Kinetics of chemotaxis, cytokine, and chemokine release of NR8383 macrophages after exposure to inflammatory and inert granular insoluble particles.
暂无分享,去创建一个
M Epple | T. Brüning | M. Epple | G. Johnen | K. Loza | J. Bünger | A. Brik | I Schremmer | A Brik | D G Weber | N Rosenkranz | A Rostek | K Loza | T Brüning | G Johnen | J Bünger | G A Westphal | A. Rostek | G. Westphal | N. Rosenkranz | D. Weber | I. Schremmer | Matthias Epple | Jürgen Bünger | Nina Rosenkranz | Isabell Schremmer | Alexander Brik | Daniel G. Weber | Georg Johnen
[1] E. Teller,et al. ADSORPTION OF GASES IN MULTIMOLECULAR LAYERS , 1938 .
[2] S. Boyden. THE CHEMOTACTIC EFFECT OF MIXTURES OF ANTIBODY AND ANTIGEN ON POLYMORPHONUCLEAR LEUCOCYTES , 1962, The Journal of experimental medicine.
[3] C. Dubois,et al. Asbestos fibers and silica particles stimulate rat alveolar macrophages to release tumor necrosis factor. Autoregulatory role of leukotriene B4. , 1989, The American review of respiratory disease.
[4] K. Driscoll,et al. Differential effects of mineral dusts on the in vitro activation of alveolar macrophage eicosanoid and cytokine release. , 1990, Toxicology in vitro : an international journal published in association with BIBRA.
[5] B. Wallaert,et al. Production of tumor necrosis factor-alpha and interleukin-6 by human alveolar macrophages exposed in vitro to coal mine dust. , 1991, American Journal of Respiratory Cell and Molecular Biology.
[6] R. Takigiku,et al. Macrophage inflammatory proteins 1 and 2: expression by rat alveolar macrophages, fibroblasts, and epithelial cells and in rat lung after mineral dust exposure. , 1993, American journal of respiratory cell and molecular biology.
[7] G. Oberdörster,et al. Lung particle overload: implications for occupational exposures to particles. , 1995, Regulatory toxicology and pharmacology : RTP.
[8] D. Warheit,et al. Differential pulmonary responses in rats inhaling crystalline, colloidal or amorphous silica dusts. , 1995, Scandinavian journal of work, environment & health.
[9] K. Donaldson,et al. Inhalation of poorly soluble particles. II. Influence Of particle surface area on inflammation and clearance. , 2000, Inhalation toxicology.
[10] K. Donaldson,et al. Inhalation of poorly soluble particles. I. Differences in inflammatory response and clearance during exposure. , 2000, Inhalation toxicology.
[11] Thomas D. Schmittgen,et al. Analysis of relative gene expression data using real-time quantitative PCR and the 2(-Delta Delta C(T)) Method. , 2001, Methods.
[12] Y. Kurokawa,et al. Distinct Requirements for Interleukin-6 in Airway Inflammation Induced by Diesel Exhaust in Mice , 2006, Immunopharmacology and immunotoxicology.
[13] J. Heyder,et al. Instillation of Six Different Ultrafine Carbon Particles Indicates a Surface Area Threshold Dose for Acute Lung Inflammation in Mice , 2005, Environmental health perspectives.
[14] C. Benz,et al. Optimized high-throughput microRNA expression profiling provides novel biomarker assessment of clinical prostate and breast cancer biopsies , 2006, Molecular Cancer.
[15] R. Boyd,et al. From growth factor dependence to growth factor responsiveness: The genesis of an alveolar macrophage cell line , 1987, In Vitro Cellular & Developmental Biology.
[16] David M. Brown,et al. Proinflammogenic Effects of Low-Toxicity and Metal Nanoparticles In Vivo and In Vitro: Highlighting the Role of Particle Surface Area and Surface Reactivity , 2007, Inhalation toxicology.
[17] R. Helmke,et al. A continuous alveolar macrophage cell line: Comparisons with freshly derived alveolar macrophages , 2007, In Vitro Cellular & Developmental Biology.
[18] David B Warheit,et al. How meaningful are the results of nanotoxicity studies in the absence of adequate material characterization? , 2008, Toxicological sciences : an official journal of the Society of Toxicology.
[19] C. L. Tran,et al. Concordance Between In Vitro and In Vivo Dosimetry in the Proinflammatory Effects of Low-Toxicity, Low-Solubility Particles: The Key Role of the Proximal Alveolar Region , 2008 .
[20] P. Borm,et al. Impact of the FcgammaII-receptor on quartz uptake and inflammatory response by alveolar macrophages. , 2008, American journal of physiology. Lung cellular and molecular physiology.
[21] A. Penn,et al. Soot nanoparticles promote biotransformation, oxidative stress, and inflammation in murine lungs. , 2008, American journal of respiratory cell and molecular biology.
[22] Helinor Johnston,et al. Development of in vitro systems for nanotoxicology: methodological considerations , 2009, Critical reviews in toxicology.
[23] J. Reynolds,et al. Effects of Titanium Dioxide Nanoparticle Exposure on Neuroimmune Responses in Rat Airways , 2010, Journal of toxicology and environmental health. Part A.
[24] Annette M Schmidt,et al. Contrasting macrophage activation by fine and ultrafine titanium dioxide particles is associated with different uptake mechanisms , 2011, Particle and Fibre Toxicology.
[25] K. Jensen,et al. Airway exposure to silica-coated TiO2 nanoparticles induces pulmonary neutrophilia in mice. , 2010, Toxicological sciences : an official journal of the Society of Toxicology.
[26] Alexandra Kroll,et al. Testing Metal‐Oxide Nanomaterials for Human Safety , 2010, Advanced materials.
[27] R. Hurt,et al. A 3-dimensional in vitro model of epithelioid granulomas induced by high aspect ratio nanomaterials , 2011, Particle and Fibre Toxicology.
[28] A. Kettle,et al. Myeloperoxidase associated with neutrophil extracellular traps is active and mediates bacterial killing in the presence of hydrogen peroxide , 2012, Journal of leukocyte biology.
[29] Alexandra Kroll,et al. Interference of engineered nanoparticles with in vitro toxicity assays , 2012, Archives of Toxicology.
[30] Robert Landsiedel,et al. Hazard identification of inhaled nanomaterials: making use of short-term inhalation studies , 2012, Archives of Toxicology.
[31] Hermann Fromme,et al. Airborne indoor particles from schools are more toxic than outdoor particles. , 2012, American journal of respiratory cell and molecular biology.
[32] M. Lag,et al. Comparison of non-crystalline silica nanoparticles in IL-1β release from macrophages , 2012, Particle and Fibre Toxicology.
[33] S. Lang,et al. Modulation of neutrophil granulocytes in the tumor microenvironment: mechanisms and consequences for tumor progression. , 2013, Seminars in cancer biology.
[34] Oded Maimon,et al. Predictive Toxicology of cobalt ferrite nanoparticles: comparative in-vitro study of different cellular models using methods of knowledge discovery from data , 2013, Particle and Fibre Toxicology.
[35] J. Schreiber,et al. MicroRNA-223 controls susceptibility to tuberculosis by regulating lung neutrophil recruitment. , 2013, The Journal of clinical investigation.
[36] H. Hsu,et al. CX3CL1(+) Microparticles Mediate the Chemoattraction of Alveolar Macrophages toward Apoptotic Acute Promyelocytic Leukemic Cells , 2014, Cellular Physiology and Biochemistry.
[37] G. Opdenakker,et al. Serum amyloid A chemoattracts immature dendritic cells and indirectly provokes monocyte chemotaxis by induction of cooperating CC and CXC chemokines , 2015, European journal of immunology.
[38] Thomas Brüning,et al. Particle-induced cell migration assay (PICMA): A new in vitro assay for inflammatory particle effects based on permanent cell lines. , 2015, Toxicology in vitro : an international journal published in association with BIBRA.
[39] Halshka Graczyk,et al. Increase in oxidative stress levels following welding fume inhalation: a controlled human exposure study , 2016, Particle and Fibre Toxicology.
[40] Robert Landsiedel,et al. An in vitro alveolar macrophage assay for predicting the short-term inhalation toxicity of nanomaterials , 2016, Journal of Nanobiotechnology.