Increased water intake decreases progression of polycystic kidney disease in the PCK rat.
暂无分享,去创建一个
H. Kurahashi | S. Nagao | K. Nishii | M. Katsuyama | T. Marunouchi | Hisahide Takahashi | D. Wallace
[1] V. Torres. Water for ADPKD? Probably, yes. , 2006, Journal of the American Society of Nephrology : JASN.
[2] G. Germino,et al. Polycystin-1 induces resistance to apoptosis through the phosphatidylinositol 3-kinase/Akt signaling pathway. , 2006, Journal of the American Society of Nephrology : JASN.
[3] D. Wallace,et al. Calcium restores a normal proliferation phenotype in human polycystic kidney disease epithelial cells. , 2005, Journal of the American Society of Nephrology : JASN.
[4] C. Edelstein. What is the Role of Tubular Epithelial Cell Apoptosis in Polycystic Kidney Disease (PKD)? , 2005, Cell cycle.
[5] H. Kurahashi,et al. Androgen receptor pathway in rats with autosomal dominant polycystic kidney disease. , 2005, Journal of the American Society of Nephrology.
[6] V. Torres,et al. Effectiveness of vasopressin V2 receptor antagonists OPC-31260 and OPC-41061 on polycystic kidney disease development in the PCK rat. , 2005, Journal of the American Society of Nephrology : JASN.
[7] W. Bennett. V2 receptor antagonists in cystic kidney diseases: an exciting step towards a practical treatment. , 2005, Journal of the American Society of Nephrology : JASN.
[8] James P Calvet,et al. Calcium Restriction Allows cAMP Activation of the B-Raf/ERK Pathway, Switching Cells to a cAMP-dependent Growth-stimulated Phenotype* , 2004, Journal of Biological Chemistry.
[9] V. Torres. Therapies to Slow Polycystic Kidney Disease , 2004, Nephron Experimental Nephrology.
[10] D. Wallace,et al. Cyclic AMP promotes growth and secretion in human polycystic kidney epithelial cells. , 2004, Kidney international.
[11] S. Somlo,et al. Effective treatment of an orthologous model of autosomal dominant polycystic kidney disease , 2004, Nature Medicine.
[12] S. Somlo,et al. Histopathological analysis of renal cystic epithelia in the Pkd2WS25/- mouse model of ADPKD. , 2003, American journal of physiology. Renal physiology.
[13] Vincent H Gattone,et al. Inhibition of renal cystic disease development and progression by a vasopressin V2 receptor antagonist , 2003, Nature Medicine.
[14] J. Pelling,et al. Cyclic AMP activates B-Raf and ERK in cyst epithelial cells from autosomal-dominant polycystic kidneys. , 2003, Kidney international.
[15] A. Levey,et al. High urine volume and low urine osmolality are risk factors for faster progression of renal disease. , 2003, American journal of kidney diseases : the official journal of the National Kidney Foundation.
[16] R. Maser,et al. Renal activation of extracellular signal-regulated kinase in rats with autosomal-dominant polycystic kidney disease. , 2003, Kidney international.
[17] P. Harris. Molecular basis of polycystic kidney disease: PKD1, PKD2 and PKHD1 , 2002, Current opinion in nephrology and hypertension.
[18] N. Gretz,et al. Molecular basis of autosomal-dominant polycystic kidney disease , 2002, Cellular and Molecular Life Sciences CMLS.
[19] G. Delitala,et al. Decreased nocturnal urinary antidiuretic hormone excretion in enuresis is increased by imipramine , 2001, BJU international.
[20] K. Tomita,et al. Physiological effects of vasopressin and atrial natriuretic peptide in the collecting duct. , 2001, Cardiovascular research.
[21] W. Guggino,et al. cAMP regulates cell proliferation and cyst formation in autosomal polycystic kidney disease cells. , 2000, Journal of the American Society of Nephrology : JASN.
[22] L P Sullivan,et al. cAMP stimulates the in vitro proliferation of renal cyst epithelial cells by activating the extracellular signal-regulated kinase pathway. , 2000, Kidney international.
[23] J. Sands,et al. Urea transport processes are induced in rat IMCD subsegments when urine concentrating ability is reduced. , 1999, American journal of physiology. Renal physiology.
[24] S. Nagao,et al. Renal accumulation and excretion of cyclic adenosine monophosphate in a murine model of slowly progressive polycystic kidney disease. , 1997, American journal of kidney diseases : the official journal of the National Kidney Foundation.
[25] P. Wilson,et al. Expression of aquaporins-1 and -2 during nephrogenesis and in autosomal dominant polycystic kidney disease. , 1996, The American journal of physiology.
[26] Patricia A. Gabow,et al. PKD2, a Gene for Polycystic Kidney Disease That Encodes an Integral Membrane Protein , 1996, Science.
[27] R. Perrone,et al. Water channel expression in human ADPKD kidneys. , 1995, The American journal of physiology.
[28] I. Ramirez. Why do sugars taste good? , 1990, Neuroscience & Biobehavioral Reviews.
[29] Ann M. Johnson,et al. The clinical utility of renal concentrating capacity in polycystic kidney disease. , 1989, Kidney international.
[30] Verani Rr,et al. Histogenesis of the renal cysts in adult (autosomal dominant) polycystic kidney disease: a histochemical study. , 1988 .
[31] A. Bakkaloğlu,et al. Mutation analysis of the entire PKD1 gene: genetic and diagnostic implications. , 2001, American journal of human genetics.
[32] V. Torres,et al. The pck rat: a new model that resembles human autosomal dominant polycystic kidney and liver disease. , 2001, Kidney international.
[33] H. Takahashi,et al. Characterization of a novel polycystic kidney rat model with accompanying polycystic liver. , 2000, Experimental animals.
[34] R. Maser,et al. Developmental expression of urine concentration-associated genes and their altered expression in murine infantile-type polycystic kidney disease. , 1999, Developmental genetics.
[35] F. Silva,et al. Histogenesis of the renal cysts in adult (autosomal dominant) polycystic kidney disease: a histochemical study. , 1988, Modern pathology : an official journal of the United States and Canadian Academy of Pathology, Inc.