Insight into the neuroproteomics effects of the food-contaminant non-dioxin like polychlorinated biphenyls.

[1]  Oscar Alzate,et al.  Aroclor 1254, a developmental neurotoxicant, alters energy metabolism- and intracellular signaling-associated protein networks in rat cerebellum and hippocampus. , 2011, Toxicology and applied pharmacology.

[2]  S. Ceccatelli,et al.  Non-dioxin-like polychlorinated biphenyls interfere with neuronal differentiation of embryonic neural stem cells. , 2011, Toxicological sciences : an official journal of the Society of Toxicology.

[3]  F. Fonnum,et al.  Multivariate toxicity profiles and QSAR modeling of non-dioxin-like PCBs--an investigation of in vitro screening data from ultra-pure congeners. , 2011, Chemosphere.

[4]  Patrick De Boever,et al.  Cerebellum proteomics addressing the cognitive deficit of rats perinatally exposed to the food-relevant polychlorinated biphenyl 138. , 2011, Toxicological sciences : an official journal of the Society of Toxicology.

[5]  T. Hamers,et al.  In vitro toxicity profiling of ultrapure non-dioxin-like polychlorinated biphenyl congeners and their relative toxic contribution to PCB mixtures in humans. , 2011, Toxicological sciences : an official journal of the Society of Toxicology.

[6]  V. Felipo,et al.  Differential long-term effects of developmental exposure to polychlorinated biphenyls 52, 138 or 180 on motor activity and neurotransmission. Gender dependence and mechanisms involved , 2011, Neurochemistry International.

[7]  Martin van den Berg,et al.  Activation and potentiation of human GABAA receptors by non-dioxin-like PCBs depends on chlorination pattern. , 2010, Toxicological sciences : an official journal of the Society of Toxicology.

[8]  Bo Zhang,et al.  The DREAM Protein Negatively Regulates the NMDA Receptor through Interaction with the NR1 Subunit , 2010, The Journal of Neuroscience.

[9]  V. Felipo,et al.  Developmental exposure to polychlorinated biphenyls 52, 138 or 180 affects differentially learning or motor coordination in adult rats. mechanisms involved , 2010, Neuroscience.

[10]  V. Felipo,et al.  Polychlorinated biphenyls PCB 52, PCB 180, and PCB 138 impair the glutamate-nitric oxide-cGMP pathway in cerebellar neurons in culture by different mechanisms. , 2010, Chemical research in toxicology.

[11]  M. van den Berg,et al.  Potentiation of the human GABA(A) receptor as a novel mode of action of lower-chlorinated non-dioxin-like PCBs. , 2010, Environmental science & technology.

[12]  F. Fonnum,et al.  Mechanisms involved in the neurotoxic effects of environmental toxicants such as polychlorinated biphenyls and brominated flame retardants , 2009, Journal of neurochemistry.

[13]  M. López de Armentia,et al.  Inhibition of cAMP response element-binding protein reduces neuronal excitability and plasticity, and triggers neurodegeneration. , 2009, Cerebral cortex.

[14]  E. Fiebiger,et al.  Structural basis and specificity of human otubain 1-mediated deubiquitination. , 2009, The Biochemical journal.

[15]  J. Sweatt,et al.  The role of calsenilin/DREAM/KChIP3 in contextual fear conditioning. , 2009, Learning & memory.

[16]  Josef M. Penninger,et al.  Lack of DREAM Protein Enhances Learning and Memory and Slows Brain Aging , 2009, Current Biology.

[17]  K. Shen,et al.  The role of the ubiquitin proteasome system in synapse remodeling and neurodegenerative diseases , 2008, BioEssays : news and reviews in molecular, cellular and developmental biology.

[18]  R. Fanelli,et al.  Assessment of the dietary exposure to non-dioxin-like PCBs of the Italian general population. , 2008, Chemosphere.

[19]  Richard F. Thompson,et al.  Progesterone receptors: Form and function in brain , 2008, Frontiers in Neuroendocrinology.

[20]  J. Hell,et al.  Displacement of alpha-actinin from the NMDA receptor NR1 C0 domain By Ca2+/calmodulin promotes CaMKII binding. , 2007, Biochemistry.

[21]  V. Felipo,et al.  The function of the glutamate-nitric oxide-cGMP pathway in brain in vivo and learning ability decrease in parallel in mature compared with young rats. , 2007, Learning & memory.

[22]  R. Burgoyne,et al.  Neuronal calcium sensor proteins: generating diversity in neuronal Ca2+ signalling , 2007, Nature Reviews Neuroscience.

[23]  C. Dotti,et al.  Transmitting on actin: synaptic control of dendritic architecture , 2007, Journal of Cell Science.

[24]  C. Shuttleworth,et al.  Microtubule disruption, not calpain-dependent loss of MAP2, contributes to enduring NMDA-induced dendritic dysfunction in acute hippocampal slices , 2006, Experimental Neurology.

[25]  J. Cordero,et al.  Birth defects and disabilities: a public health issue for the 21st century. , 2005, American journal of public health.

[26]  M. Gilbert,et al.  Accumulation of methylmercury or polychlorinated biphenyls in in vitro models of rat neuronal tissue. , 2005, Toxicology and applied pharmacology.

[27]  M. Sheng,et al.  The dynamic turnover and functional roles of α-actinin in dendritic spines , 2004, Neuropharmacology.

[28]  Aaron DiAntonio,et al.  Ubiquitin and the synapse , 2002, Nature Reviews Neuroscience.

[29]  F. Ledo,et al.  Ca2+‐dependent block of CREB–CBP transcription by repressor DREAM , 2002, The EMBO journal.

[30]  T. Schettler,et al.  Toxic threats to neurologic development of children. , 2001, Environmental health perspectives.

[31]  F. Fonnum,et al.  Effect of polychlorinated biphenyls on the uptake of dopamine into rat brain synaptic vesicles: a structure-activity study. , 2001, Toxicology and applied pharmacology.

[32]  C. Goodman,et al.  Ubiquitination-dependent mechanisms regulate synaptic growth and function , 2001, Nature.

[33]  Roberta F. White,et al.  Neurobehavioral deficits associated with PCB in 7-year-old children prenatally exposed to seafood neurotoxicants. , 2001, Neurotoxicology and teratology.

[34]  F. Fonnum,et al.  The effect of polychlorinated biphenyls on the high affinity uptake of the neurotransmitters, dopamine, serotonin, glutamate and GABA, into rat brain synaptosomes. , 2001, Toxicology.

[35]  C. D. De Rosa,et al.  Effects of polychlorinated biphenyls on the nervous system , 2000, Toxicology and industrial health.

[36]  V. Felipo,et al.  Nicotine prevents glutamate-induced proteolysis of the microtubule-associated protein MAP-2 and glutamate neurotoxicity in primary cultures of cerebellar neurons , 1998, Neuropharmacology.

[37]  G. Clark,et al.  Isolation and characterization of a novel gene induced by 2,3,7,8-tetrachlorodibenzo-p-dioxin in rat liver. , 1996, Carcinogenesis.

[38]  R. Seegal.  Epidemiological and laboratory evidence of PCB-induced neurotoxicity. , 1996, Critical reviews in toxicology.

[39]  J. Jacobson,et al.  Intellectual impairment in children exposed to polychlorinated biphenyls in utero. , 1996, The New England journal of medicine.

[40]  S. Schantz,et al.  Spatial learning deficits in adult rats exposed to ortho-substituted PCB congeners during gestation and lactation. , 1995, Fundamental and applied toxicology : official journal of the Society of Toxicology.

[41]  R. Balázs,et al.  Observations on rat cerebellar cells in vitro: influence of substratum, potassium concentration and relationship between neurones and astrocytes. , 1983, Brain research.

[42]  V. Felipo,et al.  Developmental exposure to polychlorinated biphenyls PCB153 or PCB126 impairs learning ability in young but not in adult rats , 2007 .

[43]  J. Buxbaum,et al.  Altered A Formation and Long-Term Potentiation in a Calsenilin Knock-Out , 2003 .

[44]  P G Mulder,et al.  Effects of environmental exposure to polychlorinated biphenyls and dioxins on cognitive abilities in Dutch children at 42 months of age. , 1999, The Journal of pediatrics.

[45]  I. Pessah,et al.  Symposium overview: toxicity of non-coplanar PCBs. , 1998, Toxicological sciences : an official journal of the Society of Toxicology.

[46]  H. Tilson,et al.  Neurochemical effects of polychlorinated biphenyls: an overview and identification of research needs. , 1997, Neurotoxicology.