9-Phenanthrol and flufenamic acid inhibit calcium oscillations in HL-1 mouse cardiomyocytes.
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David L. Williams | D. Hoover | R. Wondergem | M. Gao | B. Graves | Chaunfu Li | S. Fregoso | Ying Li | G. Wright | R. Burt
[1] Yilin Tai,et al. Canonical transient receptor potential 3 channels regulate mitochondrial calcium uptake , 2013, Proceedings of the National Academy of Sciences.
[2] G. Guillemette,et al. STIM1 participates in the contractile rhythmicity of HL-1 cells by moderating T-type Ca(2+) channel activity. , 2013, Biochimica et biophysica acta.
[3] E. Lakatta,et al. Modern concepts concerning the origin of the heartbeat. , 2013, Physiology.
[4] S. Parajuli,et al. Novel role for the transient potential receptor melastatin 4 channel in guinea pig detrusor smooth muscle physiology. , 2013, American journal of physiology. Cell physiology.
[5] David L. Williams,et al. Lipopolysaccharide prolongs action potential duration in HL-1 mouse cardiomyocytes. , 2012, American journal of physiology. Cell physiology.
[6] J. Rougier,et al. TRPM4 channels in the cardiovascular system: physiology, pathophysiology, and pharmacology. , 2012, Biochemical pharmacology.
[7] David L. Williams,et al. Phosphoinositide-3-kinase/akt - dependent signaling is required for maintenance of [Ca2+]i,ICa, and Ca2+ transients in HL-1 cardiomyocytes , 2012, Journal of Biomedical Science.
[8] E. Lakatta,et al. Crosstalk between Mitochondrial and Sarcoplasmic Reticulum Ca2+ Cycling Modulates Cardiac Pacemaker Cell Automaticity , 2012, PloS one.
[9] R. Rouet,et al. Transient receptor potential melastatin 4 inhibitor 9‐phenanthrol abolishes arrhythmias induced by hypoxia and re‐oxygenation in mouse ventricle , 2012, British journal of pharmacology.
[10] Dario DiFrancesco,et al. The funny current has a major pacemaking role in the sinus node. , 2012, Heart rhythm.
[11] Edward G Lakatta,et al. The funny current in the context of the coupled-clock pacemaker cell system. , 2012, Heart rhythm.
[12] M. Wacker,et al. Store-operated calcium entry is present in HL-1 cardiomyocytes and contributes to resting calcium. , 2011, Biochemical and biophysical research communications.
[13] I. Ambudkar,et al. Faculty Opinions recommendation of A forty-kilodalton protein of the inner membrane is the mitochondrial calcium uniporter. , 2011 .
[14] K. Murray,et al. Ionic Mechanisms of Pacemaker Activity in Spontaneously Contracting Atrial HL-1 Cells , 2011, Journal of cardiovascular pharmacology.
[15] S. Earley,et al. Pharmacological inhibition of TRPM4 hyperpolarizes vascular smooth muscle. , 2010, American journal of physiology. Cell physiology.
[16] David L. Williams,et al. Lipopolysaccharides directly decrease Ca2+ oscillations and the hyperpolarization-activated nonselective cation current If in immortalized HL-1 cardiomyocytes. , 2010, American journal of physiology. Cell physiology.
[17] S. Earley,et al. Ca2+ release from the sarcoplasmic reticulum is required for sustained TRPM4 activity in cerebral artery smooth muscle cells. , 2010, American journal of physiology. Cell physiology.
[18] Edward G Lakatta,et al. A coupled SYSTEM of intracellular Ca2+ clocks and surface membrane voltage clocks controls the timekeeping mechanism of the heart's pacemaker. , 2010, Circulation research.
[19] Dario DiFrancesco,et al. Cycling in the Mechanism of Pacemaking Cardiac Pacemaking : Historical Overview and Future Directions , 2010 .
[20] V. Shoshan-Barmatz,et al. NCLX is an essential component of mitochondrial Na+/Ca2+ exchange , 2009, Proceedings of the National Academy of Sciences.
[21] Ronald Wilders,et al. Pacemaker activity of the human sinoatrial node: role of the hyperpolarization-activated current, I(f). , 2009, International journal of cardiology.
[22] P. Launay,et al. 9‐Phenanthrol inhibits human TRPM4 but not TRPM5 cationic channels , 2008, British journal of pharmacology.
[23] P. Launay,et al. TRPM4, a Ca2+-activated nonselective cation channel in mouse sino-atrial node cells. , 2007, Cardiovascular research.
[24] D. Clapham,et al. An introduction to TRP channels. , 2006, Annual review of physiology.
[25] E. Lakatta,et al. Rhythmic Ca2+ Oscillations Drive Sinoatrial Nodal Cell Pacemaker Function to Make the Heart Tick , 2005, Annals of the New York Academy of Sciences.
[26] R. Vennekens,et al. Comparison of functional properties of the Ca2+-activated cation channels TRPM4 and TRPM5 from mice. , 2005, Cell calcium.
[27] M. Zhu,et al. Regulation of the Ca2+ Sensitivity of the Nonselective Cation Channel TRPM4* , 2005, Journal of Biological Chemistry.
[28] S. Earley,et al. Critical Role for Transient Receptor Potential Channel TRPM4 in Myogenic Constriction of Cerebral Arteries , 2004, Circulation research.
[29] P. Bois,et al. Functional characterization of a Ca2+‐activated non‐selective cation channel in human atrial cardiomyocytes , 2004, The Journal of physiology.
[30] W. Claycomb,et al. Cardiac physiology at the cellular level: use of cultured HL-1 cardiomyocytes for studies of cardiac muscle cell structure and function. , 2004, American journal of physiology. Heart and circulatory physiology.
[31] David L. Williams,et al. Modulating Toll-like receptor mediated signaling by (1-->3)-beta-D-glucan rapidly induces cardioprotection. , 2004, Cardiovascular research.
[32] David E. Clapham,et al. TRP channels as cellular sensors , 2003, Nature.
[33] M. Freichel,et al. Voltage Dependence of the Ca2+-activated Cation Channel TRPM4* , 2003, Journal of Biological Chemistry.
[34] M. Murakami,et al. Identification and characterization of the murine TRPM4 channel. , 2003, Biochemical and biophysical research communications.
[35] N. Ryba,et al. Coding of Sweet, Bitter, and Umami Tastes Different Receptor Cells Sharing Similar Signaling Pathways , 2003, Cell.
[36] Elisabetta Cerbai,et al. Functional expression of the hyperpolarization‐activated, non‐selective cation current If in immortalized HL‐1 cardiomyocytes , 2002, The Journal of physiology.
[37] Cori Bargmann,et al. Social feeding in Caenorhabditis elegans is induced by neurons that detect aversive stimuli , 2002, Nature.
[38] J. Lenfant,et al. Characterization of a Ca2+-activated Nonselective Cation Channel during Dedifferentiation of Cultured Rat Ventricular Cardiomyocytes , 2002, The Journal of Membrane Biology.
[39] A. Perraud,et al. TRPM4 Is a Ca2+-Activated Nonselective Cation Channel Mediating Cell Membrane Depolarization , 2002, Cell.
[40] D. Bers. Cardiac excitation–contraction coupling , 2002, Nature.
[41] M. Cyert,et al. Internal Ca2+ release in yeast is triggered by hypertonic shock and mediated by a TRP channel homologue , 2002, The Journal of cell biology.
[42] G. Crabtree. Calcium, Calcineurin, and the Control of Transcription* , 2001, The Journal of Biological Chemistry.
[43] S. Litwin,et al. Na-Ca exchange and the trigger for sarcoplasmic reticulum Ca release: studies in adult rabbit ventricular myocytes. , 1998, Biophysical journal.
[44] N J Izzo,et al. HL-1 cells: a cardiac muscle cell line that contracts and retains phenotypic characteristics of the adult cardiomyocyte. , 1998, Proceedings of the National Academy of Sciences of the United States of America.
[45] H. Brown,et al. Pacemaking in rabbit isolated sino‐atrial node cells during Cs+ block of the hyperpolarization‐activated current if. , 1990, The Journal of physiology.
[46] C. Malbon,et al. Indirect immunofluorescence localization of beta-adrenergic receptors and G-proteins in human A431 cells. , 1989, The Biochemical journal.
[47] D. Rubenstein,et al. Mechanisms of automaticity in subsidiary pacemakers from cat right atrium. , 1989, Circulation research.
[48] M. Mazzanti,et al. Properties of the hyperpolarizing‐activated current (if) in cells isolated from the rabbit sino‐atrial node. , 1986, The Journal of physiology.
[49] G. Rubin,et al. Rescue of the Drosophila phototransduction mutation trp by germline transformation. , 1985, Science.
[50] W. Giles,et al. Ionic currents that generate the spontaneous diastolic depolarization in individual cardiac pacemaker cells. , 1985, Proceedings of the National Academy of Sciences of the United States of America.
[51] W. Claycomb,et al. Isolation and culture of the terminally differentiated adult mammalian ventricular cardiac muscle cell , 1984, In Vitro.
[52] E. Neher,et al. Inward current channels activated by intracellular Ca in cultured cardiac cells , 1981, Nature.
[53] B. Sakmann,et al. Improved patch-clamp techniques for high-resolution current recording from cells and cell-free membrane patches , 1981, Pflügers Archiv.
[54] H. Irisawa,et al. Inward current activated during hyperpolarization in the rabbit sinoatrial node cell , 1980, Pflügers Archiv.
[55] J. Michael. Textbook of Medical Physiology , 2005 .
[56] M. Xia,et al. Functional expression of L- and T-type Ca2+ channels in murine HL-1 cells. , 2004, Journal of molecular and cellular cardiology.
[57] T. Holy,et al. Loss of sex discrimination and male-male aggression in mice deficient for TRP2. , 2002, Nature Reviews Genetics.
[58] Claycomb Wc. Long-term culture and characterization of the adult ventricular and atrial cardiac muscle cell. , 1985, Basic research in cardiology.
[59] H. Lüdi,et al. The interrelations between the transport of sodium and calcium in mitochondria of various mammalian tissues. , 1978, European journal of biochemistry.