Mechanisms of Glomerular Fibrin Deposition in Glomerulonephritis
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[1] R. Wiggins,et al. Tissue factor production by cultured rat mesangial cells. Stimulation by TNF alpha and lipopolysaccharide. , 1990, Kidney international.
[2] J. M. Boswell,et al. Increased tumor necrosis factor and IL-1 beta gene expression in the kidneys of mice with lupus nephritis. , 1988, Journal of immunology.
[3] P. Tipping,et al. Glomerular macrophages express augmented procoagulant activity in experimental fibrin-related glomerulonephritis in rabbits. , 1988, The Journal of clinical investigation.
[4] P. Tipping,et al. PARTICIPATION OF CELL-MEDIATED IMMUNITY IN DEPOSITION OF FIBRIN IN GLOMERULONEPHRITIS , 1988, The Lancet.
[5] R. Cotran. American Association of Pathologists president's address. New roles for the endothelium in inflammation and immunity. , 1987, The American journal of pathology.
[6] D. Nochy,et al. ENHANCED GLOMERULAR PROCOAGULANT ACTIVITY AND FIBRIN DEPOSITION IN RATS WITH MERCURIC CHLORIDE-INDUCED AUTOIMMUNE NEPHRITIS , 1987, Thrombosis and Haemostasis.
[7] J. Cameron,et al. Intraglomerular T cells and monocytes in nephritis: study with monoclonal antibodies. , 1987, Kidney international.
[8] C. Esmon. The regulation of natural anticoagulant pathways , 1987, Science.
[9] P. Tipping,et al. Quantitation and characterization of glomerular procoagulant activity in experimental glomerulonephritis. , 1987, Laboratory investigation; a journal of technical methods and pathology.
[10] P. Tipping,et al. A comparison of fibrinolytic and defibrinating agents in established experimental glomerulonephritis. , 1986, British journal of experimental pathology.
[11] M. Gimbrone,et al. Regulation of the fibrinolytic system of cultured human vascular endothelium by interleukin 1. , 1986, The Journal of clinical investigation.
[12] P. Tipping,et al. The participation of macrophages, glomerular procoagulant activity, and factor VIII in glomerular fibrin deposition. Studies on anti-GBM antibody-induced glomerulonephritis in rabbits. , 1986, The American journal of pathology.
[13] S. Gregory,et al. Tissue factor induction in human monocytes. Two distinct mechanisms displayed by different alloantigen-responsive T cell clones. , 1985, The Journal of clinical investigation.
[14] E. Génot,et al. Production of monoclonal antibodies to the high fibrin-affinity, tissue-type plasminogen activator of human plasma. Demonstration of its endothelial origin by immunolocalization. , 1985, Blood.
[15] P. Tipping,et al. Macrophage-induced glomerular fibrin deposition in experimental glomerulonephritis in the rabbit. , 1985, The Journal of clinical investigation.
[16] S. Bertoli,et al. The detection of monocytes in human glomerulonephritis. , 1985, Kidney international.
[17] R. Wiggins. Hageman factor in experimental nephrotoxic nephritis in the rabbit. , 1985, Laboratory investigation; a journal of technical methods and pathology.
[18] R. Ardaillou,et al. Lipoxygenase products mediate the attachment of rat macrophages to glomeruli in vitro. , 1985, Kidney international.
[19] P. Tipping,et al. T lymphocyte participation in antibody-induced experimental glomerulonephritis. , 1985, Kidney international.
[20] K. Tanaka,et al. Effects of fibrinogen degradation products on glomerular mesangial cells in culture. , 1984, Kidney international.
[21] T. Edgington,et al. Allogeneic induction of the human T cell-instructed monocyte procoagulant response is rapid and is elicited by HLA-DR , 1983, The Journal of experimental medicine.
[22] I. Stachura. Monocytes in human glomerulonephritis. , 1983, Clinical nephrology.
[23] Cameron Js. The role of platelets in glomerulonephritis. , 1983 .
[24] T. Naruse,et al. Effects of various antiplatelet drugs and defibrinating agent on experimental glomerulonephritis in rats. , 1982, The Journal of laboratory and clinical medicine.
[25] T. Edgington,et al. Immune complex-induced human monocyte procoagulant activity. I. a rapid unidirectional lymphocyte-instructed pathway , 1981, The Journal of experimental medicine.
[26] Magil Ab,et al. Monocytes in human glomerulonephritis. An electron microscopic study. , 1981 .
[27] J. Whitworth,et al. Hypercoagulation in glomerulonephritis. , 1981, British medical journal.
[28] F. Rickles,et al. Mononuclear cell tissue factor: cell of origin and requirements for activation , 1979 .
[29] A. Michael,et al. Renal cortical fibrinolytic activity in rabbits with chronic immune complex nephritis. , 1979, Israel journal of medical sciences.
[30] P. Verroust,et al. Glomerular fibrinolytic activity during nephrotoxic nephritis. , 1979, Laboratory investigation; a journal of technical methods and pathology.
[31] P. Naish,et al. Quantitative assessment of the effects of platelet depletion in the autologous phase of nephrotoxic serum nephritis. , 1979, Clinical and experimental immunology.
[32] G. Mazzucco,et al. The presence and possible role of monocyte infiltration in human chronic proliferative glomerulonephritides. Light microscopic, immunofluorescence, and histochemical correlations. , 1979, The American journal of pathology.
[33] J. Vaughan,et al. Leukocyte procoagulant activity: enhancement of production in vitro by IgG and antigen-antibody complexes. , 1977, The Journal of clinical investigation.
[34] R. Atkins,et al. THE MACROPHAGE IN HUMAN RAPIDLY PROGRESSIVE GLOMERULONEPHRITIS , 1976, The Lancet.
[35] D. Evans,et al. Defibrination with ancrod in experimental chronic immune complex nephritis , 1975 .
[36] D. Evans,et al. The effects of defibrination with ancrod in experimental allergic glomerular injury. , 1975, Clinical and experimental immunology.
[37] M. Pandolfi,et al. Origin of urinary fibrin/fibrinogen degradation products in glomerulonephritis. , 1975, British medical journal.
[38] A. Michael,et al. Immunofluorescent localization of antihemophilic factor antigen and fibrinogen in human renal diseases. , 1974, The Journal of clinical investigation.
[39] P. Kincaid‐smith. Coagulation and renal disease. , 1972, Kidney international.
[40] Michael Af,et al. Cortical fibrinolytic activity in normal and diseased human kidneys. , 1972 .
[41] D. Evans,et al. The effect of defibrination on nephrotoxic serum nephritis in rabbits. , 1972, Clinical science.
[42] J. Petrie,et al. Serum and Urinary Fibrin/Fibrinogen Degradation Products in Glomerulonephritis , 1971, British medical journal.
[43] R. Lohmann,et al. Nephrotic Syndrome: A Hypercoagulable State , 1971 .
[44] H. Kwaan,et al. The role of fibrinogen in renal disease. II. Effect of anticoagulants and urokinase on experimental lesions in mice. , 1969, The Journal of laboratory and clinical medicine.
[45] P. Vassalli,et al. An immunofluorescent study of pathogenic mechanisms in glomerular diseases. , 1966, The New England journal of medicine.
[46] P. Vassalli,et al. THE PATHOGENIC ROLE OF THE COAGULATION PROCESS IN RABBIT MASUGI NEPHRITIS. , 1964, The American journal of pathology.
[47] P. Tipping,et al. Glomerular interleukin 1 production is dependent on macrophage infiltration in anti-GBM glomerulonephritis. , 1991, Kidney international.
[48] E. Rondeau,et al. Glomerular hemostasis in normal and pathologic conditions. , 1988, Advances in nephrology from the Necker Hospital.
[49] P. Tipping,et al. Glomerular procoagulant activity in human proliferative glomerulonephritis. , 1988, The Journal of clinical investigation.
[50] P. Tipping,et al. Fibrinolytic therapy with streptokinase for established experimental glomerulonephritis. , 1986, Nephron.
[51] N. Vaziri. Nephrotic syndrome and coagulation and fibrinolytic abnormalities. , 1983, American journal of nephrology.
[52] K. Ham,et al. Macrophages and Glomerular Crescent Formation. Studies with Rat Nephrotoxic Nephritis , 1983, Pathology.
[53] P. Ward,et al. C5 chemotactic fragment induces leukocyte production of tissue factor activity: a link between complement and coagulation. , 1979, The Journal of clinical investigation.
[54] H. Coovadia,et al. Factor VIII procoagulant activity in children with nephrotic syndrome and post-streptococcal glomerulonephritis. , 1978, Nephron.
[55] G. Gabbiani,et al. Experimental focal glomerular lesions elicited by insoluble immune complexes. Ultrastructural and immunofluorescent studies. , 1975, Laboratory investigation; a journal of technical methods and pathology.