, Fabrisia contusion induced muscle injury dose of losartan influences the healing process after The timing of administration of a clinically relevant

[PDF] [Full Text] [Abstract] , June 5, 2013; 95 (11): 980-988. J Bone Joint Surg Am Johnny Huard Takayama, Michelle Witt, Gianluca Vadalà, Nick Oyster, Takanobu Otsuka, Freddie H. Fu and Satoshi Terada, Shusuke Ota, Makoto Kobayashi, Tetsuo Kobayashi, Yutaka Mifune, Koji Healing After Injury Use of an Antifibrotic Agent Improves the Effect of Platelet-Rich Plasma on Muscle [PDF] [Full Text] [Abstract] , August 15, 2013; 115 (4): 539-545. J Appl Physiol Mendias Jonathan P. Gumucio, Michael D. Flood, Anthony C. Phan, Susan V. Brooks and Christopher L. force production after contraction-induced skeletal muscle injury results in an initial improvement but long-term deficit in β Targeted inhibition of TGF[PDF] [Full Text] [Abstract] , November 15, 2014; 117 (10): 1120-1131. J Appl Physiol Koyal Garg, Benjamin T. Corona and Thomas J. Walters muscle loss injury Losartan administration reduces fibrosis but hinders functional recovery after volumetric

[1]  K. Patel.  Follistatin. , 2020, The international journal of biochemistry & cell biology.

[2]  T. Hornberger,et al.  Faculty Opinions recommendation of Regulation of skeletal muscle mass in mice by a new TGF-beta superfamily member. , 2014 .

[3]  G. Christ,et al.  Compression-induced muscle injury in rats that mimics compartment syndrome in humans. , 2012, The American journal of pathology.

[4]  E. Figallo,et al.  In vivo tissue engineering of functional skeletal muscle by freshly isolated satellite cells embedded in a photopolymerizable hydrogel , 2011, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

[5]  M. Koutsilieris,et al.  Muscle regeneration: cellular and molecular events. , 2009, In vivo.

[6]  K. Tsuchida,et al.  Follistatin induces muscle hypertrophy through satellite cell proliferation and inhibition of both myostatin and activin. , 2009, American journal of physiology. Endocrinology and metabolism.

[7]  J. Mendell,et al.  Inhibition of myostatin with emphasis on follistatin as a therapy for muscle disease , 2009, Muscle & nerve.

[8]  Freddie H. Fu,et al.  Improved Muscle Healing after Contusion Injury by the Inhibitory Effect of Suramin on Myostatin, a Negative Regulator of Muscle Growth , 2008, The American journal of sports medicine.

[9]  Yong Li,et al.  Angiotensin II Receptor Blockade Administered after Injury Improves Muscle Regeneration and Decreases Fibrosis in Normal Skeletal Muscle , 2008, The American journal of sports medicine.

[10]  K. Wagner,et al.  Myostatin Directly Regulates Skeletal Muscle Fibrosis* , 2008, Journal of Biological Chemistry.

[11]  K. Shyu,et al.  Angiotensin II activates myostatin expression in cultured rat neonatal cardiomyocytes via p38 MAP kinase and myocyte enhance factor 2 pathway. , 2008, The Journal of endocrinology.

[12]  J. Mendell,et al.  Long-term enhancement of skeletal muscle mass and strength by single gene administration of myostatin inhibitors , 2008, Proceedings of the National Academy of Sciences.

[13]  Konstantin I Bakhurin,et al.  Tendons of myostatin-deficient mice are small, brittle, and hypocellular , 2008, Proceedings of the National Academy of Sciences.

[14]  A. S. Greene,et al.  Angiotensin II Is a Critical Mediator of Prazosin‐Induced Angiogenesis in Skeletal Muscle , 2007, Microcirculation.

[15]  Freddie H. Fu,et al.  The Effect of Relaxin Treatment on Skeletal Muscle Injuries , 2005, The American journal of sports medicine.

[16]  L. Mcleay,et al.  Improved muscle healing through enhanced regeneration and reduced fibrosis in myostatin-null mice , 2005, Journal of Cell Science.

[17]  Y. Ohira,et al.  Possible role of calcineurin in heating-related increase of rat muscle mass. , 2005, Biochemical and biophysical research communications.

[18]  J. Huard,et al.  The Use of Relaxin Improves Healing in Injured Muscle , 2005, Annals of the New York Academy of Sciences.

[19]  K. Wagner,et al.  Muscle regeneration in the prolonged absence of myostatin. , 2005, Proceedings of the National Academy of Sciences of the United States of America.

[20]  H. Ohta,et al.  Losartan ameliorates progression of glomerular structural changes in diabetic KKAy mice. , 2004, Life sciences.

[21]  L. Liang,et al.  Myostatin signaling through Smad2, Smad3 and Smad4 is regulated by the inhibitory Smad7 by a negative feedback mechanism. , 2004, Cytokine.

[22]  Y. Ohira,et al.  Heat-stress enhances proliferative potential in rat soleus muscle. , 2004, The Japanese journal of physiology.

[23]  K. Patel,et al.  Follistatin complexes Myostatin and antagonises Myostatin-mediated inhibition of myogenesis. , 2004, Developmental biology.

[24]  Y. Ohira,et al.  Heat stress facilitates the recovery of atrophied soleus muscle in rat. , 2004, The Japanese journal of physiology.

[25]  Y. Ohira,et al.  Effects of heat stress and mechanical stretch on protein expression in cultured skeletal muscle cells , 2003, Pflügers Archiv.

[26]  Yong Li,et al.  Gamma interferon as an antifibrosis agent in skeletal muscle , 2003, Journal of orthopaedic research : official publication of the Orthopaedic Research Society.

[27]  Freddie H. Fu,et al.  Antifibrotic effects of suramin in injured skeletal muscle after laceration. , 2003, Journal of applied physiology.

[28]  K. Shelbourne,et al.  Severe Quadriceps Muscle Contusions in Athletes: A Report of Three Cases , 2003, The American journal of sports medicine.

[29]  B. Langley,et al.  Myostatin Inhibits Myoblast Differentiation by Down-regulating MyoD Expression* , 2002, The Journal of Biological Chemistry.

[30]  Yong Li,et al.  Differentiation of muscle-derived cells into myofibroblasts in injured skeletal muscle. , 2002, The American journal of pathology.

[31]  Yong Li,et al.  Muscle injuries and repair: current trends in research. , 2002, The Journal of bone and joint surgery. American volume.

[32]  J. Egido,et al.  Angiotensin II and renal fibrosis. , 2001, Hypertension.

[33]  Se-Jin Lee,et al.  Regulation of myostatin activity and muscle growth , 2001, Proceedings of the National Academy of Sciences of the United States of America.

[34]  Johnny Huard,et al.  The Use of an Antifibrosis Agent to Improve Muscle Recovery after Laceration * , 2001, The American journal of sports medicine.

[35]  J. Oldham,et al.  Myostatin regulation during skeletal muscle regeneration , 2000, Journal of cellular physiology.

[36]  C. Chatziantoniou,et al.  Angiotensin II Activates Collagen I Gene Through a Mechanism Involving the MAP/ER Kinase Pathway , 2000, Hypertension.

[37]  L. Ulbricht,et al.  Improvement of cardiac output in patients with severe heart failure by use of ACE‐inhibitors combined with the AT1‐antagonist eprosartan , 2000, European journal of heart failure.

[38]  K. Matthews,et al.  Myostatin, a transforming growth factor‐β superfamily member, is expressed in heart muscle and is upregulated in cardiomyocytes after infarct , 1999, Journal of cellular physiology.

[39]  J. Huard,et al.  Suturing versus immobilization of a muscle laceration: A morphological and functional study in a mouse model , 1999 .

[40]  Freddie H. Fu,et al.  Development of approaches to improve the healing following muscle contusion. , 1998, Cell transplantation.

[41]  Y Hochberg,et al.  Multiple test procedures for dose finding. , 1996, Biometrics.

[42]  E. Schultz,et al.  Role of satellite cells in altering myosin expression during avian skeletal muscle hypertrophy , 1994, Developmental dynamics : an official publication of the American Association of Anatomists.

[43]  H. Kalimo,et al.  Activation of myogenic precursor cells after muscle injury. , 1992, Medicine and science in sports and exercise.

[44]  U. K. Laemmli,et al.  Cleavage of Structural Proteins during the Assembly of the Head of Bacteriophage T4 , 1970, Nature.

[45]  E. Schultz,et al.  Skeletal muscle satellite cells. , 1994, Reviews of physiology, biochemistry and pharmacology.

[46]  D. A. Williams,et al.  A test for differences between treatment means when several dose levels are compared with a zero dose control. , 1971, Biometrics.