Phospholipid scramblase: An update

[1]  J. Mezey,et al.  A genome-wide linkage scan in German shepherd dogs localizes canine platelet procoagulant deficiency (Scott syndrome) to canine chromosome 27. , 2010, Gene.

[2]  A. Arachiche,et al.  Rapid Procoagulant Phosphatidylserine Exposure Relies on High Cytosolic Calcium Rather Than on Mitochondrial Depolarization , 2009, Arteriosclerosis, thrombosis, and vascular biology.

[3]  K. J. Henley,et al.  Two distinct pathways regulate platelet phosphatidylserine exposure and procoagulant function. , 2009, Blood.

[4]  Florian Lang,et al.  Erythrocyte programmed cell death , 2008, IUBMB life.

[5]  J. Molkentin,et al.  Critical role for the mitochondrial permeability transition pore and cyclophilin D in platelet activation and thrombosis. , 2008, Blood.

[6]  K. Ravichandran,et al.  Engulfment of apoptotic cells: signals for a good meal , 2007, Nature Reviews Immunology.

[7]  G. Dale,et al.  Scott syndrome dogs have impaired coated‐platelet formation and calcein‐release but normal mitochondrial depolarization , 2007, Journal of thrombosis and haemostasis : JTH.

[8]  P. Williamson,et al.  Transbilayer Phospholipid Movements in ABCA1-Deficient Cells , 2007, PloS one.

[9]  R. Schlegel,et al.  Aminophospholipid Translocase TAT-1 Promotes Phosphatidylserine Exposure during C. elegans Apoptosis , 2007, Current Biology.

[10]  S. Gummadi,et al.  Phospholipid scramblases: an overview. , 2007, Archives of biochemistry and biophysics.

[11]  G. Chimini,et al.  ABCA1, from pathology to membrane function , 2007, Pflügers Archiv - European Journal of Physiology.

[12]  G. Dale,et al.  Bax activators potentiate coated‐platelet formation , 2006, Journal of thrombosis and haemostasis : JTH.

[13]  C. Higgins,et al.  Phosphatidylserine exposure in B lymphocytes: a role for lipid packing. , 2006, Blood.

[14]  B. Huppertz,et al.  Trophoblast fusion: fusogenic proteins, syncytins and ADAMs, and other prerequisites for syncytial fusion. , 2006, Micron.

[15]  F. Kuypers,et al.  Sulphydryl modifications alter scramblase activity in murine sickle cell disease , 2006, British journal of haematology.

[16]  J. Acharya,et al.  Drosophila melanogaster Scramblases modulate synaptic transmission , 2006, The Journal of cell biology.

[17]  M. Barcinski,et al.  Mimicry of Apoptotic Cells by Exposing Phosphatidylserine Participates in the Establishment of Amastigotes of Leishmania (L) amazonensis in Mammalian Hosts1 , 2006, The Journal of Immunology.

[18]  C. Higgins,et al.  A novel missense mutation in ABCA1 results in altered protein trafficking and reduced phosphatidylserine translocation in a patient with Scott syndrome. , 2005, Blood.

[19]  J. Heemskerk,et al.  Synergistic Effect of Thrombin on Collagen-Induced Platelet Procoagulant Activity Is Mediated Through Protease-Activated Receptor-1 , 2005, Arteriosclerosis, thrombosis, and vascular biology.

[20]  J. Rasmussen,et al.  Activated scramblase and inhibited aminophospholipid translocase cause phosphatidylserine exposure in a distinct platelet fraction , 2005, Cellular and Molecular Life Sciences CMLS.

[21]  B. Gadella,et al.  Bicarbonate-induced membrane processing in sperm capacitation. , 2005, Theriogenology.

[22]  R. Szász,et al.  Role of Mitochondrial Permeability Transition Pore in Coated-Platelet Formation , 2004, Arteriosclerosis, thrombosis, and vascular biology.

[23]  G. Assmann,et al.  Impaired Platelet Activation in Familial High Density Lipoprotein Deficiency (Tangier Disease)* , 2004, Journal of Biological Chemistry.

[24]  P. Comfurius,et al.  Scott syndrome, a bleeding disorder caused by defective scrambling of membrane phospholipids. , 2004, Biochimica et biophysica acta.

[25]  J. Killian,et al.  Transbilayer movement of phospholipids in biogenic membranes. , 2004, Biochemistry.

[26]  R. Schlegel,et al.  Phosphatidylserine on HIV Envelope Is a Cofactor for Infection of Monocytic Cells1 , 2003, The Journal of Immunology.

[27]  J. Heemskerk,et al.  Store-mediated calcium entry in the regulation of phosphatidylserine exposure in blood cells from Scott patients , 2003, Thrombosis and Haemostasis.

[28]  P. Sims,et al.  Normal hemostasis but defective hematopoietic response to growth factors in mice deficient in phospholipid scramblase 1. , 2002, Blood.

[29]  H. Brown,et al.  A hereditary bleeding disorder of dogs caused by a lack of platelet procoagulant activity. , 2002, Blood.

[30]  D. Dekkers,et al.  Comparison between Ca2+-induced scrambling of various fluorescently labelled lipid analogues in red blood cells. , 2002, The Biochemical journal.

[31]  C. Reutelingsperger,et al.  Transient expression of phosphatidylserine at cell-cell contact areas is required for myotube formation. , 2001, Journal of cell science.

[32]  P. Williamson,et al.  Phospholipid scramblase activation pathways in lymphocytes. , 2001, Biochemistry.

[33]  P. Williamson,et al.  Phosphatidylserine, a death knell , 2001, Cell Death and Differentiation.

[34]  Yannick Hamon,et al.  ABC1 promotes engulfment of apoptotic cells and transbilayer redistribution of phosphatidylserine. , 2000, Nature Cell Biology.

[35]  R. Silverman,et al.  Transcriptional control of the human plasma membranephospholipid scramblase 1 gene is mediated by interferon-α , 2000 .

[36]  S. Orrenius,et al.  Phosphatidylserine exposure during apoptosis is a cell-type-specific event and does not correlate with plasma membrane phospholipid scramblase expression. , 1999, Biochemical and biophysical research communications.

[37]  E. Kable,et al.  Ca2+ sensitivity of phospholipid scrambling in human red cell ghosts. , 1999, Cell calcium.

[38]  M. Raff,et al.  Are caspases involved in the death of cells with a transcriptionally inactive nucleus? Sperm and chicken erythrocytes. , 1998, Journal of cell science.

[39]  D. Dekkers,et al.  Transmembrane phospholipid distribution in blood cells: control mechanisms and pathophysiological significance. , 1998, Biological chemistry.

[40]  P. Sims,et al.  Level of Expression of Phospholipid Scramblase Regulates Induced Movement of Phosphatidylserine to the Cell Surface* , 1998, The Journal of Biological Chemistry.

[41]  V. Fadok,et al.  Appearance of Phosphatidylserine on Apoptotic Cells Requires Calcium-mediated Nonspecific Flip-Flop and Is Enhanced by Loss of the Aminophospholipid Translocase* , 1997, The Journal of Biological Chemistry.

[42]  C. Haest,et al.  Transbilayer reorientation of phospholipid probes in the human erythrocyte membrane. Lessons from studies on electroporated and resealed cells. , 1997, Biochimica et biophysica acta.

[43]  S. Orrenius,et al.  Involvement of extracellular calcium in phosphatidylserine exposure during apoptosis , 1996, FEBS letters.

[44]  P. Sims,et al.  Isolation of an Erythrocyte Membrane Protein that Mediates Ca2+-dependent Transbilayer Movement of Phospholipid* , 1996, The Journal of Biological Chemistry.

[45]  P. Williamson,et al.  Reconstitution of phospholipid scramblase activity from human blood platelets. , 1996, Biochemistry.

[46]  R. Schlegel,et al.  A Subfamily of P-Type ATPases with Aminophospholipid Transporting Activity , 1996, Science.

[47]  J. Freyssinet,et al.  Scott syndrome, characterized by impaired transmembrane migration of procoagulant phosphatidylserine and hemorrhagic complications, is an inherited disorder. , 1996, Blood.

[48]  P. Williamson,et al.  Mechanisms of phosphatidylserine exposure, a phagocyte recognition signal, on apoptotic T lymphocytes , 1995, The Journal of experimental medicine.

[49]  P. Williamson,et al.  Continuous analysis of the mechanism of activated transbilayer lipid movement in platelets. , 1995, Biochemistry.

[50]  Seamus J. Martin,et al.  Protease activation during apoptosis: Death by a thousand cuts? , 1995, Cell.

[51]  P. Comfurius,et al.  Calcium-induced transbilayer scrambling of fluorescent phospholipid analogs in platelets and erythrocytes. , 1994, Biochimica et biophysica acta.

[52]  F. Basse,et al.  Translocation of spin-labeled phospholipids through plasma membrane during thrombin- and ionophore A23187-induced platelet activation. , 1993, Biochemistry.

[53]  P. Williamson,et al.  Ca2+ induces transbilayer redistribution of all major phospholipids in human erythrocytes. , 1992, Biochemistry.

[54]  V. Fadok,et al.  Exposure of phosphatidylserine on the surface of apoptotic lymphocytes triggers specific recognition and removal by macrophages. , 1992, Journal of immunology.

[55]  P. Williamson,et al.  Rapid loss and restoration of lipid asymmetry by different pathways in resealed erythrocyte ghosts. , 1992, Biochimica et biophysica acta.

[56]  P. Henson,et al.  A model for the extracellular release of PAF: the influence of plasma membrane phospholipid asymmetry. , 1991, Biochimica et biophysica acta.

[57]  C. Haest,et al.  Effects of divalent cations on lipid flip-flop in the human erythrocyte membrane. , 1990, Biochimica et biophysica acta.

[58]  A. Schroit,et al.  Loss of membrane phospholipid asymmetry in platelets and red cells may be associated with calcium-induced shedding of plasma membrane and inhibition of aminophospholipid translocase. , 1990, Biochimica et biophysica acta.

[59]  A. Visser,et al.  Bidirectional transbilayer lipid movement in human platelets as vizualized by the fluorescent membrane probe 1-[4-(trimethylammonio)phenyl]-6-phenyl-1,3,5-hexatriene. , 1990, Biochemistry.

[60]  P. Devaux,et al.  Ion regulation of phosphatidylserine and phosphatidylethanolamine outside-inside translocation in human erythrocytes. , 1987, Biochimica et biophysica acta.

[61]  P. Comfurius,et al.  Changes in membrane phospholipid distribution during platelet activation. , 1983, Biochimica et biophysica acta.

[62]  H. Weiss,et al.  Isolated deficiency of platelet procoagulant activity. , 1979, The American journal of medicine.

[63]  Junichi Nakai,et al.  Vaccinia Virus Uses Macropinocytosis and Apoptotic Mimicry to Enter Host Cells , 2008 .

[64]  R. Silverman,et al.  Transcriptional control of the human plasma membrane phospholipid scramblase 1 gene is mediated by interferon-alpha. , 2000, Blood.

[65]  J. Freyssinet,et al.  Annexin V as a probe of aminophospholipid exposure and platelet membrane vesiculation: a flow cytometry study showing a role for free sulfhydryl groups. , 1993, Blood.

[66]  P. Sims,et al.  Defective Ca(2+)-induced microvesiculation and deficient expression of procoagulant activity in erythrocytes from a patient with a bleeding disorder: a study of the red blood cells of Scott syndrome. , 1992, Blood.