A unified model of NMDA receptor-dependent bidirectional synaptic plasticity
暂无分享,去创建一个
[1] H. Kalmus. Biological Cybernetics , 1972, Nature.
[2] T. Bliss,et al. Long‐lasting potentiation of synaptic transmission in the dentate area of the anaesthetized rabbit following stimulation of the perforant path , 1973, The Journal of physiology.
[3] E. Bienenstock,et al. Theory for the development of neuron selectivity: orientation specificity and binocular interaction in visual cortex , 1982, The Journal of neuroscience : the official journal of the Society for Neuroscience.
[4] W. Levy,et al. Temporal contiguity requirements for long-term associative potentiation/depression in the hippocampus , 1983, Neuroscience.
[5] L. Cooper,et al. A physiological basis for a theory of synapse modification. , 1987, Science.
[6] M. Stryker,et al. Neural plasticity without postsynaptic action potentials: less-active inputs become dominant when kitten visual cortical cells are pharmacologically inhibited. , 1988, Proceedings of the National Academy of Sciences of the United States of America.
[7] W Singer,et al. Chronic recordings from single sites of kitten striate cortex during experience-dependent modifications of receptive-field properties. , 1989, Journal of neurophysiology.
[8] J. Lisman,et al. A mechanism for the Hebb and the anti-Hebb processes underlying learning and memory. , 1989, Proceedings of the National Academy of Sciences of the United States of America.
[9] W. Singer,et al. Different voltage-dependent thresholds for inducing long-term depression and long-term potentiation in slices of rat visual cortex , 1990, Nature.
[10] C. Stevens,et al. Voltage dependence of NMDA-activated macroscopic conductances predicted by single-channel kinetics , 1990, The Journal of neuroscience : the official journal of the Society for Neuroscience.
[11] G. Carmignoto,et al. Activity-dependent decrease in NMDA receptor responses during development of the visual cortex. , 1992, Science.
[12] M. Bear,et al. Homosynaptic long-term depression in area CA1 of hippocampus and effects of N-methyl-D-aspartate receptor blockade. , 1992, Proceedings of the National Academy of Sciences of the United States of America.
[13] Nathan Intrator,et al. Objective function formulation of the BCM theory of visual cortical plasticity: Statistical connections, stability conditions , 1992, Neural Networks.
[14] R. Malenka,et al. Mechanisms underlying induction of homosynaptic long-term depression in area CA1 of the hippocampus , 1992, Neuron.
[15] W. Singer,et al. Long-term depression of excitatory synaptic transmission and its relationship to long-term potentiation , 1993, Trends in Neurosciences.
[16] SM Dudek,et al. Bidirectional long-term modification of synaptic effectiveness in the adult and immature hippocampus , 1993, The Journal of neuroscience : the official journal of the Society for Neuroscience.
[17] C. Stevens,et al. Changes in reliability of synaptic function as a mechanism for plasticity , 1994, Nature.
[18] M. Bear,et al. Homosynaptic long-term depression in the visual cortex , 1994, The Journal of neuroscience : the official journal of the Society for Neuroscience.
[19] D. Signorini,et al. Neural networks , 1995, The Lancet.
[20] H. Markram,et al. Dendritic calcium transients evoked by single back‐propagating action potentials in rat neocortical pyramidal neurons. , 1995, The Journal of physiology.
[21] Michael C. Crair,et al. A critical period for long-term potentiation at thalamocortical synapses , 1995, Nature.
[22] R. Nicoll,et al. Ca2+ Signaling Requirements for Long-Term Depression in the Hippocampus , 1996, Neuron.
[23] M. Bear,et al. Metaplasticity: the plasticity of synaptic plasticity , 1996, Trends in Neurosciences.
[24] B. Sakmann,et al. Action potential initiation and propagation in rat neocortical pyramidal neurons , 1997, The Journal of physiology.
[25] H. Markram,et al. Regulation of Synaptic Efficacy by Coincidence of Postsynaptic APs and EPSPs , 1997, Science.
[26] D. Johnston,et al. A Synaptically Controlled, Associative Signal for Hebbian Plasticity in Hippocampal Neurons , 1997, Science.
[27] Daniel E Feldman,et al. Long-Term Depression at Thalamocortical Synapses in Developing Rat Somatosensory Cortex , 1998, Neuron.
[28] G. Bi,et al. Synaptic Modifications in Cultured Hippocampal Neurons: Dependence on Spike Timing, Synaptic Strength, and Postsynaptic Cell Type , 1998, The Journal of Neuroscience.
[29] D. Debanne,et al. Long‐term synaptic plasticity between pairs of individual CA3 pyramidal cells in rat hippocampal slice cultures , 1998, The Journal of physiology.
[30] R. Kempter,et al. Hebbian learning and spiking neurons , 1999 .
[31] R. Zucker,et al. Selective induction of LTP and LTD by postsynaptic [Ca2+]i elevation. , 1999, Journal of neurophysiology.
[32] E. B. Roberts,et al. Enhanced NR2A subunit expression and decreased NMDA receptor decay time at the onset of ocular dominance plasticity in the ferret. , 1999, Journal of neurophysiology.
[33] Mark F. Bear,et al. Rapid, experience-dependent expression of synaptic NMDA receptors in visual cortex in vivo , 1999, Nature Neuroscience.
[34] R. Nicoll,et al. Long-term potentiation--a decade of progress? , 1999, Science.
[35] Davide Badoni,et al. Spike-Driven Synaptic Plasticity: Theory, Simulation, VLSI Implementation , 2000, Neural Computation.
[36] Mark C. W. van Rossum,et al. Stable Hebbian Learning from Spike Timing-Dependent Plasticity , 2000, The Journal of Neuroscience.
[37] M. V. Rossum,et al. Activity Coregulates Quantal AMPA and NMDA Currents at Neocortical Synapses , 2000, Neuron.
[38] L. Abbott,et al. Competitive Hebbian learning through spike-timing-dependent synaptic plasticity , 2000, Nature Neuroscience.
[39] M. Poo,et al. Calcium stores regulate the polarity and input specificity of synaptic modification , 2000, Nature.
[40] Ken-ichi Hara,et al. A generalized Hebbian rule for activity-dependent synaptic modifications , 2000, Neural Networks.
[41] M. Constantine‐Paton,et al. Activity-Dependent Induction of Tonic Calcineurin Activity Mediates a Rapid Developmental Downregulation of NMDA Receptor Currents , 2000, Neuron.
[42] D. Feldman,et al. Timing-Based LTP and LTD at Vertical Inputs to Layer II/III Pyramidal Cells in Rat Barrel Cortex , 2000, Neuron.
[43] M. Bear,et al. Visual Experience and Deprivation Bidirectionally Modify the Composition and Function of NMDA Receptors in Visual Cortex , 2001, Neuron.
[44] Henry Markram,et al. An Algorithm for Modifying Neurotransmitter Release Probability Based on Pre- and Postsynaptic Spike Timing , 2001, Neural Computation.
[45] M. W. Brown,et al. An experimental test of the role of postsynaptic calcium levels in determining synaptic strength using perirhinal cortex of rat , 2001, The Journal of physiology.
[46] B. Sakmann,et al. Dendritic mechanisms underlying the coupling of the dendritic with the axonal action potential initiation zone of adult rat layer 5 pyramidal neurons , 2001, The Journal of physiology.
[47] Wulfram Gerstner,et al. Intrinsic Stabilization of Output Rates by Spike-Based Hebbian Learning , 2001, Neural Computation.
[48] D. Wilkin,et al. Neuron , 2001, Brain Research.
[49] P. J. Sjöström,et al. Rate, Timing, and Cooperativity Jointly Determine Cortical Synaptic Plasticity , 2001, Neuron.
[50] A. C. Greenwood,et al. Bidirectional synaptic plasticity correlated with the magnitude of dendritic calcium transients above a threshold. , 2001, Journal of neurophysiology.
[51] L. Cooper,et al. A biophysical model of bidirectional synaptic plasticity: Dependence on AMPA and NMDA receptors , 2001, Proceedings of the National Academy of Sciences of the United States of America.
[52] K. Svoboda,et al. The Life Cycle of Ca2+ Ions in Dendritic Spines , 2002, Neuron.