Quantitative trait loci for acute behavioral sensitivity to paraoxon.
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[1] J. Crabbe,et al. Type I and type II error rates for quantitative trait loci (QTL) mapping studies using recombinant inbred mouse strains , 1996, Behavior genetics.
[2] U. Francke,et al. Chromosome localizations of genes for five cAMP-specific phosphodiesterases in man and mouse , 1994, Somatic cell and molecular genetics.
[3] W. Stigelman,et al. Goodman and Gilman's the Pharmacological Basis of Therapeutics , 1986 .
[4] V. Moser. Comparisons of the acute effects of cholinesterase inhibitors using a neurobehavioral screening battery in rats. , 1995, Neurotoxicology and teratology.
[5] Lee M. Silver,et al. Mouse Genetics: Concepts and Applications , 1995 .
[6] M. Weeks,et al. Effects of single and repeated exposures to abate on rat behavior and cholinesterase activity. , 1979, Toxicology.
[7] F. Thiesen,et al. Behavioral changes and cholinesterase activity of rats acutely treated with propoxur. , 1999, Japanese journal of pharmacology.
[8] A. Eldefrawi,et al. Direct actions of organophosphate anticholinesterases on nicotinic and muscarinic acetylcholine receptors. , 1988, Journal of biochemical toxicology.
[9] F. Stephenson,et al. Molecular characterization of N-methyl-D-aspartate receptors expressed in mammalian cells yields evidence for the coexistence of three subunit types within a discrete receptor molecule. , 1994, The Journal of biological chemistry.
[10] L. Goodman,et al. The Pharmacological Basis of Therapeutics , 1941 .
[11] P. Kurtz. Dissociated behavioral and cholinesterase decrements following malathion exposure. , 1977, Toxicology and applied pharmacology.
[12] J. Crabbe,et al. Provisional mapping of quantitative trait loci for chronic ethanol withdrawal severity in BXD recombinant inbred mice. , 1998, The Journal of pharmacology and experimental therapeutics.
[13] D. Milatović,et al. Different role of carboxylesterases in toxicity and tolerance to paraoxon and DFP. , 1999, Chemico-biological interactions.
[14] G. D'mello. Behavioural Toxicity of Anticholinesterases in Humans and Animals - A Review , 1993, Human & experimental toxicology.
[15] Allan Collins,et al. Behavioral phenotypes of inbred mouse strains: implications and recommendations for molecular studies , 1997, Psychopharmacology.
[16] I. Ho,et al. An assessment of comparative acute toxicity of diisopropyl-fluorophosphate, Tabun, Sarin, and Soman in relation to cholinergic and GABAergic enzyme activities in rats , 1984 .
[17] T. Ward,et al. Organophosphorus compounds preferentially affect second messenger systems coupled to M2/M4 receptors in rat frontal cortex , 1996, Brain Research Bulletin.
[18] J. Crabbe,et al. Quantitative trait loci: mapping drug and alcohol-related genes. , 1998, Advances in pharmacology.
[19] P. Durrington,et al. Effect of the molecular polymorphisms of human paraoxonase (PON1) on the rate of hydrolysis of paraoxon , 1997, British journal of pharmacology.
[20] N. Minton,et al. A Review of Organophosphate Poisoning , 1988, Medical toxicology and adverse drug experience.
[21] J. Noebels,et al. Genetic mapping and evaluation of candidate genes for spasmodic, a neurological mouse mutation with abnormal startle response. , 1993, Genomics.
[22] B. Gordesky-Gold,et al. Interstrain variability of larval photokinesis inDrosophila melanogaster , 1996, Behavior genetics.
[23] L. Costa,et al. Serum paraoxonase status: a major factor in determining resistance to organophosphates. , 1993, Journal of toxicology and environmental health.
[24] J. Crabbe,et al. Identifying genes for alcohol and drug sensitivity: recent progress and future directions , 1999, Trends in Neurosciences.
[25] C. J. Gordon,et al. Acute and delayed effects of diisopropyl fluorophosphate on body temperature, heart rate, and motor activity in the awake, unrestrained rat. , 1993, Journal of toxicology and environmental health.
[26] J. Crabbe,et al. Common genetic determinants of the ataxic and hypothermic effects of ethanol in BXD/Ty recombinant inbred mice: genetic correlations and quantitative trait loci. , 1996, The Journal of pharmacology and experimental therapeutics.
[27] M. Festing. Recombinant Inbred Strains , 1979 .
[28] K. Kirov,et al. Correlation between concentration of cholinesterases and the resistance of animals to organophosphorus compounds. , 1993, Drug and chemical toxicology.
[29] I. Ho,et al. Effect on striatal dopamine metabolism and differential motor behavioral tolerance following chronic cholinesterase inhibition with diisopropylfluorophosphate , 1984, Pharmacology Biochemistry and Behavior.
[30] C. Cunningham,et al. Ethanol-induced conditioned taste aversion in BXD recombinant inbred mice. , 1998, Alcoholism, clinical and experimental research.
[31] T. Diepgen,et al. Interethnic differences in the detoxification of organophosphates: the human serum paraoxonase polymorphism. , 1986, Archives of toxicology. Supplement. = Archiv fur Toxikologie. Supplement.
[32] W. Dettbarn,et al. Prevention of tolerance to the organophosphorus anticholinesterase paraoxon with carboxylesterase inhibitors. , 1998, Biochemical pharmacology.
[33] S. Sarin,et al. Biochemical and Behavioral Deficits in Adult Rat Following Chronic Dichlorvos Exposure , 1998, Pharmacology Biochemistry and Behavior.
[34] P. Bushnell,et al. Repeated inhibition of cholinesterase by chlorpyrifos in rats: behavioral, neurochemical and pharmacological indices of tolerance. , 1994, The Journal of pharmacology and experimental therapeutics.
[35] E. Lander,et al. A genetic linkage map of the mouse: current applications and future prospects. , 1993, Science.
[36] L G Sultatos,et al. Mammalian toxicology of organophosphorus pesticides. , 1994, Journal of toxicology and environmental health.
[37] A. Maelicke,et al. Paraoxon: cholinesterase-independent stimulation of transmitter release and selective block of ligand-gated ion channels in cultured hippocampal neurons. , 1996, The Journal of pharmacology and experimental therapeutics.
[38] M. Lyon,et al. Genetic variants and strains of the laboratory mouse , 1989 .
[39] Kaufman Lw,et al. Diisopropyl phosphorofluoridate (DFP) disrupts circadian activity patterns. , 1983 .
[40] I. Ho,et al. A striatal serotonergic involvement in the behavioural effects of anticholinesterase organophosphates. , 1984, European journal of pharmacology.
[41] C. Gordon,et al. Strain differences in the laboratory rat: impact on the autonomic, behavioral, and biochemical response to cholinesterase inhibition. , 1995, Journal of toxicology and environmental health.
[42] C. Pope,et al. Differential modulation of organophosphate-sensitive muscarinic receptors in rat brain by parathion and chlorpyrifos. , 1993, Journal of biochemical toxicology.
[43] P. Avner,et al. Structural analysis of mouse glycine receptor alpha subunit genes. Identification and chromosomal localization of a novel variant. , 1994, The Journal of biological chemistry.
[44] A. Collins,et al. Genetically determined differences in acute responses to diisopropylfluorophosphate , 1985, Pharmacology Biochemistry and Behavior.
[45] J. Belknap,et al. Quantitative trait loci associated with brain weight in the BXD/Ty recombinant inbred mouse strains , 1992, Brain Research Bulletin.
[46] B. Taylor,et al. Prp (proline-rich protein) genes linked to markers Es-12 (esterase-12), Ea-10 (erythrocyte alloantigen), and loci on distal mouse chromosome 6. , 1989, Genomics.
[47] L. Fogelson,et al. Relationship between serum cholinesterase activity and the change in body temperature and motor activity in the rat: a dose-response study of diisopropyl fluorophosphate. , 1993, Neurotoxicology and teratology.
[48] J. Crabbe,et al. The Genetic Basis of Alcohol and Drug Actions , 1991, Springer US.
[49] S. Robinson,et al. Dissociation of locomotor depression and ChE activity after DFP, soman and sarin , 1986, Pharmacology Biochemistry and Behavior.
[50] J. Clement,et al. Differences in the toxicity of soman in various strains of mice. , 1981, Fundamental and applied toxicology : official journal of the Society of Toxicology.
[51] A. C. Collins,et al. A strain comparison of physiological and locomotor responses of mice to diisopropylfluorosphosphate , 1986, Pharmacology Biochemistry and Behavior.
[52] M. Festing. Use of genetically heterogeneous rats and mice in toxicological research: a personal perspective. , 1990, Toxicology and applied pharmacology.
[53] O. von Deimling,et al. Biochemical and genetic characterization of esterase-27 (ES-27), the major plasma cholinesterase of the house mouse (Mus musculus). , 1991, Genetical research.
[54] J. Crabbe,et al. Quantitative Trait Loci Involved in Genetic Predisposition to Acute Alcohol Withdrawal in Mice , 1997, The Journal of Neuroscience.
[55] P. Green,et al. Interactions between anticholinesterase poisoning and opioid analgesia and locomotion in mice. , 1988, Neurotoxicology and teratology.
[56] T. Johnson,et al. Mapping quantitative trait loci for behavioral traits in the mouse , 1992, Behavior genetics.
[57] J. Wehner,et al. Genetic variation in paraoxonase activity and sensitivity to diisopropylphosphofluoridate in inbred mice , 1987, Pharmacology Biochemistry and Behavior.