“Chik” from the womb!

symptomatically without further evaluation. Cutaneous examination revealed slate gray to brown pigmentation over the nose, perinasal, and perioral area. No evidence of similar pigmentation was noted anywhere else on the body. Physical evaluation and systemic examination were within normal limits. The basic hematology and blood biochemistry work up was also within normal limits. The differential diagnosis considered at this point was CKG induced pigmentation, posttraumatic (birth injury), and postinflammatory hyperpigmentation. A provisional diagnosis of CKG induced pigmentation Sir, Chikungunya (CKG), derives its name from “Kungunyala,” which means “that which bends up” in Makonde language, relating to the stooped posture adopted by these patients due to incapacitating polyarthralgia or arthritis. It is caused by alphavirus and is transmitted by the Asian tiger mosquitoes, Aedes aegypti, and Aedes albopictus. The first reported outbreak of CKG in India was from Calcutta city in 1963. The mucocutaneous manifestations of the disease include facial flush, fine discrete morbilliform exanthema, and occasionally, purpura has been reported. Other reported lesions include generalized vesiculobullous eruptions, lymphedema, and intertriginous aphthous‐like ulcers.[1,2] Herein, we report a 4 days old infant with the classical chik sign with positive IgM capture ELISA specific for CKG virus with negative titers in the mother.