Identification of cJun-responsive genes in Rat-1a cells using multiple techniques: increased expression of stathmin is necessary for cJun-mediated anchorage-independent growth

[1]  A. Bader,et al.  TOJ3, a target of the v-Jun transcription factor, encodes a protein with transforming activity related to human microspherule protein 1 (MCRS1) , 2001, Oncogene.

[2]  A. Bader,et al.  JAC, a direct target of oncogenic transcription factor Jun, is involved in cell transformation and tumorigenesis , 2001, Proceedings of the National Academy of Sciences of the United States of America.

[3]  J. Vass,et al.  Regulation of a multigenic invasion programme by the transcription factor, AP-1: re-expression of a down-regulated gene, TSC-36, inhibits invasion , 2000, Oncogene.

[4]  P. Vogt,et al.  Identification and characterization of genes upregulated in cells transformed by v-Jun , 2000, Oncogene.

[5]  A. Bader,et al.  Conditional cell transformation by doxycycline-controlled expression of the ASV17 v-jun allele. , 2000, Virology.

[6]  J. Keski‐Oja,et al.  Independent Promoters Regulate the Expression of Two Amino Terminally Distinct Forms of Latent Transforming Growth Factor-β Binding Protein-1 (LTBP-1) in a Cell Type-specific Manner* , 1999, The Journal of Biological Chemistry.

[7]  L. Kaczmarek,et al.  Neuronal Excitation-driven and AP-1-dependent Activation of Tissue Inhibitor of Metalloproteinases-1 Gene Expression in Rodent Hippocampus* , 1999, The Journal of Biological Chemistry.

[8]  B. Segerman,et al.  Op18/Stathmin Mediates Multiple Region-Specific Tubulin and Microtubule-Regulating Activities , 1999, The Journal of cell biology.

[9]  T. Miki,et al.  Distinct expression patterns and transforming properties of multiple isoforms of Ost, an exchange factor for RhoA and Cdc42 , 1999, Oncogene.

[10]  D. Mason,et al.  BTG1: a triiodothyronine target involved in the myogenic influence of the hormone. , 1999, Experimental cell research.

[11]  P. Vogt,et al.  Heparin-binding epidermal growth factor-like growth factor, a v-Jun target gene, induces oncogenic transformation. , 1999, Proceedings of the National Academy of Sciences of the United States of America.

[12]  T. Curran,et al.  Role of DNA 5-methylcytosine transferase in cell transformation by fos. , 1999, Science.

[13]  M. Lan,et al.  Genomic structure and promoter sequence of the insulin-dependent diabetes mellitus autoantigen, IA-2 (PTPRN). , 1998, Genomics.

[14]  S. Kanner,et al.  Filamin binds to the cytoplasmic domain of the beta1-integrin. Identification of amino acids responsible for this interaction. , 1998, The Journal of biological chemistry.

[15]  J. Gierthy,et al.  12‐O‐tetradecanoylphorbol‐13‐acetate upregulates the Ah receptor and differentially alters CYP1B1 and CYP1A1 expression in MCF‐7 breast cancer cells , 1998, Journal of cellular biochemistry.

[16]  P. Vogt,et al.  Glutaredoxin is a direct target of oncogenic jun , 1998, Oncogene.

[17]  D. Geschwind,et al.  Genomic organization, 5' flanking enhancer region, and chromosomal assignment of the cell cycle gene, p55Cdc. , 1998, Molecular genetics and metabolism.

[18]  S. Folta,et al.  Molecular Cloning, Characterization, and Promoter Analysis of the Mouse Crp2/SmLim Gene , 1998, The Journal of Biological Chemistry.

[19]  A. Sabichi,et al.  Retinoic acid receptor beta expression and growth inhibition of gynecologic cancer cells by the synthetic retinoid N-(4-hydroxyphenyl) retinamide. , 1998, Journal of the National Cancer Institute.

[20]  M. Hadman,et al.  Apolipoprotein A-1 is a negative target of v-Jun overexpression , 1998, Oncogene.

[21]  M. Monsigny,et al.  Modulation of the expression of the rabbit galectin-3 gene by p53 and c-Ha-ras proteins and PMA. , 1997, Glycobiology.

[22]  M. Hagiwara,et al.  Controlled gene expression with a reverse tetracycline-regulated retroviral vector (RTRV) system. , 1997, Biochemical and biophysical research communications.

[23]  E. Zandi,et al.  AP-1 function and regulation. , 1997, Current opinion in cell biology.

[24]  M. Karin,et al.  Three distinct signalling responses by murine fibroblasts to genotoxic stress , 1996, Nature.

[25]  C. Griffin,et al.  Isolation and Characterization of the Human Cytochrome P450 CYP1B1 Gene* , 1996, The Journal of Biological Chemistry.

[26]  R. Davis,et al.  Transcription factor AP-1 regulation by mitogen-activated protein kinase signal transduction pathways , 1996, Journal of Molecular Medicine.

[27]  M. Catsicas,et al.  The phosphoprotein stathmin is essential for nerve growth factor- stimulated differentiation , 1996, The Journal of cell biology.

[28]  A. Sobel,et al.  Growth and cell density-dependent expression of stathmin in C2 myoblasts in culture. , 1996, Experimental cell research.

[29]  T. Mitchison,et al.  Identification of a Protein That Interacts with Tubulin Dimers and Increases the Catastrophe Rate of Microtubules , 1996, Cell.

[30]  Z. Werb,et al.  Synergistic Transcriptional Activation of the Tissue Inhibitor of Metalloproteinases-1 Promoter via Functional Interaction of AP-1 and Ets-1 Transcription Factors (*) , 1996, The Journal of Biological Chemistry.

[31]  M. Hartl,et al.  Specific activation in jun-transformed avian fibroblasts of a gene (bkj) related to the avian beta-keratin gene family. , 1995, Proceedings of the National Academy of Sciences of the United States of America.

[32]  S. Jimenez,et al.  Increased alpha 1(I) procollagen gene expression in tight skin (TSK) mice myocardial fibroblasts is due to a reduced interaction of a negative regulatory sequence with AP-1 transcription factor. , 1995, Journal of Biological Chemistry.

[33]  R. Lebovitz,et al.  Mediation of suppression of c‐fos transcription in rasT24‐transformed rat cells by a cis‐acting represser element , 1994, Molecular carcinogenesis.

[34]  H. Katai,et al.  An AP-1-like motif in the first intron of human Proα1(I) collagen gene is a critical determinant of its transcriptional activity , 1992, Molecular and Cellular Biochemistry.

[35]  U. Schubart,et al.  Widespread differentiation stage-specific expression of the gene encoding phosphoprotein p19 (metablastin) in mammalian cells. , 1992, Differentiation; research in biological diversity.

[36]  V. Doye,et al.  High expression of stathmin in multipotential teratocarcinoma and normal embryonic cells versus their early differentiated derivatives. , 1992, Differentiation; research in biological diversity.

[37]  G. Superti-Furga,et al.  Identification of Fos target genes by the use of selective induction systems , 1992, Journal of Cell Science.

[38]  J. Strahler,et al.  Involvement of OP18 in cell proliferation. , 1991, Biochemical and biophysical research communications.

[39]  J. Minna,et al.  Deregulated expression of human c-jun transforms primary rat embryo cells in cooperation with an activated c-Ha-ras gene and transforms rat-1a cells as a single gene. , 1989, Proceedings of the National Academy of Sciences of the United States of America.

[40]  B. Franza,et al.  Fos and jun: The AP-1 connection , 1988, Cell.

[41]  T. Hunter,et al.  Oncogene jun encodes a sequence-specific trans- activator similar to AP-1 , 1988, Nature.

[42]  R. Tjian,et al.  Human proto-oncogene c-jun encodes a DNA binding protein with structural and functional properties of transcription factor AP-1. , 1987, Science.

[43]  M. Karin,et al.  Phorbol ester-inducible genes contain a common cis element recognized by a TPA-modulated trans-acting factor , 1987, Cell.

[44]  P. Vogt,et al.  Avian sarcoma virus 17 carries the jun oncogene. , 1987, Proceedings of the National Academy of Sciences of the United States of America.

[45]  John L. Wang,et al.  The human LGALS3 (galectin-3) gene: determination of the gene structure and functional characterization of the promoter. , 1998, Archives of biochemistry and biophysics.