapeutic target , 2018, Angiogenesis.
[35] Mohit Kumar Jolly,et al. Survival Outcomes in Cancer Patients Predicted by a Partial EMT Gene Expression Scoring Metric. , 2017, Cancer research.
[36] C. Frezza,et al. Metabolic reprogramming and epithelial‐to‐mesenchymal transition in cancer , 2017, The FEBS journal.
[37] P. Bertolazzi,et al. Characterization of epithelial-mesenchymal transition intermediate/hybrid phenotypes associated to resistance to EGFR inhibitors in non-small cell lung cancer cell lines , 2017, Oncotarget.
[38] X. Zong,et al. Aberrant cancer metabolism in epithelial-mesenchymal transition and cancer metastasis: Mechanisms in cancer progression. , 2017, Critical reviews in oncology/hematology.
[39] Jianpeng Ma,et al. Modeling the Genetic Regulation of Cancer Metabolism: Interplay between Glycolysis and Oxidative Phosphorylation. , 2017, Cancer research.
[40] P. Liu,et al. FOXO1 inhibits the invasion and metastasis of hepatocellular carcinoma by reversing ZEB2-induced epithelial-mesenchymal transition , 2016, Oncotarget.
[41] M. Esteller,et al. Oncometabolite Accumulation and Epithelial-to-Mesenchymal Transition: The Turn of Fumarate. , 2016, Cell metabolism.
[42] Sendurai A Mani,et al. Whom to blame for metastasis, the epithelial-mesenchymal transition or the tumor microenvironment? , 2016, Cancer letters.
[43] Kumari Sonal Choudhary,et al. EGFR Signal-Network Reconstruction Demonstrates Metabolic Crosstalk in EMT , 2016, PLoS Comput. Biol..
[44] K. Yanagihara,et al. Impaired mitophagy activates mtROS/HIF-1α interplay and increases cancer aggressiveness in gastric cancer cells under hypoxia. , 2016, International journal of oncology.
[45] E. Ben-Jacob,et al. Stability of the hybrid epithelial/mesenchymal phenotype , 2016, Oncotarget.
[46] J. Locasale,et al. The Warburg Effect: How Does it Benefit Cancer Cells? , 2016, Trends in biochemical sciences.
[47] S. Leung,et al. PDK1-Dependent Metabolic Reprogramming Dictates Metastatic Potential in Breast Cancer. , 2015, Cell metabolism.
[48] Eshel Ben-Jacob,et al. Coupling the modules of EMT and stemness: A tunable ‘stemness window’ model , 2015, Oncotarget.
[49] J. Lieberman,et al. miR-34 and p53: New Insights into a Complex Functional Relationship , 2015, PloS one.
[50] M. Zeisberg,et al. Snail Is a Direct Target of Hypoxia-inducible Factor 1α (HIF1α) in Hypoxia-induced Endothelial to Mesenchymal Transition of Human Coronary Endothelial Cells* , 2015, The Journal of Biological Chemistry.
[51] Ying Zhang,et al. Nrf2 regulates ROS production by mitochondria and NADPH oxidase , 2015, Biochimica et biophysica acta.
[52] A. Piotrowski,et al. The hypoxia‐inducible miR‐429 regulates hypoxia‐inducible factor‐1α expression in human endothelial cells through a negative feedback loop , 2015, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.
[53] Weidong Huang,et al. AMPK Plays a Dual Role in Regulation of CREB/BDNF Pathway in Mouse Primary Hippocampal Cells , 2015, Journal of Molecular Neuroscience.
[54] R. Kalluri,et al. Corrigendum: PGC-1α mediates mitochondrial biogenesis and oxidative phosphorylation in cancer cells to promote metastasis , 2014, Nature Cell Biology.
[55] C. Shapiro,et al. AMPK reverses the mesenchymal phenotype of cancer cells by targeting the Akt-MDM2-Foxo3a signaling axis. , 2014, Cancer research.
[56] T. Copetti,et al. A mitochondrial switch promotes tumor metastasis. , 2014, Cell reports.
[57] Eshel Ben-Jacob,et al. MicroRNA-based regulation of epithelial–hybrid–mesenchymal fate determination , 2013, Proceedings of the National Academy of Sciences.
[58] E. Ben-Jacob,et al. Tristability in cancer-associated microRNA-TF chimera toggle switch. , 2013, The journal of physical chemistry. B.
[59] G. Melino,et al. Identification of NCF2/p67phox as a novel p53 target gene , 2012, Cell cycle.
[60] Xueyuan Bai,et al. miR-335 and miR-34a Promote renal senescence by suppressing mitochondrial antioxidative enzymes. , 2011, Journal of the American Society of Nephrology : JASN.
[61] Harshini Sarojini,et al. Increased expression of miR-34a and miR-93 in rat liver during aging, and their impact on the expression of Mgst1 and Sirt1 , 2011, Mechanisms of Ageing and Development.
[62] J. Hayashi,et al. ROS-Generating Mitochondrial DNA Mutations Can Regulate Tumor Cell Metastasis , 2008, Science.
[63] D. Albertson,et al. Rac1b and reactive oxygen species mediate MMP-3-induced EMT and genomic instability , 2005, Nature.
[64] E. Hay,et al. The mesenchymal cell, its role in the embryo, and the remarkable signaling mechanisms that create it , 2005, Developmental dynamics : an official publication of the American Association of Anatomists.
[65] O. Warburg,et al. THE METABOLISM OF TUMORS IN THE BODY , 1927, The Journal of general physiology.
[66] J. Paul,et al. New Dimensions , 2011 .
[67] T. Clanton,et al. Journal of Applied Physiology publishes original papers that deal with diverse area of research in applied , 2007 .