The genetic architecture of breast papillary lesions as a predictor of progression to carcinoma
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S. Fox | A. Green | E. Rakha | I. Campbell | I. Miligy | D. Goode | K. Gorringe | D. Byrne | Simone M. Rowley | T. Semple | P. Hill | G. Mann | J. Pang | K. Elder | D. Cheasley | Magnus Zethoven | N. Thio | T. Kader | Tanjina Kader | A. Green | David L Goode | Ian G. Campbell | Kylie L. Gorringe | G. Mann
[1] S. Fox,et al. The genetic architecture of breast papillary lesions as a predictor of progression to carcinoma , 2020, npj Breast Cancer.
[2] O. Mariani,et al. The Genomic Landscape of Mucinous Breast Cancer. , 2019, Journal of the National Cancer Institute.
[3] E. Morris,et al. Correction to: Second International Consensus Conference on lesions of uncertain malignant potential in the breast (B3 lesions) , 2019, Breast Cancer Research and Treatment.
[4] Kylie L. Gorringe,et al. Atypical ductal hyperplasia is a multipotent precursor of breast carcinoma , 2019, The Journal of pathology.
[5] D. Karamchandani,et al. Excision recommended in high‐risk patients: Revisiting the diagnosis of papilloma on core biopsy in the context of patient risk , 2019, The breast journal.
[6] E. Morris,et al. Second International Consensus Conference on lesions of uncertain malignant potential in the breast (B3 lesions) , 2018, Breast Cancer Research and Treatment.
[7] D. Karamchandani,et al. Should we excise? Are there any clinical or histologic features that predict upgrade in papillomas, incidental or non-incidental? , 2018, Annals of diagnostic pathology.
[8] S. Noguchi,et al. Mutational Analysis of AKT1 and PIK3CA in Intraductal Papillomas of the Breast with Special Reference to Cellular Components. , 2018, The American journal of pathology.
[9] Kylie L. Gorringe,et al. Molecular analysis of PALB2‐associated breast cancers , 2018, The Journal of pathology.
[10] H. Bear,et al. Papillary lesions of the breast: To excise or observe? , 2018, The breast journal.
[11] Min Kyoon Kim,et al. Management of benign papilloma without atypia diagnosed at ultrasound-guided core needle biopsy: Scoring system for predicting malignancy. , 2018, European journal of surgical oncology : the journal of the European Society of Surgical Oncology and the British Association of Surgical Oncology.
[12] Kylie L. Gorringe,et al. Breast ductal carcinoma in situ carry mutational driver events representative of invasive breast cancer , 2017, Modern Pathology.
[13] Kylie L. Gorringe,et al. Copy number analysis by low coverage whole genome sequencing using ultra low-input DNA from formalin-fixed paraffin embedded tumor tissue , 2016, Genome Medicine.
[14] E. Morris,et al. First International Consensus Conference on lesions of uncertain malignant potential in the breast (B3 lesions) , 2016, Breast Cancer Research and Treatment.
[15] F. Cunningham,et al. The Ensembl Variant Effect Predictor , 2016, Genome Biology.
[16] Min Jung Kim,et al. Asymptomatic Benign Papilloma Without Atypia Diagnosed at Ultrasonography-Guided 14-Gauge Core Needle Biopsy: Which Subgroup can be Managed by Observation? , 2016, Annals of Surgical Oncology.
[17] James Y. Zou. Analysis of protein-coding genetic variation in 60,706 humans , 2015, Nature.
[18] A. Viale,et al. Massively Parallel Sequencing-Based Clonality Analysis of Synchronous Endometrioid Endometrial and Ovarian Carcinomas. , 2015, Journal of the National Cancer Institute.
[19] Kylie L. Gorringe,et al. Copy number analysis of ductal carcinoma in situ with and without recurrence , 2015, Modern Pathology.
[20] Masayuki Yoshida,et al. Intraductal papillomas on core biopsy can be upgraded to malignancy on subsequent excisional biopsy regardless of the presence of atypical features , 2015, Pathology international.
[21] Serafim Batzoglou,et al. Cell-lineage heterogeneity and driver mutation recurrence in pre-invasive breast neoplasia , 2015, Genome Medicine.
[22] Jos Jonkers,et al. CopywriteR: DNA copy number detection from off-target sequence data , 2015, Genome Biology.
[23] Pieter Wesseling,et al. DNA copy number analysis of fresh and formalin-fixed specimens by shallow whole-genome sequencing with identification and exclusion of problematic regions in the genome assembly , 2014, Genome research.
[24] G. McVean,et al. Integrating mapping-, assembly- and haplotype-based approaches for calling variants in clinical sequencing applications , 2014, Nature Genetics.
[25] Julie C. Sapp,et al. Clinical Delineation and Natural History of the PIK3CA-Related Overgrowth Spectrum** , 2014, American journal of medical genetics. Part A.
[26] Q. Hu,et al. Intracystic papillary carcinoma of breast: interrelationship with in situ and invasive carcinoma and a proposal of pathogenesis: array comparative genomic hybridization study of 14 cases , 2014, Modern Pathology.
[27] N. Sneige,et al. Benign papillomas without atypia diagnosed on core needle biopsy: experience from a single institution and proposed criteria for excision. , 2013, Clinical breast cancer.
[28] Mauricio O. Carneiro,et al. From FastQ Data to High‐Confidence Variant Calls: The Genome Analysis Toolkit Best Practices Pipeline , 2013, Current protocols in bioinformatics.
[29] Heng Li. Aligning sequence reads, clone sequences and assembly contigs with BWA-MEM , 2013, 1303.3997.
[30] Jiri Bartek,et al. Replication stress links structural and numerical cancer chromosomal instability , 2013, Nature.
[31] Helga Thorvaldsdóttir,et al. Integrative Genomics Viewer (IGV): high-performance genomics data visualization and exploration , 2012, Briefings Bioinform..
[32] Steven J. M. Jones,et al. Comprehensive molecular portraits of human breast tumours , 2013 .
[33] L. Wilke,et al. Risk of Invasive Breast Cancer and Ductal Carcinoma In Situ in Women with Atypical Papillary Lesions of the Breast , 2012, The breast journal.
[34] Steven J. M. Jones,et al. Comprehensive molecular portraits of human breast tumors , 2012, Nature.
[35] F. Markowetz,et al. The genomic and transcriptomic architecture of 2,000 breast tumours reveals novel subgroups , 2012, Nature.
[36] Christopher A. Miller,et al. VarScan 2: somatic mutation and copy number alteration discovery in cancer by exome sequencing. , 2012, Genome research.
[37] Tatiana Popova,et al. Supplementary Methods , 2012, Acta Neuropsychiatrica.
[38] Sunil R. Lakhani,et al. WHO classification of tumours of the breast , 2012 .
[39] W. Cheng,et al. Nonmalignant Breast Papillary Lesions at Core-Needle Biopsy: A Meta-analysis of Underestimation and Influencing Factors , 2012, Annals of Surgical Oncology.
[40] Samantha E. Boyle,et al. Identification of copy number alterations associated with the progression of DCIS to invasive ductal carcinoma , 2012, Breast Cancer Research and Treatment.
[41] T. Nagayasu,et al. Intracystic Papillary Carcinoma of Breast Harbors Significant Genomic Alteration Compared with Intracystic Papilloma: Genome‐wide Copy Number and LOH Analysis Using High‐Density Single‐Nucleotide Polymorphism Microarrays , 2011, The breast journal.
[42] Vilppu J Tuominen,et al. ImmunoRatio: a publicly available web application for quantitative image analysis of estrogen receptor (ER), progesterone receptor (PR), and Ki-67 , 2010, Breast Cancer Research.
[43] K. Byth,et al. Diagnostic evaluation of papillary lesions of the breast on core biopsy , 2010, Modern Pathology.
[44] Janice Patterson,et al. High prevalence of PIK3CA/AKT pathway mutations in papillary neoplasms of the breast , 2010, Modern Pathology.
[45] Malcolm Buchanan,et al. Benign Papilloma on Core Biopsy Requires Surgical Excision , 2008, Annals of surgical oncology.
[46] Emmanuel Barillot,et al. High-resolution mapping of DNA breakpoints to define true recurrences among ipsilateral breast cancers. , 2008, Journal of the National Cancer Institute.
[47] L. Collins,et al. Papillary lesions of the breast: selected diagnostic and management issues , 2007, Histopathology.
[48] Matthew Eckert,et al. Breast Papillomas in the Era of Percutaneous Needle Biopsy , 2007, Annals of Surgical Oncology.
[49] L. Jackson,et al. Multiple papillomas of the breast: is current management adequate? , 2006, Breast.
[50] G. Bevilacqua,et al. Papillary lesions of the breast: a molecular progression? , 2005, Breast Cancer Research and Treatment.
[51] P. V. van Diest,et al. Ductal epithelial proliferations of the breast: a biological continuum? Comparative genomic hybridization and high‐molecular‐weight cytokeratin expression patterns , 2001, The Journal of pathology.
[52] H. Koyama,et al. Diagnosis of ductal carcinoma in situ (DCIS) and intraductal papilloma using fluorescence in Situ Hybridization (FISH) analysis , 2000, Breast cancer.
[53] C K Osborne,et al. Estrogen receptor status by immunohistochemistry is superior to the ligand-binding assay for predicting response to adjuvant endocrine therapy in breast cancer. , 1999, Journal of clinical oncology : official journal of the American Society of Clinical Oncology.
[54] S. Hirohashi,et al. Detection of numerical alterations of chromosomes 3, 7, 17 and X in low-grade intracystic papillary tumors of the breast by multi-color fluorescencein situ hybridization , 1997, Breast cancer.
[55] S. Hirohashi,et al. Detection of numerical and structural alterations and fusion of chromosomes 16 and 1 in low-grade papillary breast carcinoma by fluorescence in situ hybridization. , 1997, The American journal of pathology.
[56] W. Dupont,et al. Subsequent breast carcinoma risk after biopsy with atypia in a breast papilloma , 1996, Cancer.
[57] S. Hirohashi,et al. Different Incidence of Loss of Heterozygosity on Chromosome 16q between Intraductal Papilloma and Intracystic Papillary Carcinoma of the Breast , 1994, Japanese journal of cancer research : Gann.
[58] H. Bartsch,et al. International Agency for Research on Cancer. , 1969, WHO chronicle.
[59] A. Stout,et al. THE PAPILLARY NEOPLASMS OF THE BREAST. I. BENIGN INTRADUCTAL PAPILLOMA , 1951, Annals of surgery.