Dose-dependent lesions of the dopaminergic nigrostriatal pathway induced by instrastriatal injection of 6-hydroxydopamine
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D. Togasaki | D. Donaldson | V. Jackson-Lewis | H. Jiang | H. Jiang | S. Przedbroski | M. Leviver | M. Ferreira | V. Jackson-Lewis | D. Donaldson | D. M. Togasaki | M. Ferreira | Hong Jiang | Mayra Ferreira | S. Przedbroski | M. Leviver
[1] H. Robertson,et al. Dopamine receptor interactions: some implications for the treatment of Parkinson's disease , 1992, Trends in Neurosciences.
[2] S. Snyder,et al. Differential visualization of dopamine and norepinephrine uptake sites in rat brain using [3H]mazindol autoradiography , 1985, The Journal of neuroscience : the official journal of the Society for Neuroscience.
[3] Greg A. Gerhardt,et al. Correlation of apomorphine- and amphetamine-induced turning with nigrostriatal dopamine content in unilateral 6-hydroxydopamine lesioned rats , 1993, Brain Research.
[4] B. Rosen,et al. 1‐Methyl‐4‐Phenylpyridinium Produces Excitotoxic Lesions in Rat Striatum as a Result of Impairment of Oxidative Metabolism , 1992, Journal of neurochemistry.
[5] M. Kilbourn,et al. Repeated reserpine administration reduces in vivo [18F]GBR 13119 binding to the dopamine uptake site. , 1992, European journal of pharmacology.
[6] C. Marsden,et al. New insights into the cause of Parkinson's disease , 1992, Neurology.
[7] C. Pycock. Turning behaviour in animals , 1980, Neuroscience.
[8] A. Herbet,et al. Specificity of dopaminergic neuronal degeneration induced by intracerebral injection of 6-hydroxydopamine in the nigrostriatal dopamine system , 1976, Brain Research.
[9] M. Verrier,et al. MPTP produces reversible disappearance of tyrosine hydroxylase-containing retinal amacrine cells , 1990, Brain Research.
[10] R. H. Stellwagen,et al. Slow changes of tyrosine hydroxylase gene expression in dopaminergic brain neurons after neurotoxin lesioning: a model for neuron aging. , 1992, Brain research. Molecular brain research.
[11] Y. Agid,et al. Tyrosine hydroxylase protein and messenger RNA in the dopaminergic nigral neurons of patients with Parkinson's disease , 1993, Brain Research.
[12] C. Gerfen. The neostriatal mosaic: multiple levels of compartmental organization in the basal ganglia. , 1992, Annual review of neuroscience.
[13] J. Cadet,et al. Effect of Unilateral Perinatal Hypoxic‐Ischemic Brain Injury in the Rat on Dopamine D1 and D2 Receptors and Uptake Sites: A Quantitative Autoradiographic Study , 1991, Journal of neurochemistry.
[14] K. S. Bankiewicz,et al. A 6-hydroxydopamine-induced selective parkinsonian rat model , 1989, Brain Research.
[15] W. Tatton,et al. Dose-dependent destruction of the coeruleus-cortical and nigral-striatal projections by MPTP , 1990, Brain Research.
[16] E. Hirsch,et al. Decreased tyrosine hydroxylase messenger RNA in the surviving dopamine neurons of the substantia nigra in parkinson's disease: An in situ hybridization study , 1990, Neuroscience.
[17] A. D. Smith,et al. The neural network of the basal ganglia as revealed by the study of synaptic connections of identified neurones , 1990, Trends in Neurosciences.
[18] T. Joh,et al. Transneuronal regulation of tyrosine hydroxylase expression in olfactory bulb of mouse and rat , 1983, The Journal of neuroscience : the official journal of the Society for Neuroscience.
[19] B. Hyman,et al. Age‐Dependent Striatal Excitotoxic Lesions Produced by the Endogenous Mitochondrial Inhibitor Malonate , 1993, Journal of neurochemistry.
[20] Anders Björklund. Neural transplantation — an experimental tool with clinical possibilities , 1991, Trends in Neurosciences.
[21] E. Richfield,et al. Quantitative autoradiography of [3H]sulpiride binding sites in rat brain , 1984, Neuroscience Letters.
[22] G. Wooten,et al. Differential localization of A2a adenosine receptor mRNA with D1 and D2 dopamine receptor mRNA in striatal output pathways following a selective lesion of striatonigral neurons , 1993, Brain Research.
[23] Marian DiFiglia,et al. Excitotoxic injury of the neostriatum: a model for Huntington's disease , 1990, Trends in Neurosciences.
[24] M. Dubocovich,et al. Binding characteristics of the dopamine uptake inhibitor [3H]nomifensine to striatal membranes. , 1985, Biochemical pharmacology.
[25] M. Abercrombie. Estimation of nuclear population from microtome sections , 1946, The Anatomical record.
[26] R. Burke,et al. Naturally Occurring Cell Death during Postnatal Development of the Substantia Nigra Pars Compacta of Rat , 1993, Molecular and Cellular Neuroscience.
[27] U. Ungerstedt,et al. Quantitative recording of rotational behavior in rats after 6-hydroxy-dopamine lesions of the nigrostriatal dopamine system. , 1970, Brain research.
[28] R. Porter,et al. Inhibition of Succinate Dehydrogenase by Malonic Acid Produces an “Excitotoxic” Lesion in Rat Striatum , 1993, Journal of neurochemistry.
[29] R. Wurtman,et al. Partial lesions of the dopaminergic nigrostriatal system in rat brain: biochemical characterization , 1980, Brain Research.
[30] G. Paxinos,et al. The Rat Brain in Stereotaxic Coordinates , 1983 .
[31] C. Finch,et al. Chronic lesions differentially decrease tyrosine hydroxylase messenger RNA in dopaminergic neurons of the substantia nigra. , 1989, Brain research. Molecular brain research.
[32] J. Obeso,et al. Decreased tyrosine hydroxylase content in the dopaminergic neurons of MPTP–intoxicated monkeys: Effect of levodopa and GM1 ganglioside therapy , 1994, Annals of neurology.
[33] U. Muthane,et al. Differences in Nigral Neuron Number and Sensitivity to 1-Methyl-4-phenyl-1,2,3,6-tetrahydropyridine in C57/bl and CD-1 Mice , 1994, Experimental Neurology.
[34] R. Beninger,et al. On the interpretation of asymmetries of posture and locomotion produced with dopamine agonists in animals with unilateral depletion of striatal dopamine , 1991, Progress in Neurobiology.
[35] R. Baldessarini,et al. Lack of increase in dopamine transporter binding or function in rat brain tissue after treatment with blockers of neuronal uptake of dopamine , 1991, Neuropharmacology.
[36] W. Weiner,et al. 1-Methyl-4-phenylpyridinium (MPP+) but not 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine (MPTP) selectively destroys dopaminergic neurons in cultures of dissociated rat mesencephalic neurons , 1986, Neuroscience Letters.
[37] E. Eidelberg,et al. Injury of Catecholaminergic Neurons after Acute Exposure to MPTP , 1987 .
[38] W. Turski,et al. Aminooxyacetic acid produces excitotoxic lesions in the rat striatum , 1991, Synapse.
[39] D. Gash,et al. Effect of bilateral 6-OHDA lesions of the substantia nigra on locomotor activity in the rat , 1994, Brain Research.
[40] M. Sofroniew,et al. Loss of transmitter-associated enzyme staining following axotomy does not indicate death of brainstem cholinergic neurons , 1988, Brain Research.
[41] R. Wise,et al. Retrograde fluorescent tracing of substantia nigra neurons combined with catecholamine histofluorescence , 1980, Brain Research.
[42] U. Ungerstedt. Striatal dopamine release after amphetamine or nerve degeneration revealed by rotational behaviour. , 1971, Acta physiologica Scandinavica. Supplementum.
[43] Stephen J. Fink,et al. Colocalization of D1 and D2 dopamine receptor mRNAs in striatal neurons , 1993, Brain Research.
[44] T. H. Burnstine,et al. Movement Disorders 2 , 1988, Neurology.
[45] B. Parsons,et al. Adenosine receptor antagonists potentiate dopamine receptor agonist-induced rotational behavior in 6-hydroxydopamine-lesioned rats , 1993, Brain Research.
[46] J. Cadet,et al. Long-term behavioral and biochemical effects of 6-hydroxydopamine injections in rat caudate-putamen , 1991, Brain Research Bulletin.
[47] J. Palacios,et al. Adenosine A2 receptors: Selective localization in the human basal ganglia and alterations with disease , 1991, Neuroscience.
[48] Fred H. Gage,et al. Partial lesion of the substantia nigra: relation between extent of lesion and rotational behavior , 1991, Brain Research.
[49] R. Albin,et al. Alternative excitotoxic hypotheses , 1992, Neurology.
[50] M. Törnwall,et al. Effects of three types of catechol O-methylation inhibitors on L-3,4-dihydroxyphenylalanine-induced circling behaviour in rats. , 1993, European journal of pharmacology.
[51] M. Beal,et al. Does impairment of energy metabolism result in excitotoxic neuronal death in neurodegenerative illnesses? , 1992, Annals of neurology.
[52] W. Tatton,et al. Transmitter synthesis increases in substantia nigra neurons of the aged mouse , 1991, Neuroscience Letters.
[53] T. Ho¨kfelt,et al. Retrograde peroxidase tracing of neurons combined with transmitter histochemistry , 1975, Brain Research.
[54] J. Cedarbaum,et al. The Therapeutic Potential of Neurotrophic Factors in the Treatment of Parkinson's Disease , 1993, Experimental Neurology.
[55] Charles Watson,et al. Bregma, lambda and the interaural midpoint in stereotaxic surgery with rats of different sex, strain and weight , 1985, Journal of Neuroscience Methods.
[56] Y. Agid,et al. Immunocytochemical Quantification of Tyrosine Hydroxylase at a Cellular Level in the Mesencephalon of Control Subjects and Patients with Parkinson's and Alzheimer's Disease , 1993, Journal of neurochemistry.
[57] R. Burke,et al. Effect of striatal lesion with quinolinate on the development of substantia nigra dopaminergic neurons: a quantitative morphological analysis. , 1992, Developmental neuroscience.
[58] J. Sladek,et al. Dopamine cell replacement: Parkinson's disease. , 1990, Annual review of neuroscience.
[59] J. Cadet,et al. Retrograde degeneration of nigrostriatal neurons induced by intrastriatal 6-hydroxydopamine injection in rats , 1991, Brain Research Bulletin.
[60] D. Reis,et al. Reversible changes in the activities and amounts of tyrosine hydroxylase in dopamine neurons of the substantia nigra in response to axonal injury as studied by immunochemical and immunocytochemical methods , 1978, Brain Research.
[61] P. Seeman,et al. The Dopamine Transporter Is Absent in Parkinsonian Putamen and Reduced in the Caudate Nucleus , 1991, Journal of neurochemistry.
[62] U. McCann,et al. Neurotoxic Amphetamine Analogues: Effects in Monkeys and Implications for Humans a , 1992, Annals of the New York Academy of Sciences.
[63] M. Kuhar,et al. Quantitative receptor mapping by autoradiography: some current technical problems , 1985, Trends in Neurosciences.
[64] W. H. Oertel,et al. Progressive degeneration of nigrostriatal dopamine neurons following intrastriatal terminal lesions with 6-hydroxydopamine: A combined retrograde tracing and immunocytochemical study in the rat , 1994, Neuroscience.
[65] M. Williams,et al. Direct autoradiographic localization of adenosine A2 receptors in the rat brain using the A2-selective agonist, [3H]CGS 21680. , 1989, European journal of pharmacology.
[66] T. Hattori,et al. Is there a non-dopaminergic nigrostriatal pathway? , 1981, Neuroscience.
[67] T. Stone. Handbook of chemical neuroanatomy vol. 1: Methods in chemical neuroanatomy: edited by A. Björklund and T. Hökfelt, Elsevier Science Publishers, 1983. Dfl 330.00/US$140.50 (xxvi + 548 pages) ISBN 0 444 90281 3 , 1984 .
[68] R. Burke,et al. Neonatal hypoxic-ischemic or excitotoxic striatal injury results in a decreased adult number of substantia nigra neurons , 1992, Neuroscience.
[69] H. Teräväinen,et al. Animal models of parkinsonism. , 1990, Pharmacology & toxicology.
[70] U. Ungerstedt,et al. Supersensitivity to apomorphine following destruction of the ascending dopamine neurons: quantification using the rotational model. , 1977, European journal of pharmacology.
[71] S. Snyder,et al. Dopamine receptor binding enhancement accompanies lesion-induced behavioral supersensitivity. , 1977, Science.
[72] B. Hyman,et al. Aminooxyacetic Acid Results in Excitotoxin Lesions by a Novel Indirect Mechanism , 1991, Journal of neurochemistry.
[73] W. Weiner,et al. Selective Destruction of Cultured Dopaminergic Neurons from Fetal Rat Mesencephalon by 1‐Methyl‐4‐Phenylpyridinium: Cytochemical and Morphological Evidence , 1988, Journal of neurochemistry.
[74] C. D. Stern,et al. Handbook of Chemical Neuroanatomy Methods in Chemical Neuroanatomy. Edited by A. Bjorklund and T. Hokfelt. Elsevier, Amsterdam, 1983. Cloth bound, 548 pp. UK £140. (Volume 1 in the series). , 1986, Neurochemistry International.
[75] J. Cadet,et al. Sham transplantation protects against 6-hydroxydopamine-induced dopaminergic toxicity in rats: behavioral and morphological evidence , 1991, Brain Research.