Electrophysiologic evidence that retinotectal synaptic transmission in the goldfish is nicotinic cholinergic

[1]  John T. Schmidt,et al.  Localization of α-bungarotoxin binding sites to the goldfish retinotectal projection , 1980, Brain Research.

[2]  J. Freeman,et al.  Characterization of the nicotinic acetylcholine receptor isolated from goldfish brain. , 1979, The Journal of biological chemistry.

[3]  R. Ng,et al.  Deenergization of nerve terminals by beta-bungarotoxin. , 1978, Biochemistry.

[4]  N. Schellart,et al.  A golgi study of goldfish optic tectum , 1978, The Journal of comparative neurology.

[5]  N. Hirokawa Characterization of various nervous tissues of the chick embryos through responses to chronic application and immunocytochemistry of β‐bungarotoxin , 1978, The Journal of comparative neurology.

[6]  S. Easter,et al.  Expansion of the half retinal projection to the tectum in goldfish: An electrophysiological and Anatomical study , 1978, The Journal of comparative neurology.

[7]  J. Schmidt,et al.  Retinal fibers alter tectal positional markers during the expansion of the half retinal projection in goldfish , 1978, The Journal of comparative neurology.

[8]  J. Freeman Possible regulatory function of acetylcholine receptor in maintenance of retinotectal synapses , 1977, Nature.

[9]  S. Easter,et al.  The role of the optic tectum in various visually mediated behaviors of goldfish , 1977, Brain Research.

[10]  R. Miledi,et al.  Acute muscle denervation induced by β-bungarotoxin , 1976, Proceedings of the Royal Society of London. Series B. Biological Sciences.

[11]  R H Masland,et al.  Responses to acetylcholine of ganglion cells in an isolated mammalian retina. , 1976, Journal of neurophysiology.

[12]  P. Roberts,et al.  Tectal deafferentation in the frog: Selective loss of l-glutamate and γ-aminobutyrate , 1976, Neuroscience.

[13]  M. Murray Regeneration of retinal axons into the goldfish optic tectum , 1976, The Journal of comparative neurology.

[14]  E. Hulme,et al.  BIOCHEMICAL STUDIES ON MUSCARINIC ACETYLCHOLINE RECEPTORS , 1976, Journal of neurochemistry.

[15]  P. Beart An evaluation ofl-glutamate as the transmitter released from optic nerve terminals of the pigeon , 1976, Brain Research.

[16]  M. Cuénod,et al.  EFFECTS OF RETINAL ABLATION ON UPTAKE OF GLUTAMATE, GLYCINE, GABA, PROLINE AND CHOLINE IN PIGEON TECTUM , 1976, Journal of neurochemistry.

[17]  W. Catterall Sodium transport by the acetylcholine receptor of cultured muscle cells. , 1975, The Journal of biological chemistry.

[18]  C. Nicholson,et al.  Theory of current source-density analysis and determination of conductivity tensor for anuran cerebellum. , 1975, Journal of neurophysiology.

[19]  C. Nicholson,et al.  Experimental optimization of current source-density technique for anuran cerebellum. , 1975, Journal of neurophysiology.

[20]  T. Bliss,et al.  The synaptic organization of optic afferents in the amphibian tectum , 1974, Proceedings of the Royal Society of London. Series B. Biological Sciences.

[21]  M. Kuhar,et al.  Action of hemicholinium-3 on cholinergic nerve terminals after alteration of neuronal impulse flow. , 1974, Neuropharmacology.

[22]  D. R. Curtis,et al.  ANTAGONISM OF INHIBITORY AMINO ACID ACTION BY TUBOCURARINE , 1974, British journal of pharmacology.

[23]  K. Krnjević,et al.  Chemical Nature of Synaptic Transmission in Vertebrates , 1974 .

[24]  R. J. Stevens A model of an early 'off' response in frog optic tectum. , 1974, Brain research.

[25]  S. Snyder,et al.  HIGH AFFINITY TRANSPORT OF CHOLINE INTO SYNAPTOSOMES OF RAT BRAIN 1 , 1973, Journal of neurochemistry.

[26]  R J Stevens,et al.  A cholinergic inhibitory system in the frog optic tectum: its role in visual electrical responses and feeding behavior. , 1973, Brain research.

[27]  H. Mcilwain,et al.  IONIC BASIS FOR THE DEPOLARIZATION OF CEREBRAL TISSUES BY EXCITATORY ACIDIC AMINO ACIDS , 1966, Journal of neurochemistry.

[28]  B. L. Ginsborg THE PHYSIOLOGY OF SYNAPSES , 1964 .

[29]  B. Katz,et al.  A study of the ‘desensitization’ produced by acetylcholine at the motor end‐plate , 1957, The Journal of physiology.

[30]  R. Kelly,et al.  Biochemical and physiological properties of a purified snake venom neurotoxin which acts presynaptically. , 1974, Journal of neurobiology.

[31]  C. Y. Lee,et al.  Chromatographic separation of the venom of Bungarus multicinctus and characterization of its components. , 1972, Journal of chromatography.

[32]  I. Chen,et al.  Ultrastructural changes in the motor nerve terminals caused by β-bungarotoxin , 1970, Virchows Archiv B Cell Pathology.

[33]  R. M. Gaze The formation of nerve connections , 1970 .