Multiple Local Contact Sites are Induced by GPI‐Linked Influenza Hemagglutinin During Hemifusion and Flickering Pore Formation
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P. Bronk | T. Reese | J. Zimmerberg | M. Cho | V. Frolov | Joshua Zimmerberg | Vadim A. Frolov | Myoung‐Soon Cho | Peter Bronk | Thomas S. Reese | Peter Bronk
[1] Y. Henis,et al. GPI- and transmembrane-anchored influenza hemagglutinin differ in structure and receptor binding activity , 1993, The Journal of cell biology.
[2] E. Neher. Asymmetric membranes resulting from the fusion of two black lipid bilayers. , 1974, Biochimica et biophysica acta.
[3] F S Cohen,et al. Fusion of phospholipid vesicles with planar phospholipid bilayer membranes. I. Discharge of vesicular contents across the planar membrane , 1980, The Journal of general physiology.
[4] J. Zimmerberg,et al. Restricted movement of lipid and aqueous dyes through pores formed by influenza hemagglutinin during cell fusion , 1994, The Journal of cell biology.
[5] G. Melikyan,et al. The lipid-anchored ectodomain of influenza virus hemagglutinin (GPI-HA) is capable of inducing nonenlarging fusion pores. , 2000, Molecular biology of the cell.
[6] J. Zimmerberg,et al. Fusion pore conductance: experimental approaches and theoretical algorithms. , 1998, Biophysical Journal.
[7] J. Zimmerberg,et al. Fusion of phospholipid vesicles with planar phospholipid bilayer membranes. II. Incorporation of a vesicular membrane marker into the planar membrane , 1980, The Journal of general physiology.
[8] A. Herrmann,et al. Meta-stability of the hemifusion intermediate induced by glycosylphosphatidylinositol-anchored influenza hemagglutinin. , 1997, Biophysical journal.
[9] J. White. Membrane fusion: the influenza paradigm. , 1995, Cold Spring Harbor symposia on quantitative biology.
[10] George Palade,et al. Intracellular Aspects of the Process of Protein Synthesis , 1975, Science.
[11] J. Heuser,et al. Arrest of membrane fusion events in mast cells by quick-freezing , 1980, The Journal of cell biology.
[12] P. Bronk,et al. The Pathway of Membrane Fusion Catalyzed by Influenza Hemagglutinin: Restriction of Lipids, Hemifusion, and Lipidic Fusion Pore Formation , 1998, The Journal of cell biology.
[13] D. Z. Cleverley,et al. The transmembrane domain in viral fusion: essential role for a conserved glycine residue in vesicular stomatitis virus G protein. , 1998, Proceedings of the National Academy of Sciences of the United States of America.
[14] J. White,et al. GPI-anchored influenza hemagglutinin induces hemifusion to both red blood cell and planar bilayer membranes , 1995, The Journal of cell biology.
[15] H. Plattner,et al. Membrane events in adrenal chromaffin cells during exocytosis: a freeze-etching analysis after rapid cryofixation. , 1983, European journal of cell biology.
[16] D. P. Sarkar,et al. Hemagglutinin-neuraminidase enhances F protein-mediated membrane fusion of reconstituted Sendai virus envelopes with cells , 1993, Journal of virology.
[17] W. Almers,et al. Membrane flux through the pore formed by a fusogenic viral envelope protein during cell fusion , 1993, The Journal of cell biology.
[18] L. Chernomordik,et al. Biomembrane fusion: a new concept derived from model studies using two interacting planar lipid bilayers. , 1987, Biochimica et biophysica acta.
[19] T. Reese,et al. Beginning of exocytosis captured by rapid-freezing of Limulus amebocytes , 1981, The Journal of cell biology.
[20] H. Plattner,et al. Synchronous exocytosis in Paramecium cells. II. Intramembranous changes analysed by freeze-fracturing. , 1984, Experimental cell research.
[21] J. Zimmerberg,et al. Flickering fusion pores comparable with initial exocytotic pores occur in protein-free phospholipid bilayers. , 1997, Proceedings of the National Academy of Sciences of the United States of America.
[22] G. Melikyan,et al. Tension of membranes expressing the hemagglutinin of influenza virus inhibits fusion. , 1999, Biophysical journal.
[23] R. Lamb,et al. Truncation of the COOH-terminal region of the paramyxovirus SV5 fusion protein leads to hemifusion but not complete fusion , 1996, The Journal of cell biology.
[24] J. Skehel,et al. N- and C-terminal residues combine in the fusion-pH influenza hemagglutinin HA(2) subunit to form an N cap that terminates the triple-stranded coiled coil. , 1999, Proceedings of the National Academy of Sciences of the United States of America.
[25] P. Bronk,et al. An Early Stage of Membrane Fusion Mediated by the Low pH Conformation of Influenza Hemagglutinin Depends upon Membrane Lipids , 1997, The Journal of cell biology.
[26] J. Skehel,et al. Structure of influenza haemagglutinin at the pH of membrane fusion , 1994, Nature.
[27] S. Durell,et al. Dilation of the influenza hemagglutinin fusion pore revealed by the kinetics of individual cell-cell fusion events , 1996, The Journal of cell biology.
[28] R. Assoian,et al. Anchorage-dependent Cell Cycle Progression , 1997, The Journal of cell biology.
[29] J. Heuser. Membrane Traffic in Anaglyph Stereo , 2000, Traffic.
[30] M. Kozlov,et al. A mechanism of protein-mediated fusion: coupling between refolding of the influenza hemagglutinin and lipid rearrangements. , 1998, Biophysical journal.
[31] S. Pelletier,et al. Membrane fusion mediated by the influenza virus hemagglutinin requires the concerted action of at least three hemagglutinin trimers , 1996, The Journal of cell biology.
[32] G. Melikyan,et al. Inner but Not Outer Membrane Leaflets Control the Transition from Glycosylphosphatidylinositol-anchored Influenza Hemagglutinin-induced Hemifusion to Full Fusion , 1997, The Journal of cell biology.
[33] L. Chernomordik,et al. Reversible merger of membranes at the early stage of influenza hemagglutinin-mediated fusion. , 2000, Molecular biology of the cell.
[34] F S Cohen,et al. A specific point mutant at position 1 of the influenza hemagglutinin fusion peptide displays a hemifusion phenotype. , 1999, Molecular biology of the cell.
[35] G. Palade,et al. STRUCTURAL MODULATIONS OF PLASMALEMMAL VESICLES , 1968, The Journal of cell biology.