论文信息 - Colocalization of low and oscillatory coronary wall shear stress with subsequent culprit lesion resulting in myocardial infarction in an orthotopic heart transplant patient.

Colocalization of low and oscillatory coronary wall shear stress with subsequent culprit lesion resulting in myocardial infarction in an orthotopic heart transplant patient.

Low and oscillatory wall shear stress (WSS) have been implicated in the pathogenesis of atherosclerosis [(1,2)][1]. Furthermore, cardiac allograft vasculopathy (CAV) has the highest rate of progression opposite flow dividers, suggesting a role of regional fluid dynamics [(3)][2]. However, to our

[1] A. Haverich,et al. Impact of vascular branching sites on focal progression of allograft vasculopathy in transplanted hearts. , 2001, Atherosclerosis.

[2] Michael C. McDaniel,et al. Coronary Artery Wall Shear Stress Is Associated With Progression and Transformation of Atherosclerotic Plaque and Arterial Remodeling in Patients With Coronary Artery Disease , 2011, Circulation.

[3] D. Ku,et al. Pulsatile Flow and Atherosclerosis in the Human Carotid Bifurcation: Positive Correlation between Plaque Location and Low and Oscillating Shear Stress , 1985, Arteriosclerosis.