Anoctamin 6 differs from VRAC and VSOAC but is involved in apoptosis and supports volume regulation in the presence of Ca2+
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E. Hoffmann | I. H. Lambert | E. Larsen | K. Poulsen | S. Grubb | C. A. Juul | T. Kyed | N. Hashem
[1] B. Nilius,et al. Molecular functions of anoctamin 6 (TMEM16F): a chloride channel, cation channel, or phospholipid scramblase? , 2014, Pflügers Archiv - European Journal of Physiology.
[2] Anant K. Menon,et al. Ca2+-dependent phospholipid scrambling by a reconstituted TMEM16 ion channel , 2013, Nature Communications.
[3] D. Sauter,et al. The role of TMEM16A (ANO1) and TMEM16F (ANO6) in cell migration , 2013, Pflügers Archiv - European Journal of Physiology.
[4] C. I. Zeeuw,et al. Slc26a11 is prominently expressed in the brain and functions as a chloride channel: expression in Purkinje cells and stimulation of V H+-ATPase , 2013, Pflügers Archiv - European Journal of Physiology.
[5] E. Hoffmann,et al. TMEM16F (Anoctamin 6), an anion channel of delayed Ca2+ activation , 2013, The Journal of general physiology.
[6] Takahiro Shimizu,et al. TMEM16F is a component of a Ca2+-activated Cl- channel but not a volume-sensitive outwardly rectifying Cl- channel. , 2013, American journal of physiology. Cell physiology.
[7] Uwe Kornak,et al. Inactivation of anoctamin‐6/Tmem16f, a regulator of phosphatidylserine scrambling in osteoblasts, leads to decreased mineral deposition in skeletal tissues , 2013, Journal of bone and mineral research : the official journal of the American Society for Bone and Mineral Research.
[8] Y. Jan,et al. TMEM16F Forms a Ca2+-Activated Cation Channel Required for Lipid Scrambling in Platelets during Blood Coagulation , 2012, Cell.
[9] F. Lang,et al. Role of ion transport in control of apoptotic cell death. , 2012, Comprehensive Physiology.
[10] Fan Wang,et al. The calcium-activated chloride channel anoctamin 1 acts as a heat sensor in nociceptive neurons , 2012, Nature Neuroscience.
[11] P. Kongsuphol,et al. Expression and function of epithelial anoctamins , 2012, Experimental physiology.
[12] I. H. Lambert,et al. Regulation of Taurine Transport Systems by Protein Kinase CK2 in Mammalian Cells , 2011, Cellular Physiology and Biochemistry.
[13] P. Kongsuphol,et al. Anoctamin 6 is an essential component of the outwardly rectifying chloride channel , 2011, Proceedings of the National Academy of Sciences.
[14] H. C. Hartzell,et al. Physiological roles and diseases of tmem16/anoctamin proteins: are they all chloride channels? , 2011, Acta Pharmacologica Sinica.
[15] E. Hoffmann,et al. Pinpointing Differences in Cisplatin-induced Apoptosis in Adherent and Non-adherent Cancer Cells , 2011, Cellular Physiology and Biochemistry.
[16] S. Nagata,et al. Calcium-dependent phospholipid scrambling by TMEM16F , 2010, Nature.
[17] J. Tazi,et al. Exon-based clustering of murine breast tumor transcriptomes reveals alternative exons whose expression is associated with metastasis. , 2010, Cancer research.
[18] B. Harfe,et al. TMEM16 Proteins Produce Volume-regulated Chloride Currents That Are Reduced in Mice Lacking TMEM16A* , 2009, The Journal of Biological Chemistry.
[19] A. Menini,et al. TMEM16B induces chloride currents activated by calcium in mammalian cells , 2009, Pflügers Archiv - European Journal of Physiology.
[20] Min Ho Tak,et al. TMEM16A confers receptor-activated calcium-dependent chloride conductance , 2008, Nature.
[21] Roberto Ravazzolo,et al. TMEM16A, A Membrane Protein Associated with Calcium-Dependent Chloride Channel Activity , 2008, Science.
[22] Yuh Nung Jan,et al. Expression Cloning of TMEM16A as a Calcium-Activated Chloride Channel Subunit , 2008, Cell.
[23] H. Sontheimer,et al. Cytoplasmic condensation is both necessary and sufficient to induce apoptotic cell death , 2008, Journal of Cell Science.
[24] C. Hougaard,et al. Cell cycle‐dependent activity of the volume‐ and Ca2+‐activated anion currents in Ehrlich lettre ascites cells , 2007, Journal of cellular physiology.
[25] E. Hoffmann,et al. Cell shrinkage as a signal to apoptosis in NIH 3T3 fibroblasts , 2005, The Journal of physiology.
[26] B. Nilius,et al. Amazing chloride channels: an overview. , 2003, Acta physiologica Scandinavica.
[27] Y. Okada,et al. Apoptosis, cell volume regulation and volume-regulatory chloride channels. , 2001, Comparative biochemistry and physiology. Part A, Molecular & integrative physiology.
[28] Y Ishizaki,et al. Normotonic cell shrinkage because of disordered volume regulation is an early prerequisite to apoptosis. , 2000, Proceedings of the National Academy of Sciences of the United States of America.
[29] B. Nilius,et al. Separate Swelling- and Ca2+-activated Anion Currents in Ehrlich Ascites Tumor Cells , 1998, The Journal of Membrane Biology.
[30] E. Hoffmann,et al. Swelling-activated potassium currents of Ehrlich ascites tumour cells. , 1998, Biochimica et biophysica acta.
[31] A. Brown,et al. On the Role of Calcium in the Regulatory Volume Decrease (RVD) Response in Ehrlich Mouse Ascites Tumor Cells , 1997, The Journal of Membrane Biology.
[32] E. Hoffmann,et al. Amino acid transport and cell volume regulation in Ehrlich ascites tumour cells. , 1983, The Journal of physiology.
[33] A. Wyllie,et al. Apoptosis: A Basic Biological Phenomenon with Wide-ranging Implications in Tissue Kinetics , 1972, British Journal of Cancer.
[34] C. Hougaard,et al. Deregulation of apoptotic volume decrease and ionic movements in multidrug-resistant tumor cells: role of chloride channels. , 2010, American journal of physiology. Cell physiology.
[35] E. Hoffmann,et al. Physiology of cell volume regulation in vertebrates. , 2009, Physiological reviews.
[36] I. H. Lambert. Regulation of the Cellular Content of the Organic Osmolyte Taurine in Mammalian Cells , 2004, Neurochemical Research.
[37] E. Hoffmann,et al. TMEM 16 F ( Anoctamin 6 ) , an anion channel of delayed Ca 2 + activation , 2022 .
[38] Pierre Hilson,et al. TMEM 16 A , A Membrane Protein Associated with Calcium-Dependent Chloride Channel Activity , 2022 .