The effect of 5-hydroxytryptamine 3A and 3B receptor genes on nausea induced by paroxetine
暂无分享,去创建一个
N. Fukui | T. Sugai | T. Someya | K. Sawamura | Yutaro Suzuki | Y. Inoue | Y. Suzuki
[1] Margot Albus,et al. Investigation of the human serotonin receptor gene HTR3B in bipolar affective and schizophrenic patients , 2004, American journal of medical genetics. Part B, Neuropsychiatric genetics : the official publication of the International Society of Psychiatric Genetics.
[2] T. Someya,et al. Effects of dosage and CYP2D6-mutated allele on plasma concentration of paroxetine , 2004, European Journal of Clinical Pharmacology.
[3] R. Kaiser,et al. Investigation of the association between 5-HT3A receptor gene polymorphisms and efficiency of antiemetic treatment with 5-HT3 receptor antagonists. , 2004, Pharmacogenetics.
[4] A. Schatzberg,et al. Pharmacogenetics of antidepressant medication intolerance. , 2003, The American journal of psychiatry.
[5] R. Kaiser,et al. Variations in the 5-hydroxytryptamine type 3B receptor gene as predictors of the efficacy of antiemetic treatment in cancer patients. , 2003, Journal of clinical oncology : official journal of the American Society of Clinical Oncology.
[6] K. Otani,et al. Relationship between clinical effects of fluvoxamine and the steady-state plasma concentrations of fluvoxamine and its major metabolite fluvoxamino acid in Japanese depressed patients , 2003, Psychopharmacology.
[7] B. Gutiérrez,et al. Novel mutations in 5-HT3A and 5-HT3B receptor genes not associated with clozapine response , 2002, Schizophrenia Research.
[8] M. Morales,et al. Differential Composition of 5-Hydroxytryptamine3Receptors Synthesized in the Rat CNS and Peripheral Nervous System , 2002, The Journal of Neuroscience.
[9] M. Cazzola,et al. A novel deletion of the l‐ferritin iron‐responsive element responsible for severe hereditary hyperferritinaemia–cataract syndrome , 2002, British journal of haematology.
[10] Y. Sawada,et al. [Analysis of 5-HT3 receptor antagonist, ramosetron hydrochloride, based on receptor occupancy considering its active metabolite]. , 2001, Yakugaku zasshi : Journal of the Pharmaceutical Society of Japan.
[11] M. Rietschel,et al. Association between the 5' UTR variant C178T of the serotonin receptor gene HTR3A and bipolar affective disorder. , 2001, Pharmacogenetics.
[12] Trevor Sharp,et al. A review of central 5-HT receptors and their function , 1999, Neuropharmacology.
[13] D. Menon,et al. Adverse effects associated with selective serotonin reuptake inhibitors and tricyclic antidepressants: a meta-analysis. , 1998, CMAJ : Canadian Medical Association journal = journal de l'Association medicale canadienne.
[14] O. Andreassen,et al. Detection of the poor metabolizer-associated CYP2D6(D) gene deletion allele by long-PCR technology. , 1995, Pharmacogenetics.
[15] M. Ingelman-Sundberg,et al. Genetic analysis of the Chinese cytochrome P4502D locus: characterization of variant CYP2D6 genes present in subjects with diminished capacity for debrisoquine hydroxylation. , 1994, Molecular pharmacology.
[16] P. Blier,et al. Cisapride for the treatment of nausea produced by selective serotonin reuptake inhibitors. , 1994, The American journal of psychiatry.
[17] J. Hallas,et al. The relationship between paroxetine and the sparteine oxidation polymorphism , 1992, Clinical pharmacology and therapeutics.
[18] D. Stewart,et al. Cancer therapy, vomiting, and antiemetics. , 1990, Canadian journal of physiology and pharmacology.
[19] C. Kaye,et al. A review of the metabolism and pharmacokinetics of paroxetine in man , 1989, Acta psychiatrica Scandinavica. Supplementum.
[20] M. Tyers,et al. Mechanism of the anti-emetic activity of 5-HT3 receptor antagonists. , 1992, Oncology.
[21] H. Möller,et al. Comparative efficacy of antidepressants. Discussion , 1992 .
[22] K. Bunce,et al. Pharmacological and anti-emetic properties of ondansetron. , 1989, European journal of cancer & clinical oncology.