A critical role for Pin1 in allergic pulmonary eosinophilia in rats.

[1]  R. Braun,et al.  Pin1 Modulates the Type 1 Immune Response , 2007, PloS one.

[2]  R. Sorkness,et al.  Altered allergen-induced eosinophil trafficking and physiological dysfunction in airways with preexisting virus-induced injury. , 2007, American journal of physiology. Lung cellular and molecular physiology.

[3]  S. Esnault,et al.  The Peptidyl-Prolyl Isomerase Pin1 Regulates Granulocyte-Macrophage Colony-Stimulating Factor mRNA Stability in T Lymphocytes1 , 2006, The Journal of Immunology.

[4]  L. Hassman,et al.  A central regulatory role for eosinophils and the eotaxin/CCR3 axis in chronic experimental allergic airway inflammation , 2006, Proceedings of the National Academy of Sciences.

[5]  J. Strominger.  The tortuous journey of a biochemist to immunoland and what he found there. , 2006, Annual review of immunology.

[6]  Esther B. E. Becker,et al.  Pin1 Mediates Neural-Specific Activation of the Mitochondrial Apoptotic Machinery , 2006, Neuron.

[7]  S. Esnault,et al.  The peptidyl-prolyl isomerase Pin1 regulates the stability of granulocyte-macrophage colony-stimulating factor mRNA in activated eosinophils , 2005, Nature Immunology.

[8]  Akira Yamaguchi,et al.  Stable Suppression of Tumorigenicity by Pin1-Targeted RNA Interference in Prostate Cancer , 2005, Clinical Cancer Research.

[9]  L. Burka,et al.  Metabolism and disposition of juglone in male F344 rats , 2005, Xenobiotica; the fate of foreign compounds in biological systems.

[10]  R. Sorkness,et al.  Systemic and pulmonary effects of fluticasone administered through a metered-dose inhaler in rats. , 2004, The Journal of allergy and clinical immunology.

[11]  S. Orkin,et al.  A Critical Role for Eosinophils in Allergic Airways Remodeling , 2004, Science.

[12]  E. Lenkiewicz,et al.  Defining a Link with Asthma in Mice Congenitally Deficient in Eosinophils , 2004, Science.

[13]  M. Dallman,et al.  Relation of CD4+CD25+ regulatory T-cell suppression of allergen-driven T-cell activation to atopic status and expression of allergic disease , 2004, The Lancet.

[14]  A. Ryo,et al.  Regulation of NF-kappaB signaling by Pin1-dependent prolyl isomerization and ubiquitin-mediated proteolysis of p65/RelA. , 2003, Molecular cell.

[15]  Margarita Martinez-Moczygemba,et al.  Biology of common β receptor–signaling cytokines: IL-3, IL-5, and GM-CSF , 2003 .

[16]  Tony Hunter,et al.  Role of the prolyl isomerase Pin1 in protecting against age-dependent neurodegeneration , 2003, Nature.

[17]  A. Kay,et al.  Eosinophil's role remains uncertain as anti-interleukin-5 only partially depletes numbers in asthmatic airway. , 2003, American journal of respiratory and critical care medicine.

[18]  Lin Ying Liu,et al.  Decreased Expression of Membrane IL-5 Receptor α on Human Eosinophils: I. Loss of Membrane IL-5 Receptor α on Airway Eosinophils and Increased Soluble IL-5 Receptor α in the Airway After Allergen Challenge1 , 2002, The Journal of Immunology.

[19]  S. Romagnani.  Cytokines and chemoattractants in allergic inflammation. , 2002, Molecular immunology.

[20]  Xiao Zhen Zhou,et al.  Binding and regulation of the transcription factor NFAT by the peptidyl prolyl cis–trans isomerase Pin1 , 2001, FEBS letters.

[21]  A. Greenleaf,et al.  Juglone, an inhibitor of the peptidyl-prolyl isomerase Pin1, also directly blocks transcription. , 2001, Nucleic acids research.

[22]  R. Sorkness,et al.  Reduced interferon-gamma secretion by natural killer cells from rats susceptible to postviral chronic airway dysfunction. , 2001, American journal of respiratory cell and molecular biology.

[23]  S. Esnault,et al.  Primary peripheral blood eosinophils rapidly degrade transfected granulocyte-macrophage colony-stimulating factor mRNA. , 1999, Journal of immunology.

[24]  G. Fischer,et al.  Selective inactivation of parvulin-like peptidyl-prolyl cis/trans isomerases by juglone. , 1998, Biochemistry.

[25]  T. Hunter,et al.  A human peptidyl–prolyl isomerase essential for regulation of mitosis , 1996, Nature.

[26]  R. Coffman,et al.  IL-13 selectively induces vascular cell adhesion molecule-1 expression in human endothelial cells. , 1995, Journal of immunology.

[27]  C. Sanderson.  Interleukin-5, Eosinophils, and Disease , 1992 .

[28]  K. Blaser,et al.  An improved immunomagnetic procedure for the isolation of highly purified human blood eosinophils. , 1991, Journal of immunological methods.

[29]  H. Kita,et al.  Materials and Methods Brief Definitive Report Granulocyte/macrophage Colony-stimulating Factor and Interleukin 3 Release from Human Peripheral Blood Eosinophils and Neutrophils , 2022 .

[30]  Stuart L. Schreiber,et al.  Calcineurin is a common target of cyclophilin-cyclosporin A and FKBP-FK506 complexes , 1991, Cell.

[31]  J. Bousquet,et al.  Eosinophilic inflammation in asthma. , 1990, The New England journal of medicine.

[32]  P. Tai,et al.  The effects of recombinant granulocyte‐macrophage colony‐stimulating factor (GM‐CSF) and interleukin‐3 on the secretory capacity of human blood eosinophils , 1990, Clinical and experimental immunology.

[33]  K. S. Ramesh,et al.  Phosphorylation and dephosphorylation of soluble proteins in human eosinophils , 1987, Journal of cellular biochemistry.