Relations of Biomarkers Representing Distinct Biological Pathways to Left Ventricular Geometry
暂无分享,去创建一个
D. Levy | E. Benjamin | R. Vasan | W. Lieb | Thomas J. Wang | Philimon N. Gona | M. Larson | G. Tofler | J. Aragam | Raghava S. Velagaleti | D. Levy | P. Gona
[1] T. Cappola. Molecular remodeling in human heart failure. , 2008, Journal of the American College of Cardiology.
[2] J. Danesh,et al. Associations of plasma fibrinogen levels with established cardiovascular disease risk factors, inflammatory markers, and other characteristics: individual participant meta-analysis of 154,211 adults in 31 prospective studies: the fibrinogen studies collaboration. , 2007, American journal of epidemiology.
[3] D. Levy,et al. Multimarker Approach to Evaluate the Incidence of the Metabolic Syndrome and Longitudinal Changes in Metabolic Risk Factors: The Framingham Offspring Study , 2007, Circulation.
[4] A. Yamashina,et al. Left ventricular hypertrophy and geometry in untreated essential hypertension is associated with blood levels of aldosterone and procollagen type III amino-terminal peptide. , 2007, Circulation journal : official journal of the Japanese Circulation Society.
[5] F. Neumann,et al. Regression of Left Ventricular Hypertrophy following Stenting of Renal Artery Stenosis , 2007, Journal of endovascular therapy : an official journal of the International Society of Endovascular Specialists.
[6] D. Levy,et al. Clinical and Genetic Correlates of Aldosterone-to-Renin Ratio and Relations to Blood Pressure in a Community Sample , 2007, Hypertension.
[7] Nader Rifai,et al. Multiple Biomarkers and the Risk of Incident Hypertension , 2007, Hypertension.
[8] Marc A Pfeffer,et al. Controversies in ventricular remodelling , 2006, The Lancet.
[9] D. Conen,et al. Usefulness of B-type natriuretic peptide and C-reactive protein in predicting the presence or absence of left ventricular hypertrophy in patients with systemic hypertension. , 2006, The American journal of cardiology.
[10] J. Pratt. Too much aldosterone--more common than we ever realized? , 2006, American journal of hypertension.
[11] M. Kubo,et al. Elevated C-reactive protein is a predictor of the development of diabetes in a general Japanese population: the Hisayama Study. , 2005, Diabetes care.
[12] J. Jansson,et al. Comparison of soluble thrombomodulin, von Willebrand factor, tPA/PAI-1 complex, and high-sensitivity CRP concentrations in serum, EDTA plasma, citrated plasma, and acidified citrated plasma (Stabilyte) stored at -70 degrees C for 8-11 years. , 2005, Thrombosis research.
[13] A. Temizhan,et al. The influence of aldosterone on the development of left ventricular geometry and hypertrophy in patients with essential hypertension. , 2004, Japanese heart journal.
[14] L. Niskanen,et al. C-reactive protein and the development of the metabolic syndrome and diabetes in middle-aged men , 2004, Diabetologia.
[15] D. Levy,et al. Serum aldosterone and the incidence of hypertension in nonhypertensive persons. , 2004, The New England journal of medicine.
[16] J. Murabito,et al. Relations of Serum Aldosterone to Cardiac Structure: Gender-Related Differences in the Framingham Heart Study , 2004, Hypertension.
[17] D. Levy,et al. Relations of plasma homocysteine to left ventricular structure and function: the Framingham Heart Study. , 2004, European heart journal.
[18] B. Pitt. “Escape” of aldosterone production in patients with left ventricular dysfunction treated with an angiotensin converting enzyme inhibitor: Implications for therapy , 1995, Cardiovascular Drugs and Therapy.
[19] C. Bloor,et al. Increased gene expression of plasminogen activators and inhibitors in left ventricular hypertrophy , 1997, Molecular and Cellular Biochemistry.
[20] P. Ridker,et al. C-reactive protein and the risk of developing hypertension. , 2003, JAMA.
[21] N. Reichek,et al. Hypertension and Left Ventricular Hypertrophy : The 4 E Effects of Eplerenone , Enalapril , and Eplerenone / Enalapril in Patients With Essential , 2003 .
[22] M. Nieminen,et al. Change of left ventricular geometric pattern after 1 year of antihypertensive treatment: the Losartan Intervention For Endpoint reduction in hypertension (LIFE) study. , 2002, American heart journal.
[23] G. Berglund,et al. Blood Pressure Increase and Incidence of Hypertension in Relation to Inflammation-Sensitive Plasma Proteins , 2002, Arteriosclerosis, thrombosis, and vascular biology.
[24] T. Horio,et al. Influence of plasma aldosterone on left ventricular geometry and diastolic function in treated essential hypertension. , 2002, Hypertension research : official journal of the Japanese Society of Hypertension.
[25] D. Levy,et al. Plasma natriuretic peptides for community screening for left ventricular hypertrophy and systolic dysfunction: the Framingham heart study. , 2002, JAMA.
[26] R. Tracy,et al. Longitudinal Stability of Coagulation, Fibrinolysis, and Inflammation Factors in Stored Plasma Samples , 2001, Thrombosis and Haemostasis.
[27] B. Howard,et al. Fibrinogen and Preclinical Echocardiographic Target Organ Damage: The Strong Heart Study , 2001, Hypertension.
[28] B. Woodhams,et al. Stability of coagulation proteins in frozen plasma , 2001, Blood coagulation & fibrinolysis : an international journal in haemostasis and thrombosis.
[29] H. S. Klopfenstein,et al. M-mode echocardiographic predictors of six- to seven-year incidence of coronary heart disease, stroke, congestive heart failure, and mortality in an elderly cohort (the Cardiovascular Health Study). , 2001, The American journal of cardiology.
[30] T. Saruta,et al. Aldosterone Escape during Angiotensinconverting Enzyme Inhibitor Therapy in Essential Hypertensive Patients with Left Ventricular Hypertrophy , 2001, The Journal of international medical research.
[31] C. Brilla. Renin-angiotensin system mediated mechanisms: cardioreparation and cardioprotection , 2000, Heart.
[32] N. Lawson,et al. Aldosterone and renin measurements , 2000, Annals of clinical biochemistry.
[33] J. Cohn,et al. Cardiac remodeling--concepts and clinical implications: a consensus paper from an international forum on cardiac remodeling. Behalf of an International Forum on Cardiac Remodeling. , 2000, Journal of the American College of Cardiology.
[34] D. Levy,et al. Association of blood pressure with fibrinolytic potential in the Framingham offspring population. , 2000, Circulation.
[35] A. Levey,et al. A More Accurate Method To Estimate Glomerular Filtration Rate from Serum Creatinine: A New Prediction Equation , 1999, Annals of Internal Medicine.
[36] B. Pitt,et al. Regression of left ventricular hypertrophy in patients with hypertension: blockade of the renin-angiotensin-aldosterone system. , 1998, Circulation.
[37] H. Schunkert,et al. Neurohormonal activity and left ventricular geometry in patients with essential arterial hypertension. , 1998, American heart journal.
[38] T. Yoshimoto,et al. Left ventricular hypertrophy is more prominent in patients with primary aldosteronism than in patients with other types of secondary hypertension. , 1997, Hypertension research : official journal of the Japanese Society of Hypertension.
[39] H. Hense,et al. Associations between circulating components of the renin-angiotensin-aldosterone system and left ventricular mass. , 1997, Heart.
[40] D. Levy,et al. Prognosis of left ventricular geometric patterns in the Framingham Heart Study. , 1995, Journal of the American College of Cardiology.
[41] H. Matsuoka,et al. Clinical evidence for an association between left ventricular geometric adaptation and extracardiac target organ damage in essential hypertension , 1995, Journal of hypertension.
[42] D. Levy,et al. Impact of Left Ventricular Structure on the Incidence of Hypertension: The Framingham Heart Study , 1994, Circulation.
[43] D. Levy,et al. Sex differences in cardiac adaptation to isolated systolic hypertension. , 1993, The American journal of cardiology.
[44] J. Laragh,et al. Relation of left ventricular mass and geometry to morbidity and mortality in uncomplicated essential hypertension. , 1991, Annals of internal medicine.
[45] M. Alessi,et al. Measurement of plasminogen activator inhibitor 1 in biologic fluids with a murine monoclonal antibody-based enzyme-linked immunosorbent assay. , 1988, Blood.
[46] N. Reichek,et al. Echocardiographic assessment of left ventricular hypertrophy: comparison to necropsy findings. , 1986, The American journal of cardiology.
[47] W. Kannel,et al. An investigation of coronary heart disease in families. The Framingham offspring study. , 1979, American journal of epidemiology.
[48] A. DeMaria,et al. Recommendations Regarding Quantitation in M-Mode Echocardiography: Results of a Survey of Echocardiographic Measurements , 1978, Circulation.