Pathogenesis of acute coronary syndromes.
暂无分享,去创建一个
[1] Hisashi Adachi,et al. Inflammation, atherosclerosis, and coronary artery disease. , 2005, The New England journal of medicine.
[2] A. Davies,et al. Imaging intraplaque inflammation in carotid atherosclerosis with 11C-PK11195 positron emission tomography/computed tomography. , 2012, European heart journal.
[3] I. Loftus,et al. High Levels of Costimulatory Receptors OX40 and 4-1BB Characterize CD4+CD28null T Cells in Patients With Acute Coronary Syndrome , 2012, Circulation research.
[4] Akiko Maehara,et al. Longitudinal distribution of plaque burden and necrotic core-rich plaques in nonculprit lesions of patients presenting with acute coronary syndromes. , 2012, JACC. Cardiovascular imaging.
[5] E. Braunwald. Creating controversy where none exists: the important role of C-reactive protein in the CARE, AFCAPS/TexCAPS, PROVE IT, REVERSAL, A to Z, JUPITER, HEART PROTECTION, and ASCOT trials. , 2012, European heart journal.
[6] W. R. Taylor,et al. Pharmacological Suppression of Hepcidin Increases Macrophage Cholesterol Efflux and Reduces Foam Cell Formation and Atherosclerosis , 2012, Arteriosclerosis, thrombosis, and vascular biology.
[7] B. Gersh. 3-Year Follow-Up of Patients With Coronary Artery Spasm as Cause of Acute Coronary Syndrome: The CASPAR (Coronary Artery Spasm in Patients With Acute Coronary Syndrome) Study Follow-Up , 2012 .
[8] J. Kastelein,et al. Inhibition of Secretory Phospholipase A2 in Patients with Acute Coronary Syndromes: Rationale and Design of the Vascular Inflammation Suppression to Treat Acute Coronary Syndrome for 16 Weeks (VISTA-16) Trial , 2012, Cardiovascular Drugs and Therapy.
[9] F. Crea,et al. High-sensitivity C-reactive protein is within normal levels at the very onset of first ST-segment elevation acute myocardial infarction in 41% of cases: a multiethnic case-control study. , 2011, Journal of the American College of Cardiology.
[10] C. Pepine,et al. Microvascular dysfunction: what have we learned from WISE? , 2011, Expert review of cardiovascular therapy.
[11] F. Crea,et al. Anti-inflammatory treatment of acute coronary syndromes. , 2011, Current pharmaceutical design.
[12] Chunhong Ma,et al. IL-17 induces apoptosis of vascular endothelial cells: a potential mechanism for human acute coronary syndrome. , 2011, Clinical immunology.
[13] F. Crea,et al. Contemporary Reviews in Cardiovascular Medicine Mechanisms of Coronary Artery Spasm , 2011 .
[14] T. Lüscher,et al. Myeloid related proteins activate Toll-like receptor 4 in human acute coronary syndromes. , 2011, Atherosclerosis.
[15] D. Wood,et al. Mechanisms of Myocardial Infarction in Women Without Angiographically Obstructive Coronary Artery Disease , 2011, Circulation.
[16] Deepak L. Bhatt,et al. Apixaban with antiplatelet therapy after acute coronary syndrome. , 2011, The New England journal of medicine.
[17] J. Freedman,et al. Regulatory effects of TLR2 on megakaryocytic cell function. , 2011, Blood.
[18] F. Crea,et al. Expansion of CD4+CD28null T-lymphocytes in diabetic patients: exploring new pathogenetic mechanisms of increased cardiovascular risk in diabetes mellitus. , 2011, European heart journal.
[19] Mitsuaki Isobe,et al. In vivo critical fibrous cap thickness for rupture-prone coronary plaques assessed by optical coherence tomography. , 2011, European heart journal.
[20] P. Libby,et al. Progress and challenges in translating the biology of atherosclerosis , 2011, Nature.
[21] W. Wongcharoen,et al. Influenza vaccination reduces cardiovascular events in patients with acute coronary syndrome. , 2011, European heart journal.
[22] Akiko Maehara,et al. A prospective natural-history study of coronary atherosclerosis. , 2011, The New England journal of medicine.
[23] Deepak L. Bhatt,et al. APPRAISE 2. Apixaban with Antiplatelet Therapy after Acute Coronary Syndrome for the APPRAISE-2 Investigators* , 2011 .
[24] R. Virmani,et al. Response to Letter Regarding Article, “High Levels of Systemic Myeloperoxidase Are Associated With Coronary Plaque Erosion in Patients With Acute Coronary Syndromes: A Clinicopathological Study” , 2011 .
[25] P. Kovanen,et al. Cholesterol Crystals Activate the NLRP3 Inflammasome in Human Macrophages: A Novel Link between Cholesterol Metabolism and Inflammation , 2010, PloS one.
[26] D. Newby. Triggering of acute myocardial infarction: beyond the vulnerable plaque , 2010, Heart.
[27] Andrew D. Johnson,et al. Relation of Platelet and Leukocyte Inflammatory Transcripts to Body Mass Index in the Framingham Heart Study , 2010, Circulation.
[28] R. Virmani,et al. Concept of vulnerable/unstable plaque. , 2010, Arteriosclerosis, thrombosis, and vascular biology.
[29] Carl J Pepine,et al. Coronary microvascular reactivity to adenosine predicts adverse outcome in women evaluated for suspected ischemia results from the National Heart, Lung and Blood Institute WISE (Women's Ischemia Syndrome Evaluation) study. , 2010, Journal of the American College of Cardiology.
[30] A. Rebuzzi,et al. Reversible coronary microvascular dysfunction: a common pathogenetic mechanism in Apical Ballooning or Tako-Tsubo Syndrome. , 2010, European heart journal.
[31] F. Crea,et al. Primary Coronary Microvascular Dysfunction: Clinical Presentation, Pathophysiology, and Management , 2010, Circulation.
[32] I. Mitroulis,et al. Targeting IL-1beta in disease; the expanding role of NLRP3 inflammasome. , 2010, European journal of internal medicine.
[33] Hong Wang,et al. Th 17 cells interplay with Foxp3+ Tregs in regulation of inflammation and autoimmunity. , 2010, Frontiers in bioscience.
[34] F. Eberli,et al. Cellular actors, Toll-like receptors, and local cytokine profile in acute coronary syndromes. , 2010, European heart journal.
[35] G. Biondi-Zoccai,et al. Interleukin-1 blockade with anakinra to prevent adverse cardiac remodeling after acute myocardial infarction (Virginia Commonwealth University Anakinra Remodeling Trial [VCU-ART] Pilot study). , 2010, The American journal of cardiology.
[36] Shuang Chen,et al. Emerging Role of IL-17 in Atherosclerosis , 2010, Journal of Innate Immunity.
[37] K. Moore,et al. NLRP3 inflamasomes are required for atherogenesis and activated by cholesterol crystals that form early in disease , 2010, Nature.
[38] J. Kaski,et al. C-reactive protein improves risk prediction in patients with acute coronary syndrome, or does it? , 2010, European heart journal.
[39] Hiroto Tsujioka,et al. Multiple coronary lesion instability in patients with acute myocardial infarction as determined by optical coherence tomography. , 2010, The American journal of cardiology.
[40] F. Geissmann,et al. Monocytes in atherosclerosis: subsets and functions , 2010, Nature Reviews Cardiology.
[41] V. Kuchroo,et al. Interleukin-17 and type 17 helper T cells. , 2009, The New England journal of medicine.
[42] Paul M Ridker,et al. Inflammation in atherosclerosis: from pathophysiology to practice. , 2009, Journal of the American College of Cardiology.
[43] F. Crea,et al. Delayed neutrophil apoptosis in patients with unstable angina: relation to C-reactive protein and recurrence of instability. , 2009, European heart journal.
[44] T. Akasaka,et al. Impact of heterogeneity of human peripheral blood monocyte subsets on myocardial salvage in patients with primary acute myocardial infarction. , 2009, Journal of the American College of Cardiology.
[45] P. Libby,et al. Monocyte Subset Dynamics in Human Atherosclerosis Can Be Profiled with Magnetic Nano-Sensors , 2009, PloS one.
[46] K. Aziz,et al. Effect of cholesterol crystals on plaques and intima in arteries of patients with acute coronary and cerebrovascular syndromes. , 2009, The American journal of cardiology.
[47] G. Szot,et al. Expansion of Human Regulatory T-Cells From Patients With Type 1 Diabetes , 2009, Diabetes.
[48] J. Freedman,et al. Stimulation of Toll-Like Receptor 2 in Human Platelets Induces a Thromboinflammatory Response Through Activation of Phosphoinositide 3-Kinase , 2009, Circulation research.
[49] M. Cheitlin. Coronary vasospasm as the underlying cause for chest pain in patients with PVB19 myocarditis , 2009 .
[50] J. Kaski,et al. CD4+ CD28 null T cells in coronary artery disease: when helpers become killers. , 2009, Cardiovascular research.
[51] A. Maseri,et al. Expansion of T-Cell Receptor &zgr;dim Effector T Cells in Acute Coronary Syndromes , 2008, Arteriosclerosis, thrombosis, and vascular biology.
[52] P. Libby,et al. Rosuvastatin to prevent vascular events in men and women with elevated C-reactive protein. , 2008, The New England journal of medicine.
[53] J. Kaski,et al. Interleukin-8 is increased in the membrane of circulating erythrocytes in patients with acute coronary syndrome. , 2008, European heart journal.
[54] J. Danesh,et al. C‐reactive protein and coronary heart disease: a critical review , 2008, Journal of internal medicine.
[55] Frits Mastik,et al. Effects of the Direct Lipoprotein-Associated Phospholipase A2 Inhibitor Darapladib on Human Coronary Atherosclerotic Plaque , 2008, Circulation.
[56] P. Libby,et al. Diversity of denizens of the atherosclerotic plaque: not all monocytes are created equal. , 2008, Circulation.
[57] J. Bluestone,et al. Human regulatory T cells: role in autoimmune disease and therapeutic opportunities , 2008, Immunological reviews.
[58] Yong Chen,et al. The Th17/Treg imbalance in patients with acute coronary syndrome. , 2008, Clinical immunology.
[59] K. Klingel,et al. Coronary vasospasm as the underlying cause for chest pain in patients with PVB19 myocarditis , 2008, Heart.
[60] Fred S Apple,et al. Universal definition of myocardial infarction. , 2007, Journal of the American College of Cardiology.
[61] G. Niccoli,et al. High telomerase activity in neutrophils from unstable coronary plaques. , 2007, Journal of the American College of Cardiology.
[62] M. Shin,et al. Synergistic Proinflammatory Effects of the Antiviral Cytokine Interferon-&agr; and Toll-Like Receptor 4 Ligands in the Atherosclerotic Plaque , 2007, Circulation.
[63] A. Rebuzzi,et al. Unusual CD4+CD28nullT Lymphocytes and Recurrence of Acute Coronary Events , 2007 .
[64] Peng Liu,et al. The opposite-direction modulation of CD4+CD25+ Tregs and T helper 1 cells in acute coronary syndromes. , 2007, Clinical immunology.
[65] F. Crea,et al. Coronary microvascular dysfunction. , 2013, The New England journal of medicine.
[66] J. Kaski,et al. Total cholesterol content of erythrocyte membranes is increased in patients with acute coronary syndrome: a new marker of clinical instability? , 2007, Journal of the American College of Cardiology.
[67] B. Gersh,et al. Microcirculatory dysfunction in ST-elevation myocardial infarction: cause, consequence, or both? , 2007, European heart journal.
[68] P. Libby,et al. Intertwining of thrombosis and inflammation in atherosclerosis , 2007, Current opinion in hematology.
[69] W. Frishman. Patients With Acute Coronary Syndrome Show Oligoclonal T-Cell Recruitment Within Unstable Plaque: Evidence for a Local, Intracoronary Immunologic Mechanism , 2007 .
[70] G. Lip,et al. A systematic review of the effects of acute psychological stress and physical activity on haemorheology, coagulation, fibrinolysis and platelet reactivity: Implications for the pathogenesis of acute coronary syndromes. , 2007, Thrombosis research.
[71] A. Rebuzzi,et al. Unusual CD4+CD28null T lymphocytes and recurrence of acute coronary events. , 2007, Journal of the American College of Cardiology.
[72] G. Keren,et al. Altered status of CD4(+)CD25(+) regulatory T cells in patients with acute coronary syndromes. , 2006, European heart journal.
[73] R. Bugiardini,et al. Unanswered questions for management of acute coronary syndrome: risk stratification of patients with minimal disease or normal findings on coronary angiography. , 2006, Archives of internal medicine.
[74] Kayoko Sato,et al. T Cell Recognition and Killing of Vascular Smooth Muscle Cells in Acute Coronary Syndrome , 2006, Circulation research.
[75] J. Kaski,et al. Cardiac syndrome X in women: the role of oestrogen deficiency , 2006, Heart.
[76] R. Frye,et al. Interleukin 12 Induces T-Cell Recruitment Into the Atherosclerotic Plaque , 2006, Circulation research.
[77] M. Chiariello,et al. Patients With Acute Coronary Syndrome Show Oligoclonal T-Cell Recruitment Within Unstable Plaque: Evidence for a Local, Intracoronary Immunologic Mechanism , 2006, Circulation.
[78] R. Flavell,et al. Natural regulatory T cells control the development of atherosclerosis in mice , 2006, Nature Medicine.
[79] A. Takeshita,et al. Rho-kinase is an important therapeutic target in cardiovascular medicine. , 2009, Arteriosclerosis, thrombosis, and vascular biology.
[80] T. van der Poll,et al. Two-way interactions between inflammation and coagulation. , 2005, Trends in cardiovascular medicine.
[81] Aloke V. Finn,et al. Atherosclerotic Plaque Progression and Vulnerability to Rupture: Angiogenesis as a Source of Intraplaque Hemorrhage , 2005, Arteriosclerosis, thrombosis, and vascular biology.
[82] P. Serruys,et al. The role of shear stress in the destabilization of vulnerable plaques and related therapeutic implications , 2005, Nature Clinical Practice Cardiovascular Medicine.
[83] M. Thomas. Are drug eluting stents really worth the money? , 2005, Heart.
[84] P. Libby. Act local, act global: inflammation and the multiplicity of "vulnerable" coronary plaques. , 2005, Journal of the American College of Cardiology.
[85] K. Shimada,et al. Multiple plaque rupture and C-reactive protein in acute myocardial infarction. , 2005, Journal of the American College of Cardiology.
[86] Elena Bonanno,et al. Diffuse and active inflammation occurs in both vulnerable and stable plaques of the entire coronary tree: a histopathologic study of patients dying of acute myocardial infarction. , 2005, Journal of the American College of Cardiology.
[87] C Noel Bairey Merz,et al. Angina with "normal" coronary arteries. , 2005 .
[88] R. Hubbard,et al. Risk of myocardial infarction and stroke after acute infection or vaccination. , 2004, The New England journal of medicine.
[89] S. Nissen. High-dose statins in acute coronary syndromes: not just lipid levels. , 2004, JAMA.
[90] S. Yusuf,et al. Effect of potentially modifiable risk factors associated with myocardial infarction in 52 countries (the INTERHEART study): case-control study , 2004, The Lancet.
[91] J. Kaski,et al. Markers of inflammation and multiple complex stenoses (pancoronary plaque vulnerability) in patients with non-ST segment elevation acute coronary syndromes , 2004, Heart.
[92] Qingbo Xu,et al. Heat-Shock Protein 60-Reactive CD4+CD28null T Cells in Patients With Acute Coronary Syndromes , 2004, Circulation.
[93] K. Klingel,et al. Parvovirus B19 Infection Mimicking Acute Myocardial Infarction , 2003, Circulation.
[94] F. Crea,et al. Widespread coronary inflammation in unstable angina. , 2003, The New England journal of medicine.
[95] R. Frye,et al. T-Cell–Mediated Lysis of Endothelial Cells in Acute Coronary Syndromes , 2002, Circulation.
[96] A. Maseri,et al. Evidence for Antigen-Driven T-Cell Response in Unstable Angina , 2000, Circulation.
[97] R. Frye,et al. Monoclonal T-cell proliferation and plaque instability in acute coronary syndromes. , 2000, Circulation.
[98] R. Frye,et al. Perturbation of the T-cell repertoire in patients with unstable angina. , 1999, Circulation.
[99] A. Rebuzzi,et al. The prognostic value of C-reactive protein and serum amyloid a protein in severe unstable angina. , 1994, The New England journal of medicine.