Free radicals and vascular disease: how much do we know?

Childhood Cancer Research Group, University of Oxford, Oxford OX2 6HJ 1 National Radiological Protection Board. Report of an advisory group on non-ionising radiation. Electromagnetic fields and the risk of cancer. Chilton: NRPB. 1992. (Documents of the NRPB. Vol 3No. 1.) 2 Jones TL, Shih CH, Thurston DH, Ware BJ, Cole P. Selection bias from differentiated residential mobility as an explanation for associations of wire codes with childhood cancer. J Clin Epidemiol 1993;46:891-5. 3 Olsen JH, Nielsen A, Schulgen G. Residence near high voltage facilities and risk of cancer in children. BMJ 1993;307:891-5. 4 Verkasalo PK, Pukkala E, Hongisto MY, Valjus JE, Jlrvinen PJ, Heikkila KV, et al. Risk of cancer in Finnish children living close to power lines. BMJ 1993;307:895-9. 5 Feychting M, Ahlbom A. Magnetic fields and cancer in people residing near Swedish high voltage power lines. Stockholm: Karolinska Institutet. 1992. (IMM report 6/92.) 6 [in SR, Lu PY. An epidemiologic study of childhood cancer in relation to residential exposure to electromagnetic fields (abstract). In: Department of the Environment, EPRI. Contractors' meeting. Portland, Oregon: DoE/EPRI, 1989.

[1]  B. Halliwell,et al.  Free radicals in biology and medicine , 1985 .

[2]  B. Freeman,et al.  Apparent hydroxyl radical production by peroxynitrite: implications for endothelial injury from nitric oxide and superoxide. , 1990, Proceedings of the National Academy of Sciences of the United States of America.

[3]  C. Arroyo,et al.  Endothelial cells as a source of oxygen-free radicals. An ESR study. , 1990, Free radical research communications.

[4]  S. Moncada,et al.  Nitric oxide: physiology, pathophysiology, and pharmacology. , 1991, Pharmacological reviews.

[5]  B. Herman,et al.  Cytosolic free Ca2+ and proteolysis in lethal oxidative injury in endothelial cells. , 1991, The American journal of physiology.

[6]  R. Thies,et al.  Reactive oxygen injury to cultured pulmonary artery endothelial cells: mediation by poly(ADP-ribose) polymerase activation causing NAD depletion and altered energy balance. , 1991, Archives of biochemistry and biophysics.

[7]  A. Verkerk,et al.  Vascular cells under peroxide induced oxidative stress: a balance study on in vitro peroxide handling by vascular endothelial and smooth muscle cells. , 1992, Free radical research communications.

[8]  R. Stocker,et al.  High density lipoprotein is the major carrier of lipid hydroperoxides in human blood plasma from fasting donors. , 1992, Proceedings of the National Academy of Sciences of the United States of America.

[9]  A. M. Lefer,et al.  Pharmacology of the endothelium in ischemia-reperfusion and circulatory shock. , 1993, Annual review of pharmacology and toxicology.

[10]  S. Padmaja,et al.  The reaction of no with superoxide. , 1993, Free radical research communications.

[11]  F. Parhami,et al.  Minimally modified low density lipoprotein-induced inflammatory responses in endothelial cells are mediated by cyclic adenosine monophosphate. , 1993, The Journal of clinical investigation.

[12]  R. Ross.  The pathogenesis of atherosclerosis: a perspective for the 1990s , 1993, Nature.

[13]  D. Leake.  Oxidised low density lipoproteins and atherogenesis. , 1993, British heart journal.

[14]  E. Anggard,et al.  Probucol inhibits mononuclear cell adhesion to vascular endothelium in the cholesterol-fed rabbit. , 1993, Atherosclerosis.

[15]  J. Pober,et al.  Endothelial activation by hydrogen peroxide. Selective increases of intercellular adhesion molecule-1 and major histocompatibility complex class I. , 1993, The American journal of pathology.