Lipoteichoic acid induces surfactant protein-A biosynthesis in human alveolar type II epithelial cells through activating the MEK1/2-ERK1/2-NF-κB pathway

[1]  M. Bullock,et al.  Multikinase inhibitors: a new option for the treatment of thyroid cancer , 2011, Nature Reviews Endocrinology.

[2]  Ta-Liang Chen,et al.  Lipopolysaccharide stimulates syntheses of toll-like receptor 2 and surfactant protein-A in human alveolar epithelial A549 cells through upregulating phosphorylation of MEK1 and ERK1/2 and sequential activation of NF-κB. , 2011, Cytokine.

[3]  Ta-Liang Chen,et al.  Toll-like receptor 2-mediated sequential activation of MyD88 and MAPKs contributes to lipopolysaccharide-induced sp-a gene expression in human alveolar epithelial cells. , 2011, Immunobiology.

[4]  Ta-Liang Chen,et al.  Lipopolysaccharide induces apoptotic insults to human alveolar epithelial A549 cells through reactive oxygen species-mediated activation of an intrinsic mitochondrion-dependent pathway , 2011, Archives of Toxicology.

[5]  Victor M. Goldberg,et al.  Bacterial Pathogen-associated Molecular Patterns Stimulate Biological Activity of Orthopaedic Wear Particles by Activating Cognate Toll-like Receptors* , 2010, The Journal of Biological Chemistry.

[6]  O. Soehnlein,et al.  Roles of Heparin-Binding Protein in Bacterial Infections , 2010, Journal of Innate Immunity.

[7]  Ming-Yang Chang,et al.  ACUTE KIDNEY INJURY CLASSIFICATION: COMPARISON OF AKIN AND RIFLE CRITERIA , 2010, Shock.

[8]  Ruei-Ming Chen,et al.  LIPOTEICHOIC ACID-INDUCED TNF-&agr; AND IL-6 GENE EXPRESSIONS AND OXIDATIVE STRESS PRODUCTION IN MACROPHAGES ARE SUPPRESSED BY KETAMINE THROUGH DOWNREGULATING TOLL-LIKE RECEPTOR 2-MEDIATED ACTIVATION OF ERK1/2 AND NF&kgr;B , 2009, Shock.

[9]  C. Ince,et al.  The response of the host microcirculation to bacterial sepsis: does the pathogen matter? , 2010, Journal of Molecular Medicine.

[10]  Ta-Liang Chen,et al.  Molecular mechanisms of lipopolysaccharide-caused induction of surfactant protein-A gene expression in human alveolar epithelial A549 cells. , 2009, Toxicology letters.

[11]  Wen-Ta Chiu,et al.  Propofol inhibits lipoteichoic acid-induced iNOS gene expression in macrophages possibly through downregulation of toll-like receptor 2-mediated activation of Raf-MEK1/2-ERK1/2-IKK-NFkappaB. , 2009, Chemico-biological interactions.

[12]  Ta-Liang Chen,et al.  Cytoskeleton Interruption in Human Hepatoma HepG2 Cells Induced by Ketamine Occurs Possibly through Suppression of Calcium Mobilization and Mitochondrial Function , 2009, Drug Metabolism and Disposition.

[13]  A. Delaney,et al.  Polyclonal intravenous immunoglobulin for the treatment of severe sepsis and septic shock in critically ill adults: A systematic review and meta‐analysis * , 2007, Critical care medicine.

[14]  K. Clément,et al.  Macrophage-secreted factors impair human adipogenesis: involvement of proinflammatory state in preadipocytes. , 2007, Endocrinology.

[15]  T. Hartung,et al.  Highly purified lipoteichoic acid activates neutrophil granulocytes and delays their spontaneous apoptosis via CD14 and TLR2 , 2004, Journal of leukocyte biology.

[16]  M. Kavuru,et al.  Surfactant blocks lipopolysaccharide signaling by inhibiting both mitogen-activated protein and IkappaB kinases in human alveolar macrophages. , 2004, American journal of respiratory cell and molecular biology.

[17]  S. Foster,et al.  Peptidoglycan and Lipoteichoic Acid in Gram-Positive Bacterial Sepsis: Receptors, Signal Transduction, Biological Effects, and Synergism , 2003, Shock.

[18]  T. van der Poll,et al.  Lipoteichoic Acid and Peptidoglycan from Staphylococcus aureus Synergistically Induce Neutrophil Influx into the Lungs of Mice , 2003, Clinical Diagnostic Laboratory Immunology.

[19]  U. Göbel,et al.  Lipoteichoic Acid (LTA) of Streptococcus pneumoniaeand Staphylococcus aureus Activates Immune Cells via Toll-like Receptor (TLR)-2, Lipopolysaccharide-binding Protein (LBP), and CD14, whereas TLR-4 and MD-2 Are Not Involved* , 2003, The Journal of Biological Chemistry.

[20]  A. Jobe,et al.  Surfactant protein B inhibits endotoxin-induced lung inflammation. , 2003, American journal of respiratory cell and molecular biology.

[21]  T. Hartung,et al.  Lipoteichoic acid from Staphylococcus aureus is a potent stimulus for neutrophil recruitment. , 2003, Immunobiology.

[22]  Jonathan Cohen.  The immunopathogenesis of sepsis , 2002, Nature.

[23]  M. Schmitz,et al.  Lipoteichoic acid selectively induces the ERK signaling pathway in the cornea. , 2002, Investigative ophthalmology & visual science.

[24]  N. Palaniyar,et al.  Pulmonary innate immune proteins and receptors that interact with gram-positive bacterial ligands. , 2002, Immunobiology.

[25]  C. Basbaum,et al.  Platelet-activating factor receptor and ADAM10 mediate responses to Staphylococcus aureus in epithelial cells , 2002, Nature Medicine.

[26]  C. Ruppert,et al.  Surfactant alteration and replacement in acute respiratory distress syndrome , 2001, Respiratory research.

[27]  H. Kuo,et al.  Induction of cyclooxygenase‐2 protein by lipoteichoic acid from Staphylococcus aureus in human pulmonary epithelial cells: involvement of a nuclear factor‐κB‐dependent pathway , 2001, British journal of pharmacology.

[28]  S. Rooney.  Regulation of surfactant secretion. , 2001, Comparative biochemistry and physiology. Part A, Molecular & integrative physiology.

[29]  T. Hartung,et al.  Structure-function relationship of cytokine induction by lipoteichoic acid from Staphylococcus aureus. , 2001 .

[30]  J. M. Snyder,et al.  Surfactant protein A (SP‐A): the alveolus and beyond , 2001, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

[31]  G. Baldacci,et al.  Rap1A protein interferes with various MAP kinase activating pathways in skeletal myogenic cells , 2000, Oncogene.

[32]  J. Whitsett,et al.  Distinct Effects of Surfactant Protein A or D Deficiency During Bacterial Infection on the Lung1 , 2000, The Journal of Immunology.

[33]  M. Bates,et al.  ERK1 and ERK2 Activation by Chemotactic Factors in Human Eosinophils Is Interleukin 5-dependent and Contributes to Leukotriene C4 Biosynthesis* , 2000, The Journal of Biological Chemistry.

[34]  Sarma,et al.  LeVine AM, Whitsett JA, Gwozdz JA, Richardson TR, Fisher JH, Burhans MS, Korfhagen TR. Distinct effects of surfactant protein A or D deficiency during bacterial infection in the lung. J Immunol 2000;165:3934-3940. , 2000 .

[35]  R. Nau,et al.  Clinical outcome in pneumococcal meningitis correlates with CSF lipoteichoic acid concentrations. , 1999, Neurology.

[36]  A. Hamsten,et al.  Very Low-Density Lipoprotein Activates Nuclear Factor-κB in Endothelial Cells , 1999 .

[37]  A. Hamsten,et al.  Very-low-density lipoprotein activates nuclear factor kappa B in endothelial cells , 1998 .