Deep vein thrombosis resolution is not accelerated with increased neovascularization.
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T. Wakefield | S. Kunkel | G. Upchurch | P. Henke | N. Dewyer | K. Deatrick | M. Varma | D. Moaveni | A. Varga | Manu R. Varma
[1] T. Wakefield,et al. Neutropenia impairs venous thrombosis resolution in the rat. , 2003, Journal of vascular surgery.
[2] T. Wakefield,et al. P-selectin inhibition decreases post-thrombotic vein wall fibrosis in a rat model. , 2003, Surgery.
[3] L. DiPietro,et al. Accelerated wound closure in neutrophil‐depleted mice , 2003, Journal of leukocyte biology.
[4] L. Greenfield,et al. P-selectin inhibition enhances thrombus resolution and decreases vein wall fibrosis in a rat model. , 2002, Journal of vascular surgery.
[5] A. Proudfoot. Chemokine receptors: multifaceted therapeutic targets , 2002, Nature Reviews Immunology.
[6] J. Fowlkes,et al. Interleukin-8 administration enhances venous thrombosis resolution in a rat model. , 2001, The Journal of surgical research.
[7] M. Waltham,et al. Recruitment of Labelled Monocytes by Experimental Venous Thrombi , 2001, Thrombosis and Haemostasis.
[8] B. Bennett,et al. Polymorphonuclear leucocytes mediate endogenous thrombus lysis via a u‐PA‐dependent mechanism , 2001, British journal of haematology.
[9] M. Waltham,et al. Vascular endothelial growth factor and basic fibroblast growth factor are found in resolving venous thrombi. , 2000, Journal of vascular surgery.
[10] J. Ward,et al. Human endothelial cells express CCR2 and respond to MCP-1: direct role of MCP-1 in angiogenesis and tumor progression. , 2000, Blood.
[11] A. Feldman,et al. The angiogenesis inhibitor, endostatin, does not affect murine cutaneous wound healing. , 2000, The Journal of surgical research.
[12] T. Wakefield. Treatment options for venous thrombosis. , 2000, Journal of vascular surgery.
[13] A. Zlotnik,et al. The biology of chemokines and their receptors. , 2000, Annual review of immunology.
[14] J. Wang,et al. Proinflammatory mediators stimulate neutrophil-directed angiogenesis. , 1999, Archives of surgery.
[15] M. Burdick,et al. Neovascularization during venous thrombosis organization: a preliminary study. , 1999, Journal of vascular surgery.
[16] M. Waltham,et al. Monocyte chemotactic protein-1 (MCP-1) accelerates the organization and resolution of venous thrombi. , 1999, Journal of vascular surgery.
[17] J. Isner,et al. Impaired collateral vessel development associated with reduced expression of vascular endothelial growth factor in ApoE-/- mice. , 1999, Circulation.
[18] N. Polissar,et al. Determinants of chronic venous disease after acute deep venous thrombosis. , 1998, Journal of vascular surgery.
[19] J. Garb,et al. Basic fibroblast growth factor stimulates angiogenesis in the hindlimb of hyperglycemic rats. , 1998, The Journal of surgical research.
[20] R. Cardiff,et al. Delayed wound healing and disorganized neovascularization in transgenic mice expressing the IP-10 chemokine. , 1998, Proceedings of the Association of American Physicians.
[21] A. Luster,et al. Chemokines--chemotactic cytokines that mediate inflammation. , 1998, The New England journal of medicine.
[22] R. Whyte,et al. The CXC chemokines, IL-8 and IP-10, regulate angiogenic activity in idiopathic pulmonary fibrosis. , 1997, Journal of immunology.
[23] M. Pepper,et al. Manipulating angiogenesis. From basic science to the bedside. , 1997, Arteriosclerosis, thrombosis, and vascular biology.
[24] L. Bobrow,et al. The tissue plasminogen activator and urokinase response in vivo during natural resolution of venous thrombus. , 1995, Journal of vascular surgery.
[25] M. Shikimori,et al. The cleavage and inactivation of plasminogen activator inhibitor type 1 by neutrophil elastase: the evaluation of its physiologic relevance in fibrinolysis. , 1995, Blood.
[26] Cord Sunderkötter,et al. Macrophages and angiogenesis , 1994, Journal of leukocyte biology.
[27] T. Standiford,et al. The immunopathology of chemotactic cytokines: the role of interleukin-8 and monocyte chemoattractant protein-1. , 1994, The Journal of laboratory and clinical medicine.
[28] G. Stewart,et al. Neutrophils and deep venous thrombosis. , 1993, Haemostasis.
[29] R. Strieter,et al. The immunopathology of chemotactic cytokines. , 1993, Advances in experimental medicine and biology.
[30] R. Strieter,et al. Interleukin-8 as a macrophage-derived mediator of angiogenesis. , 1992, Science.
[31] K. Burnand,et al. The Response of the Vessel Wall to Thrombosis: The in Vivo Study of Venous Thrombolysis , 1992, Annals of the New York Academy of Sciences.
[32] P. Walker,et al. A population based perspective of the hospital incidence and case fatality rates of deep vein thrombosis and pulmonary embolism: The Worcester DVT Study. Anderson FA, Wheeler HB, Goldberg RJ, et al. Arch Intern Med 1991;151:933-8 , 1992 .
[33] D. Hosmer,et al. A population-based perspective of the hospital incidence and case-fatality rates of deep vein thrombosis and pulmonary embolism. The Worcester DVT Study. , 1991, Archives of internal medicine.
[34] E. Kovacs. Fibrogenic cytokines: the role of immune mediators in the development of scar tissue. , 1991, Immunology today.
[35] J. W. Moore,et al. Comparison of the neovascular effects of stimulated macrophages and neutrophils in autologous rabbit corneas. , 1985, The American journal of pathology.
[36] S. Sevitt. The vascularisation of deep‐vein thrombi and their fibrous residue: A post‐mortem angiographic study , 1973, The Journal of pathology.