Clinical Significance of B7-H1 and B7-1 Expressions in Pancreatic Carcinoma

[1]  H. Friess,et al.  Clinical significance and regulation of the costimulatory molecule B7-H1 in pancreatic cancer. , 2008, Cancer letters.

[2]  G. Zhu,et al.  B7-H1 is a ubiquitous antiapoptotic receptor on cancer cells. , 2008, Blood.

[3]  J. Lukens,et al.  Blockade of PD-1/B7-H1 Interaction Restores Effector CD8+ T Cell Responses in a Hepatitis C Virus Core Murine Model1 , 2008, The Journal of Immunology.

[4]  Shusen Zheng,et al.  B7-H1 up-regulated expression in human pancreatic carcinoma tissue associates with tumor progression , 2008, Journal of Cancer Research and Clinical Oncology.

[5]  N. Katsilambros,et al.  Perturbed Autonomic Nervous System Function in Metabolic Syndrome , 2008, NeuroMolecular Medicine.

[6]  D. Lim,et al.  Effect of anabasine on catecholamine secretion from the perfused rat adrenal medulla. , 2007, Journal of cardiology.

[7]  S. Dermime,et al.  Expression of B7‐H1 in breast cancer patients is strongly associated with high proliferative Ki‐67‐expressing tumor cells , 2007, International journal of cancer.

[8]  N. Sarvetnick,et al.  B7-1 mediated costimulation regulates pancreatic autoimmunity. , 2007, Molecular immunology.

[9]  J. Cheville,et al.  Survivin and B7-H1 Are Collaborative Predictors of Survival and Represent Potential Therapeutic Targets for Patients with Renal Cell Carcinoma , 2007, Clinical Cancer Research.

[10]  Robert D. Schreiber,et al.  Interferons, immunity and cancer immunoediting , 2006, Nature Reviews Immunology.

[11]  A. Mackensen,et al.  Blockade of PD‐L1 (B7‐H1) augments human tumor‐specific T cell responses in vitro , 2006, International journal of cancer.

[12]  M. Colombo,et al.  Low surface expression of B7-1 (CD80) is an immunoescape mechanism of colon carcinoma. , 2006, Cancer research.

[13]  R. DiPaola,et al.  A Phase I Trial of Pox PSA vaccines (PROSTVAC®-VF) with B7-1, ICAM-1, and LFA-3 co-stimulatory molecules (TRICOM™) in Patients with Prostate Cancer , 2006, Journal of Translational Medicine.

[14]  R. Straub,et al.  [Stress, hormones, and neuronal signals in the pathophysiology of rheumatoid arthritis. The negative impact on chronic inflammation]. , 2005, Medizinische Klinik.

[15]  R. Straub,et al.  Stress, Hormone und neuronale Signale bei rheumatoider Arthritis , 2005, Medizinische Klinik.

[16]  Lieping Chen,et al.  Role of B7-H1 and B7-H4 molecules in down-regulating effector phase of T-cell immunity: novel cancer escaping mechanisms. , 2005, Frontiers in bioscience : a journal and virtual library.

[17]  Weiping Zou,et al.  Immunosuppressive networks in the tumour environment and their therapeutic relevance , 2005, Nature Reviews Cancer.

[18]  G. Zhu,et al.  Blockade of B7-H1 and PD-1 by monoclonal antibodies potentiates cancer therapeutic immunity. , 2005, Cancer research.

[19]  E. Reiche,et al.  Stress and depression-induced immune dysfunction: Implications for the development and progression of cancer , 2005, International review of psychiatry.

[20]  Edna Maria Vissoci Reiche,et al.  Stress, depression, the immune system, and cancer. , 2004, The Lancet. Oncology.

[21]  Lieping Chen.  Co-inhibitory molecules of the B7–CD28 family in the control of T-cell immunity , 2004, Nature Reviews Immunology.

[22]  J. Cheville,et al.  B7-H1 blockade augments adoptive T-cell immunotherapy for squamous cell carcinoma. , 2003, Cancer research.

[23]  T. Curiel,et al.  Blockade of B7-H1 improves myeloid dendritic cell–mediated antitumor immunity , 2003, Nature Medicine.

[24]  G. Freeman,et al.  Blockade of Programmed Death-1 Ligands on Dendritic Cells Enhances T Cell Activation and Cytokine Production 1 , 2003, The Journal of Immunology.

[25]  B. Pützer,et al.  Improved treatment of pancreatic cancer by IL-12 and B7.1 costimulation: antitumor efficacy and immunoregulation in a nonimmunogenic tumor model. , 2002, Molecular therapy : the journal of the American Society of Gene Therapy.

[26]  H. Friess,et al.  Molecular Mechanisms in Chronic Pancreatitis , 2001, Zentralblatt fur Chirurgie.

[27]  R. Lindeman,et al.  CD80 (B7-1) expression on human acute myeloid leukaemic cells cultured with GM-CSF, IL-3 and IL-6 , 2001, Cancer Immunology, Immunotherapy.

[28]  J. Miller,et al.  B7-1 expression by a non-antigen presenting cell-derived tumor. , 1995, Cancer research.

[29]  T. Nagakawa,et al.  A clinicopathologic study on neural invasion in cancer of the pancreatic head , 1992, Cancer.

[30]  D. Casarini,et al.  Diabetes induces changes of catecholamines in primary mesangial cells. , 2008, The international journal of biochemistry & cell biology.

[31]  G. Zhu,et al.  Tumor-associated B7-H1 promotes T-cell apoptosis: A potential mechanism of immune evasion , 2002, Nature Medicine.

[32]  Chen Mei-zhen.  IMMUNOCYTOCHEMISTRY STUDY OF B7.1 IN SIX TUMOR CELL LINES , 2000 .