Periodontal Injection of Lipopolysaccharide Promotes Arthritis Development in Mice

[1]  Hyerin Jung,et al.  Arthritic role of Porphyromonas gingivalis in collagen-induced arthritis mice , 2017, PloS one.

[2]  J. Cirelli,et al.  The aggravation of arthritis by periodontitis is dependent of IL‐17 receptor A activation , 2017, Journal of clinical periodontology.

[3]  J. Potempa,et al.  The case for periodontitis in the pathogenesis of rheumatoid arthritis , 2017, Nature Reviews Rheumatology.

[4]  F. Curylofo-Zotti,et al.  A Chemically Modified Curcumin (CMC 2.24) Inhibits Nuclear Factor κB Activation and Inflammatory Bone Loss in Murine Models of LPS-Induced Experimental Periodontitis and Diabetes-Associated Natural Periodontitis , 2017, Inflammation.

[5]  Y. Fujiwara,et al.  Lidocaine Prevents Oxidative Stress-Induced Endothelial Dysfunction of the Systemic Artery in Rats With Intermittent Periodontal Inflammation , 2017, Anesthesia and analgesia.

[6]  M. G. Corrêa,et al.  Periodontitis increases rheumatic factor serum levels and citrullinated proteins in gingival tissues and alter cytokine balance in arthritic rats , 2017, PloS one.

[7]  E. Assier,et al.  Arthritis models: usefulness and interpretation , 2017, Seminars in Immunopathology.

[8]  G. Gislason,et al.  Risk of periodontitis in patients with psoriasis and psoriatic arthritis , 2017, Journal of the European Academy of Dermatology and Venereology : JEADV.

[9]  P. Emery,et al.  Periodontal disease and periodontal bacteria as triggers for rheumatoid arthritis. , 2017, Best practice & research. Clinical rheumatology.

[10]  Y. Jang,et al.  Fucoidan inhibits LPS‐induced inflammation in vitro and during the acute response in vivo , 2017, International immunopharmacology.

[11]  M. Feres,et al.  Does obesity influence the subgingival microbiota composition in periodontal health and disease? , 2016, Journal of clinical periodontology.

[12]  S. Yamasaki,et al.  Porphyromonas gingivalis infection exacerbates the onset of rheumatoid arthritis in SKG mice , 2016, Clinical and experimental immunology.

[13]  S. Zheng,et al.  Bone loss and aggravated autoimmune arthritis in HLA-DRβ1-bearing humanized mice following oral challenge with Porphyromonas gingivalis , 2016, Arthritis Research & Therapy.

[14]  L. Kesavalu,et al.  Periodontal bacterial colonization in synovial tissues exacerbates collagen-induced arthritis in B10.RIII mice , 2016, Arthritis Research & Therapy.

[15]  N. Fuggle,et al.  Hand to Mouth: A Systematic Review and Meta-Analysis of the Association between Rheumatoid Arthritis and Periodontitis , 2016, Front. Immunol..

[16]  E. Riboli,et al.  Smoking, Porphyromonas gingivalis and the immune response to citrullinated autoantigens before the clinical onset of rheumatoid arthritis in a Southern European nested case–control study , 2015, BMC Musculoskeletal Disorders.

[17]  U. Lerner,et al.  Porphyromonas gingivalis Stimulates Bone Resorption by Enhancing RANKL (Receptor Activator of NF-κB Ligand) through Activation of Toll-like Receptor 2 in Osteoblasts* , 2015, The Journal of Biological Chemistry.

[18]  Tetsuo Kobayashi,et al.  Host Responses in the Link Between Periodontitis and Rheumatoid Arthritis , 2014, Current Oral Health Reports.

[19]  T. Mikuls,et al.  The Link Between Periodontitis and Rheumatoid Arthritis: A Periodontist’s Perspective , 2014, Current Oral Health Reports.

[20]  C. Murray,et al.  Global Burden of Severe Periodontitis in 1990-2010 , 2014, Journal of dental research.

[21]  C. Marchant,et al.  Porphyromonas gingivalis Peptidylarginine Deiminase, a Key Contributor in the Pathogenesis of Experimental Periodontal Disease and Experimental Arthritis , 2014, PloS one.

[22]  G. Pruijn,et al.  Periodontal Pathogens Directly Promote Autoimmune Experimental Arthritis by Inducing a TLR2- and IL-1–Driven Th17 Response , 2014, The Journal of Immunology.

[23]  W. Robinson,et al.  Periodontitis and Porphyromonas gingivalis in Patients With Rheumatoid Arthritis , 2014, Arthritis & rheumatology.

[24]  A. Wolk,et al.  Cigarette smoking and risk of rheumatoid arthritis: a dose-response meta-analysis , 2014, Arthritis Research & Therapy.

[25]  G. Núñez,et al.  Porphyromonas gingivalis oral infection exacerbates the development and severity of collagen-induced arthritis , 2013, Arthritis Research & Therapy.

[26]  J. Cirelli,et al.  Host responses induced by different animal models of periodontal disease: a literature review. , 2013, Journal of investigative and clinical dentistry.

[27]  R. Jonsson,et al.  Porphyromonas gingivalis Facilitates the Development and Progression of Destructive Arthritis through Its Unique Bacterial Peptidylarginine Deiminase (PAD) , 2013, PLoS pathogens.

[28]  K. Üstün,et al.  Periodontal Disease in Patients with Psoriatic Arthritis , 2013, Inflammation.

[29]  A. Vissink,et al.  Periodontitis in established rheumatoid arthritis patients: a cross-sectional clinical, microbiological and serological study , 2012, Arthritis Research & Therapy.

[30]  D. Puleo,et al.  Animal Models for Periodontal Disease , 2011, Journal of biomedicine & biotechnology.

[31]  K. Lundberg,et al.  Periodontitis in RA—the citrullinated enolase connection , 2010, Nature Reviews Rheumatology.

[32]  Lucio Lo Russo,et al.  Dentistry and internal medicine: from the focal infection theory to the periodontal medicine concept. , 2010, European journal of internal medicine.

[33]  L. Shapira,et al.  Genetic and environmental risk factors for chronic periodontitis and aggressive periodontitis. , 2010, Periodontology 2000.

[34]  R. Toes,et al.  Diagnostic value of anti-MCV antibodies in differentiating early inflammatory arthritis , 2009, Annals of the rheumatic diseases.

[35]  W. B. Berg Lessons from animal models of arthritis over the past decade , 2009, Arthritis Research & Therapy.

[36]  L. C. Spolidorio,et al.  Signaling pathways associated with the expression of inflammatory mediators activated during the course of two models of experimental periodontitis. , 2009, Life sciences.

[37]  Thomas Dietrich,et al.  Periodontitis in systemic rheumatic diseases , 2009, Nature Reviews Rheumatology.

[38]  T. Ukai,et al.  T cells are able to promote lipopolysaccharide-induced bone resorption in mice in the absence of B cells. , 2008, Journal of periodontal research.

[39]  W. Giannobile,et al.  Actinobacillus actinomycetemcomitans lipopolysaccharide-mediated experimental bone loss model for aggressive periodontitis. , 2007, Journal of periodontology.

[40]  S. Genevay,et al.  Anti-cyclic citrullinated peptide antibodies, IgM and IgA rheumatoid factors in the diagnosis and prognosis of rheumatoid arthritis. , 2003, Rheumatology.

[41]  Tun-Yi Hsu,et al.  Bacteria Induce Osteoclastogenesis via an Osteoblast-Independent Pathway , 2002, Infection and Immunity.

[42]  Y. Shi,et al.  Porphyromonas gingivalis proteinases as virulence determinants in progression of periodontal diseases. , 2000, Journal of biochemistry.

[43]  J. Potempa,et al.  Purification, Characterization, and Sequence Analysis of a Potential Virulence Factor from Porphyromonas gingivalis, Peptidylarginine Deiminase , 1999, Infection and Immunity.

[44]  T. Nagata,et al.  Inhibition of Osteoblastic Cell Differentiation by Lipopolysaccharide Extract from Porphyromonas gingivalis , 1999, Infection and Immunity.

[45]  C. Gleissner,et al.  Risk for periodontal disease in patients with longstanding rheumatoid arthritis. , 1997, Arthritis and rheumatism.

[46]  S. Holt,et al.  Implantation of Bacteroides gingivalis in nonhuman primates initiates progression of periodontitis. , 1988, Science.

[47]  W. Ketterl [Periodontal diseases]. , 1971, Der Zahnarzt; Colloquium med. dent.