Low density lipoprotein receptor-negative mice expressing human apolipoprotein B-100 develop complex atherosclerotic lesions on a chow diet: no accentuation by apolipoprotein(a).
暂无分享,去创建一个
R. Hammer | H. Hobbs | V. Mooser | D. Sanan | S. Marcovina | D A Sanan | D L Newland | R Tao | S Marcovina | J Wang | V Mooser | R E Hammer | H H Hobbs | J. Wang | Jin Wang | D. Newland | R. Tao | Vincent Mooser
[1] R. Hammer,et al. Hypercholesterolemia in low density lipoprotein receptor knockout mice and its reversal by adenovirus-mediated gene delivery. , 1993, The Journal of clinical investigation.
[2] R. Krauss,et al. Overexpression of human apolipoprotein B-100 in transgenic rabbits results in increased levels of LDL and decreased levels of HDL. , 1995, Arteriosclerosis, thrombosis, and vascular biology.
[3] S. Young,et al. Conservation of the Low Density Lipoprotein Receptor‐Binding Domain of Apoprotein B , 1986, Arteriosclerosis.
[4] A. Lusis,et al. Genetic control of inflammatory gene induction and NF-kappa B-like transcription factor activation in response to an atherogenic diet in mice. , 1993, The Journal of clinical investigation.
[5] S. Young,et al. Susceptibility to atherosclerosis in mice expressing exclusively apolipoprotein B48 or apolipoprotein B100. , 1997, The Journal of clinical investigation.
[6] W. H. Hannon,et al. International Federation of Clinical Chemistry standardization project for measurements of apolipoproteins A-I and B. III. Comparability of apolipoprotein A-I values by use of international reference material. , 1993 .
[7] R. Lawn,et al. Activation of transforming growth factor-β is inhibited in transgenic apolipoprotein(a) mice , 1994, Nature.
[8] R. Hammer,et al. Relative contributions of apolipoprotein(a) and apolipoprotein-B to the development of fatty lesions in the proximal aorta of mice. , 1995, Arteriosclerosis, thrombosis, and vascular biology.
[9] E. Rubin,et al. Feedback Mechanism of Focal Vascular Lesion Formation in Transgenic Apolipoprotein(a) Mice* , 1996, The Journal of Biological Chemistry.
[10] E. Rubin,et al. Severe hypercholesterolemia and atherosclerosis in apolipoprotein E-deficient mice created by homologous recombination in ES cells , 1992, Cell.
[11] R. Hammer,et al. Atherogenesis in transgenic mice expressing human apolipoprotein(a) , 1992, Nature.
[12] N. Maeda,et al. Diet-induced atherosclerosis in mice heterozygous and homozygous for apolipoprotein E gene disruption. , 1994, The Journal of clinical investigation.
[13] S. Ishibashi,et al. Rapid genotyping of low density lipoprotein receptor knockout mice using a polymerase chain reaction technique , 1995, Laboratory animals.
[14] S. Young,et al. Transgenic mice expressing both human apolipoprotein B and human CETP have a lipoprotein cholesterol distribution similar to that of normolipidemic humans. , 1995, Journal of lipid research.
[15] V. Armstrong,et al. The association between serum Lp(a) concentrations and angiographically assessed coronary atherosclerosis. Dependence on serum LDL levels. , 1986, Atherosclerosis.
[16] M. Callow,et al. Atherogenesis in transgenic mice with human apolipoprotein B and lipoprotein (a). , 1995, The Journal of clinical investigation.
[17] R. Ross,et al. ApoE-deficient mice develop lesions of all phases of atherosclerosis throughout the arterial tree. , 1994, Arteriosclerosis and thrombosis : a journal of vascular biology.
[18] M. Callow,et al. Expression of human apolipoprotein B and assembly of lipoprotein(a) in transgenic mice. , 1994, Proceedings of the National Academy of Sciences of the United States of America.
[19] E. Rubin,et al. Apolipoprotein AI transgene corrects apolipoprotein E deficiency-induced atherosclerosis in mice. , 1994, The Journal of clinical investigation.
[20] E M Rubin,et al. Quantitation of atherosclerosis in murine models: correlation between lesions in the aortic origin and in the entire aorta, and differences in the extent of lesions between sexes in LDL receptor-deficient and apolipoprotein E-deficient mice. , 1995, Journal of lipid research.
[21] R. Hammer,et al. Reconstitution of lipoprotein(a) by infusion of human low density lipoprotein into transgenic mice expressing human apolipoprotein(a). , 1992, The Journal of biological chemistry.
[22] N. Maeda,et al. Atherosclerosis in mice lacking apo E. Evaluation of lesional development and progression. , 1994, Arteriosclerosis and thrombosis : a journal of vascular biology.
[23] D K Burns,et al. Massive xanthomatosis and atherosclerosis in cholesterol-fed low density lipoprotein receptor-negative mice. , 1994, The Journal of clinical investigation.
[24] N. Maeda,et al. Spontaneous hypercholesterolemia and arterial lesions in mice lacking apolipoprotein E. , 1992, Science.
[25] J. Breslow,et al. Human apolipoprotein A-I gene expression increases high density lipoprotein and suppresses atherosclerosis in the apolipoprotein E-deficient mouse. , 1994, Proceedings of the National Academy of Sciences of the United States of America.
[26] E. Rubin,et al. Modification of apolipoprotein(a) lysine binding site reduces atherosclerosis in transgenic mice. , 1997, The Journal of clinical investigation.
[27] Robert V Farese,et al. Transgenic mice expressing high levels of human apolipoprotein B develop severe atherosclerotic lesions in response to a high-fat diet. , 1995, The Journal of clinical investigation.
[28] E. Rubin,et al. Human apolipoprotein A-I prevents atherosclerosis associated with apolipoprotein[a] in transgenic mice. , 1994, Journal of lipid research.
[29] F. James Rohlf,et al. Biometry: The Principles and Practice of Statistics in Biological Research , 1969 .