CD16 (FcRγIII) as a potential marker of osteoclast precursors in psoriatic arthritis
暂无分享,去创建一个
[1] Lionel B Ivashkiv,et al. Cross-regulation of signaling by ITAM-associated receptors , 2009, Nature Immunology.
[2] W. Wu,et al. Expansion of CD14+CD16+ peripheral monocytes among patients with aseptic loosening , 2009, Inflammation Research.
[3] J. Hamilton,et al. The proliferative human monocyte subpopulation contains osteoclast precursors , 2009, Arthritis research & therapy.
[4] J. Reuben,et al. PI3K limits TNF‐α production in CD16‐activated monocytes , 2009, European journal of immunology.
[5] J. Edwards,et al. Exploring the full spectrum of macrophage activation , 2008, Nature Reviews Immunology.
[6] S. Gordon,et al. Essential Role of DAP12 Signaling in Macrophage Programming into a Fusion-Competent State , 2008, Science Signaling.
[7] M. Benhamou,et al. Inhibitory ITAMs: a matter of life and death. , 2008, Trends in immunology.
[8] E. Schwarz,et al. VEGF-C, a Lymphatic Growth Factor, Is a RANKL Target Gene in Osteoclasts That Enhances Osteoclastic Bone Resorption through an Autocrine Mechanism* , 2008, Journal of Biological Chemistry.
[9] I. Mikhailenko,et al. IL‐4 promotes the formation of multinucleated giant cells from macrophage precursors by a STAT6‐dependent, homotypic mechanism: contribution of E‐cadherin , 2007, Journal of leukocyte biology.
[10] L. Ziegler‐Heitbrock,et al. Elevated CD16 expression by monocytes from patients with tumor necrosis factor receptor-associated periodic syndrome. , 2007, Arthritis and rheumatism.
[11] I. Velasco,et al. CD16 promotes Escherichia coli sepsis through an FcRγ inhibitory pathway that prevents phagocytosis and facilitates inflammation , 2007, Nature Medicine.
[12] D. Mosser,et al. Monocyte subpopulations and their differentiation patterns during infection , 2007, Journal of leukocyte biology.
[13] N. Takeyama,et al. Selective expansion of the CD14+/CD16bright subpopulation of circulating monocytes in patients with hemophagocytic syndrome , 2007, Annals of Hematology.
[14] H. Makino,et al. Recruitment of CD16+ monocytes into synovial tissues is mediated by fractalkine and CX3CR1 in rheumatoid arthritis patients. , 2007, Acta medica Okayama.
[15] L. Ziegler‐Heitbrock,et al. The CD14+ CD16+ blood monocytes: their role in infection and inflammation , 2007, Journal of leukocyte biology.
[16] Jianbin Wang,et al. CD16+ monocytes produce IL‐6, CCL2, and matrix metalloproteinase‐9 upon interaction with CX3CL1‐expressing endothelial cells , 2006, Journal of leukocyte biology.
[17] N. Miyasaka,et al. Identification of a human peripheral blood monocyte subset that differentiates into osteoclasts , 2006, Arthritis research & therapy.
[18] E. Schwarz,et al. Autoimmunity and Bone , 2006, Annals of the New York Academy of Sciences.
[19] L. Lanier,et al. Role of ITAM‐containing adapter proteins and their receptors in the immune system and bone , 2005, Immunological reviews.
[20] S. Gordon,et al. Monocyte and macrophage heterogeneity , 2005, Nature Reviews Immunology.
[21] D. Kane,et al. Early psoriatic arthritis. , 2005, Rheumatic diseases clinics of North America.
[22] L. Lanier,et al. TREM2, a DAP12‐Associated Receptor, Regulates Osteoclast Differentiation and Function , 2005, Journal of bone and mineral research : the official journal of the American Society for Bone and Mineral Research.
[23] D. F. Barber,et al. PI3Kγ inhibition blocks glomerulonephritis and extends lifespan in a mouse model of systemic lupus , 2005, Nature Medicine.
[24] J. Hamilton,et al. Regulation of Toll‐like receptor (TLR)2 and TLR4 on CD14dimCD16+ monocytes in response to sepsis‐related antigens , 2005, Clinical and experimental immunology.
[25] T. Taniguchi,et al. Costimulatory signals mediated by the ITAM motif cooperate with RANKL for bone homeostasis , 2004, Nature.
[26] L. Lanier,et al. The Signaling Adapter Protein DAP12 Regulates Multinucleation During Osteoclast Development , 2003, Journal of bone and mineral research : the official journal of the American Society for Bone and Mineral Research.
[27] S. Teitelbaum,et al. Genetic regulation of osteoclast development and function , 2003, Nature Reviews Genetics.
[28] F. Luscinskas,et al. Fractalkine Preferentially Mediates Arrest and Migration of CD16+ Monocytes , 2003, The Journal of experimental medicine.
[29] E. Schwarz,et al. Mechanisms of TNF-alpha- and RANKL-mediated osteoclastogenesis and bone resorption in psoriatic arthritis. , 2003, The Journal of clinical investigation.
[30] K. Schäkel,et al. The CD16+ (FcγRIII+) Subset of Human Monocytes Preferentially Becomes Migratory Dendritic Cells in a Model Tissue Setting , 2002, The Journal of experimental medicine.
[31] T. Espevik,et al. The Proinflammatory CD14+CD16+DR++ Monocytes Are a Major Source of TNF1 , 2002, The Journal of Immunology.
[32] D. Elewaut,et al. Human cartilage gp-39+,CD16+ monocytes in peripheral blood and synovium: correlation with joint destruction in rheumatoid arthritis. , 2000, Arthritis and rheumatism.
[33] C. Langlet,et al. Membrane rafts and signaling by the multichain immune recognition receptors. , 2000, Current opinion in immunology.
[34] J. Scherberich,et al. Expanded CD14+ CD16+Monocyte Subpopulation in Patients with Acute and Chronic Infections Undergoing Hemodialysis , 1998, Infection and Immunity.
[35] H. Ziegler-Heitbrock,et al. Heterogeneity of human blood monocytes: the CD14+ CD16+ subpopulation. , 1996, Immunology today.
[36] B. Passlick,et al. The novel subset of CD14+/CD16+ blood monocytes is expanded in sepsis patients , 1993 .
[37] B. Perussia. Fc receptors on natural killer cells. , 1998, Current topics in microbiology and immunology.
[38] B. Passlick,et al. The novel subset of CD14+/CD16+ blood monocytes is expanded in sepsis patients. , 1993, Blood.
[39] W. Eyler,et al. PSORIATIC ARTHRITIS. , 1965, Henry Ford Hospital medical bulletin.