Calnexin controls TrkB cell surface transport and ER-phagy in mouse cerebral cortex development.
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M. Sendtner | Patrick Lüningschrör | A. Denzel | Thomas Andreska | Mehri Moradi | Alexander Veh | Daniel Wolf | Neha Jadhav Giridhar
[1] Teresa Klein,et al. Plastin 3 rescues cell surface translocation and activation of TrkB in spinal muscular atrophy , 2023, The Journal of cell biology.
[2] H. Nakatogawa,et al. ER‐phagy: selective autophagy of the endoplasmic reticulum , 2022, EMBO reports.
[3] M. Sauer,et al. Dynamic remodeling of ribosomes and endoplasmic reticulum in axon terminals of motoneurons. , 2021, Journal of cell science.
[4] P. Arvan,et al. PGRMC1 acts as a size-selective cargo receptor to drive ER-phagic clearance of mutant prohormones , 2021, Nature Communications.
[5] M. Molinari,et al. N‐glycan processing selects ERAD‐resistant misfolded proteins for ER‐to‐lysosome‐associated degradation , 2021, The EMBO journal.
[6] M. Molinari. ER-phagy responses in yeast, plants, and mammalian cells and their crosstalk with UPR and ERAD. , 2021, Developmental cell.
[7] M. Sauer,et al. Induction of BDNF Expression in Layer II/III and Layer V Neurons of the Motor Cortex Is Essential for Motor Learning , 2020, The Journal of Neuroscience.
[8] M. Sendtner,et al. Regulation of TrkB cell surface expression—a mechanism for modulation of neuronal responsiveness to brain-derived neurotrophic factor , 2020, Cell and Tissue Research.
[9] M. Molinari,et al. A selective ER‐phagy exerts procollagen quality control via a Calnexin‐FAM134B complex , 2018, The EMBO journal.
[10] A. Cuervo,et al. Coordinate regulation of mutant NPC1 degradation by selective ER autophagy and MARCH6-dependent ERAD , 2018, Nature Communications.
[11] M. Molinari,et al. ER‐to‐lysosome‐associated degradation of proteasome‐resistant ATZ polymers occurs via receptor‐mediated vesicular transport , 2018, The EMBO journal.
[12] S. Petri,et al. Plekhg5-regulated autophagy of synaptic vesicles reveals a pathogenic mechanism in motoneuron disease , 2017, Nature Communications.
[13] S. Jablonka,et al. BDNF/trkB Induction of Calcium Transients through Cav2.2 Calcium Channels in Motoneurons Corresponds to F-actin Assembly and Growth Cone Formation on β2-Chain Laminin (221) , 2017, Front. Mol. Neurosci..
[14] E. Schuman,et al. Unconventional secretory processing diversifies neuronal ion channel properties , 2016, eLife.
[15] Álvaro Sebastián‐Serrano,et al. Cux1 Enables Interhemispheric Connections of Layer II/III Neurons by Regulating Kv1-Dependent Firing , 2016, Neuron.
[16] I. Katona,et al. Regulation of endoplasmic reticulum turnover by selective autophagy , 2015, Nature.
[17] M. Molinari,et al. N-linked sugar-regulated protein folding and quality control in the ER. , 2015, Seminars in cell & developmental biology.
[18] R. Campbell,et al. Palmitoylation is the switch that assigns calnexin to quality control or ER Ca2+ signaling , 2013, Journal of Cell Science.
[19] F. G. van der Goot,et al. Calnexin controls the STAT3-mediated transcriptional response to EGF. , 2013, Molecular cell.
[20] M. Sendtner,et al. EGF transactivation of Trk receptors regulates the migration of newborn cortical neurons , 2013, Nature Neuroscience.
[21] Kelley W. Moremen,et al. Vertebrate protein glycosylation: diversity, synthesis and function , 2012, Nature Reviews Molecular Cell Biology.
[22] M. Michalak,et al. Enhanced Clathrin-Dependent Endocytosis in the Absence of Calnexin , 2011, PloS one.
[23] P. H. Cameron,et al. Calnexin phosphorylation: linking cytoplasmic signalling to endoplasmic reticulum lumenal functions. , 2010, Seminars in cell & developmental biology.
[24] K. Krause,et al. Calnexin Deficiency Leads to Dysmyelination* , 2010, The Journal of Biological Chemistry.
[25] M. Chao,et al. Trk activation in the secretory pathway promotes Golgi fragmentation , 2010, Molecular and Cellular Neuroscience.
[26] P. H. Cameron,et al. Calnexin Phosphorylation Attenuates the Release of Partially Misfolded α1-Antitrypsin to the Secretory Pathway* , 2009, The Journal of Biological Chemistry.
[27] T. Simmen,et al. The subcellular distribution of calnexin is mediated by PACS-2. , 2008, Molecular biology of the cell.
[28] I. Sora,et al. Dopamine D1 Receptor-induced Signaling through TrkB Receptors in Striatal Neurons* , 2008, Journal of Biological Chemistry.
[29] D. Kaplan,et al. Trk signaling regulates neural precursor cell proliferation and differentiation during cortical development , 2007, Development.
[30] M. Chao,et al. A role for Fyn in Trk receptor transactivation by G-protein-coupled receptor signaling , 2006, Molecular and Cellular Neuroscience.
[31] Hideyuki Okano,et al. Suppression of basal autophagy in neural cells causes neurodegenerative disease in mice , 2006, Nature.
[32] R. Hardie,et al. Calnexin Is Essential for Rhodopsin Maturation, Ca2+ Regulation, and Photoreceptor Cell Survival , 2006, Neuron.
[33] Paola Arlotta,et al. Neuronal Subtype-Specific Genes that Control Corticospinal Motor Neuron Development In Vivo , 2005, Neuron.
[34] K. Unsicker,et al. TrkB regulates neocortex formation through the Shc/PLCγ‐mediated control of neuronal migration , 2004, The EMBO journal.
[35] Kanefusa Kato,et al. Cleavage of calnexin caused by apoptotic stimuli: implication for the regulation of apoptosis. , 2004, Journal of biochemistry.
[36] F. Lee,et al. Transactivation of Trk Neurotrophin Receptors by G-Protein-Coupled Receptor Ligands Occurs on Intracellular Membranes , 2004, The Journal of Neuroscience.
[37] A. Helenius,et al. Roles of N-linked glycans in the endoplasmic reticulum. , 2004, Annual review of biochemistry.
[38] G. Stamp,et al. Early Postnatal Death and Motor Disorders in Mice Congenitally Deficient in Calnexin Expression , 2002, Molecular and Cellular Biology.
[39] A. Lupas,et al. Calreticulin and calnexin in the endoplasmic reticulum are important for phagocytosis , 2001, The EMBO journal.
[40] F. Lee,et al. Activation of Trk neurotrophin receptors in the absence of neurotrophins , 2001, Proceedings of the National Academy of Sciences of the United States of America.
[41] H. Ohno,et al. Cell Surface Expression of Calnexin, a Molecular Chaperone in the Endoplasmic Reticulum* , 2000, The Journal of Biological Chemistry.
[42] Takeshi Noda,et al. LC3, a mammalian homologue of yeast Apg8p, is localized in autophagosome membranes after processing , 2000, The EMBO journal.
[43] H. Roderick,et al. Cytosolic Phosphorylation of Calnexin Controls Intracellular Ca2+ Oscillations via an Interaction with Serca2b , 2000, The Journal of cell biology.
[44] M. Lavail,et al. Role of Neurotrophin Receptor TrkB in the Maturation of Rod Photoreceptors and Establishment of Synaptic Transmission to the Inner Retina , 1999, The Journal of Neuroscience.
[45] J. Lindstrom,et al. Inhibition of Glucose Trimming with Castanospermine Reduces Calnexin Association and Promotes Proteasome Degradation of the α-Subunit of the Nicotinic Acetylcholine Receptor* , 1998, The Journal of Biological Chemistry.
[46] V. Katta,et al. Extracellular domain of neurotrophin receptor trkB: disulfide structure, N-glycosylation sites, and ligand binding. , 1995, Archives of biochemistry and biophysics.
[47] D. Vestweber,et al. The integrin chains beta 1 and alpha 6 associate with the chaperone calnexin prior to integrin assembly. , 1994, The Journal of biological chemistry.
[48] R. Spiro,et al. Inhibition of glucose trimming by castanospermine results in rapid degradation of unassembled major histocompatibility complex class I molecules. , 1993, The Journal of biological chemistry.