SNPs in KCNQ1 are associated with susceptibility to type 2 diabetes in East Asian and European populations
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T. Hansen | O. Pedersen | T. Jørgensen | Yusuke Nakamura | T. Kawaguchi | T. Tsunoda | E. Tai | J. Holmkvist | M. Horikoshi | M. Kubo | K. Borch-Johnsen | T. Lauritzen | A. Takahashi | N. Kamatani | T. Kadowaki | Y. Iwamoto | R. Kawamori | A. Kashiwagi | R. Kikkawa | G. Andersen | A. Sandbæk | K. Hara | S. Maeda | H. Hirose | M. Hayashi | K. Kaku | D. Ng | T. Babazono | H. Unoki | S. Nurbaya | Knut Borch-Johnsen | M. Kubo | T. Hansen
[1] Yusuke Nakamura,et al. Association of CDKAL1, IGF2BP2, CDKN2A/B, HHEX, SLC30A8, and KCNJ11 With Susceptibility to Type 2 Diabetes in a Japanese Population , 2008, Diabetes.
[2] T. Nielsen,et al. The GCKR rs780094 polymorphism is associated with elevated fasting serum triacylglycerol, reduced fasting and OGTT-related insulinaemia, and reduced risk of type 2 diabetes , 2007, Diabetologia.
[3] Zhaohui S. Qin,et al. A second generation human haplotype map of over 3.1 million SNPs , 2007, Nature.
[4] M. McCarthy,et al. Replication of Genome-Wide Association Signals in UK Samples Reveals Risk Loci for Type 2 Diabetes , 2007, Science.
[5] Marcia M. Nizzari,et al. Genome-Wide Association Analysis Identifies Loci for Type 2 Diabetes and Triglyceride Levels , 2007, Science.
[6] J. Gulcher,et al. A variant in CDKAL1 influences insulin response and risk of type 2 diabetes , 2007, Nature Genetics.
[7] G. Abecasis,et al. A Genome-Wide Association Study of Type 2 Diabetes in Finns Detects Multiple Susceptibility Variants , 2007, Science.
[8] Bba,et al. Clinical Aspects of Type-1 Long-QT Syndrome by Location, Coding Type, and Biophysical Function of Mutations Involving the KCNQ1 Gene , 2007, Circulation.
[9] T. Hudson,et al. A genome-wide association study identifies novel risk loci for type 2 diabetes , 2007, Nature.
[10] Yusuke Nakamura,et al. Association of CDKAL 1 , IGF 2 BP 2 , CDKN 2 A / B , HHEX , SLC 30 A 8 and KCNJ 11 with susceptibility to type 2 diabetes in a Japanese population , 2007 .
[11] D. Reich,et al. Principal components analysis corrects for stratification in genome-wide association studies , 2006, Nature Genetics.
[12] H. Stefánsson,et al. Variant of transcription factor 7-like 2 (TCF7L2) gene confers risk of type 2 diabetes , 2006, Nature Genetics.
[13] G. Abecasis,et al. Joint analysis is more efficient than replication-based analysis for two-stage genome-wide association studies , 2006, Nature Genetics.
[14] F. Lang,et al. KCNQ1-dependent transport in renal and gastrointestinal epithelia. , 2005, Proceedings of the National Academy of Sciences of the United States of America.
[15] G. Abecasis,et al. A note on exact tests of Hardy-Weinberg equilibrium. , 2005, American journal of human genetics.
[16] Nicholas J. Wareham,et al. Genetic Factors in Type 2 Diabetes: The End of the Beginning? , 2005, Science.
[17] Charlotte Glümer,et al. A randomized non-pharmacological intervention study for prevention of ischaemic heart disease: baseline results Inter99 (1) , 2003, European journal of cardiovascular prevention and rehabilitation : official journal of the European Society of Cardiology, Working Groups on Epidemiology & Prevention and Cardiac Rehabilitation and Exercise Physiology.
[18] Yusuke Nakamura,et al. Gene-based SNP discovery as part of the Japanese Millennium Genome Project: identification of 190 562 genetic variations in the human genome , 2002, Journal of Human Genetics.
[19] D. Clayton,et al. A unified stepwise regression procedure for evaluating the relative effects of polymorphisms within a gene using case/control or family data: application to HLA in type 1 diabetes. , 2002, American journal of human genetics.
[20] Yusuke Nakamura,et al. A high-throughput SNP typing system for genome-wide association studies , 2001, Journal of Human Genetics.
[21] M. Franz,et al. Targeted disruption of the Kcnq1 gene produces a mouse model of Jervell and Lange– Nielsen Syndrome , 2001, Proceedings of the National Academy of Sciences of the United States of America.
[22] S. Chew,et al. Levels of cardiovascular disease risk factors in Singapore following a national intervention programme. , 2001, Bulletin of the World Health Organization.
[23] A. Feinberg,et al. Targeted disruption of the Kvlqt1 gene causes deafness and gastric hyperplasia in mice. , 2000, The Journal of clinical investigation.
[24] T Lauritzen,et al. The ADDITION study: proposed trial of the cost-effectiveness of an intensive multifactorial intervention on morbidity and mortality among people with Type 2 diabetes detected by screening , 2000, International Journal of Obesity.
[25] P. Coumel,et al. A novel mutation in the potassium channel gene KVLQT1 causes the Jervell and Lange-Nielsen cardioauditory syndrome , 1997, Nature Genetics.
[26] N. Risch,et al. A comparison of linkage disequilibrium measures for fine-scale mapping. , 1995, Genomics.