HIV-1 replication and gene expression occur at higher levels in neonatal blood naive and memory T-lymphocytes compared with adult blood cells.

[1]  J. Zack,et al.  Differential expression and interaction of host factors augment HIV-1 gene expression in neonatal mononuclear cells. , 2010, Virology.

[2]  S. Saxena,et al.  Differential HIV-1 replication in neonatal and adult blood mononuclear cells is influenced at the level of HIV-1 gene expression. , 2006, Proceedings of the National Academy of Sciences of the United States of America.

[3]  L. Kuhn,et al.  Immune pathogenesis of pediatric HIV-1 infection , 2006, Current HIV/AIDS reports.

[4]  R. Chakraborty HIV-1 infection in children: a clinical and immunologic overview. , 2005, Current HIV research.

[5]  L. Clement Isoforms of the CD45 common leukocyte antigen family: Markers for human T-cell differentiation , 2004, Journal of Clinical Immunology.

[6]  A. Perales,et al.  Comparison between two strategies for umbilical cord blood collection , 2003, Bone Marrow Transplantation.

[7]  T. Hahn,et al.  Biological characterization of HIV type 1 envelope V3 regions from mothers and infants associated with perinatal transmission. , 2001, AIDS research and human retroviruses.

[8]  S. L. Prescott,et al.  Cord blood memory responses: are we being naïve? , 2001, Clinical and experimental allergy : journal of the British Society for Allergy and Clinical Immunology.

[9]  S. W. Browning,et al.  CCR5 and CXCR4 Expression on Memory and Naive T Cells in HIV‐1 Infection and Response to Highly Active Antiretroviral Therapy , 2001, Journal of acquired immune deficiency syndromes.

[10]  C. Casper,et al.  Mother-to-child transmission of transmission of HIV-1: The role of HIV-1 variability and the placental barrier , 2001 .

[11]  E. Fenyö,et al.  Mother-to-child transmission of HIV-1: the role of HIV-1 variability and the placental barrier. , 2001, Acta microbiologica et immunologica Hungarica.

[12]  Y. Rivière,et al.  CD8+-Cell Antiviral Factor Activity Is Not Restricted to Human Immunodeficiency Virus (HIV)-Specific T Cells and Can Block HIV Replication after Initiation of Reverse Transcription , 2000, Journal of Virology.

[13]  S. Sánchez-Ramón,et al.  Predictive Markers of Clinical Outcome in Vertically HIV-1–Infected Infants. A Prospective Longitudinal Study , 2000, Pediatric Research.

[14]  H. Schuitemaker,et al.  In vivo HIV-1 infection of CD45RA(+)CD4(+) T cells is established primarily by syncytium-inducing variants and correlates with the rate of CD4(+) T cell decline. , 2000, Proceedings of the National Academy of Sciences of the United States of America.

[15]  J. Phair,et al.  Chemokine Coreceptor Usage by Diverse Primary Isolates of Human Immunodeficiency Virus Type 1 , 1998, Journal of Virology.

[16]  B. Levine,et al.  Naı̈ve and Memory CD4 T Cells Differ in Their Susceptibilities to Human Immunodeficiency Virus Type 1 Infection following CD28 Costimulation: Implications for Transmission and Pathogenesis , 1998, Journal of Virology.

[17]  M. Kalish,et al.  Association of human immunodeficiency virus (HIV) load early in life with disease progression among HIV-infected infants. New York City Perinatal HIV Transmission Collaborative Study Group. , 1998, The Journal of infectious diseases.

[18]  J. Moore,et al.  Expression patterns of the HIV type 1 coreceptors CCR5 and CXCR4 on CD4+ T cells and monocytes from cord and adult blood. , 1998, AIDS research and human retroviruses.

[19]  H. Ullum,et al.  Increased losses of CD4+CD45RA+ cells in late stages of HIV infection is related to increased risk of death: evidence from a cohort of 347 HIV‐infected individuals , 1997, AIDS.

[20]  C. Macken,et al.  Maternal HIV-1 viral load and vertical transmission of infection: The Ariel Project for the prevention of HIV transmission from mother to infant , 1997, Nature Medicine.

[21]  D. Richman,et al.  Preferential replication of HIV-1 in the CD45RO memory cell subset of primary CD4 lymphocytes in vitro. , 1997, The Journal of clinical investigation.

[22]  S. Moretti,et al.  CD8 lymphocytes in HIV infection: helpful and harmful. , 1997, Journal of clinical & laboratory immunology.

[23]  S. Skoda-Smith,et al.  CD4+ memory T cells are the predominant population of HIV‐1‐infected lymphocytes in neonates and children , 1996, AIDS.

[24]  W. Scott,et al.  Longitudinal studies of viral sequence, viral phenotype, and immunologic parameters of human immunodeficiency virus type 1 infection in perinatally infected twins with discordant disease courses , 1996, Journal of virology.

[25]  S. Ray,et al.  Convergent evolution within the V3 loop domain of human immunodeficiency virus type 1 in association with disease progression , 1995, Journal of virology.

[26]  J J Goedert,et al.  Natural history of HIV-1 cell-free viremia. , 1995, JAMA.

[27]  R. Connor,et al.  Vpr is required for efficient replication of human immunodeficiency virus type-1 in mononuclear phagocytes. , 1995, Virology.

[28]  C. Chappey,et al.  Genetic analysis of human immunodeficiency virus type 1 envelope V3 region isolates from mothers and infants after perinatal transmission , 1995, Journal of virology.

[29]  P. Thomas,et al.  Age at AIDS diagnosis for children with perinatally acquired HIV. , 1993, Journal of acquired immune deficiency syndromes.

[30]  N. Principi,et al.  Prognostic factors and survival in children with perinatal HIV‐1 infection , 1993 .

[31]  D. Deluca,et al.  Phenotypic and functional immaturity of human umbilical cord blood T lymphocytes. , 1992, Proceedings of the National Academy of Sciences of the United States of America.

[32]  A. Plebani,et al.  Prognostic factors and survival in children with perinatal HIV-1 infection , 1992, The Lancet.

[33]  M. de Martino,et al.  Prognostic factors and survival in children with perinatal HIV-1 infection. The Italian Register for HIV Infections in Children. , 1992, Lancet.

[34]  J. Justement,et al.  Preferential infection of CD4+ memory T cells by human immunodeficiency virus type 1: evidence for a role in the selective T-cell functional defects observed in infected individuals. , 1990, Proceedings of the National Academy of Sciences of the United States of America.

[35]  N. Ahmad,et al.  Rev-induced modulation of Nef protein underlies temporal regulation of human immunodeficiency virus replication. , 1989, Proceedings of the National Academy of Sciences of the United States of America.

[36]  R. Gaynor,et al.  Interactions of cellular proteins involved in the transcriptional regulation of the human immunodeficiency virus. , 1987, The EMBO journal.

[37]  H. Jaffe,et al.  Acquired immunodeficiency syndrome in children: report of the Centers for Disease Control National Surveillance, 1982 to 1985. , 1987, Pediatrics.