Myocardial Preservation during Cardiopulmonary Resuscitation

[1]  C M O'Connor,et al.  Cardiopulmonary resuscitation: historical perspective to recent investigations. , 1999, American heart journal.

[2]  Y. Sato,et al.  High-energy defibrillation increases the severity of postresuscitation myocardial dysfunction. , 1997, Circulation.

[3]  D. Meldrum,et al.  Adenosine preconditioning of human myocardium is dependent upon the ATP-sensitive K+ channel. , 1997, Journal of molecular and cellular cardiology.

[4]  Wanchun Tang,et al.  Effects of buffer agents on postresuscitation myocardial dysfunction. , 1996, Critical care medicine.

[5]  F. Crea,et al.  Preconditioning by transient myocardial ischemia confers protection against ischemia-induced ventricular arrhythmias in variant angina. , 1996, Circulation.

[6]  R. Damiano,et al.  Myocardial protection with potassium-channel openers is as effective as St. Thomas' solution in the rabbit heart. , 1996, The Annals of thoracic surgery.

[7]  Wanchun Tang,et al.  Myocardial dysfunction after successful resuscitation from cardiac arrest. , 1996, Critical care medicine.

[8]  A. Takeshita,et al.  ATP-sensitive K+ channel opener pinacidil augments beta 1-adrenoceptor-induced coronary vasodilation in dogs. , 1996, The American journal of physiology.

[9]  E. Marbán,et al.  Synergistic modulation of ATP-sensitive K+ currents by protein kinase C and adenosine. Implications for ischemic preconditioning. , 1996, Circulation research.

[10]  R. Bolli,et al.  Evidence for an essential role of reactive oxygen species in the genesis of late preconditioning against myocardial stunning in conscious pigs. , 1996, The Journal of clinical investigation.

[11]  E. Shoubridge,et al.  Intracellular Calcium Dynamics and Cellular Energetics in Ischemic NG108‐15 Cells Studied by Concurrent 31P/19F and 23Na Double‐Quantum Filtered NMR Spectroscopy , 1996, Journal of neurochemistry.

[12]  D. Duncker,et al.  Rapid ventricular pacing produces myocardial protection by nonischemic activation of KATP+ channels. , 1996, Circulation.

[13]  P. Menasche,et al.  Preconditioning with potassium channel openers. A new concept for enhancing cardioplegic protection? , 1995, The Journal of thoracic and cardiovascular surgery.

[14]  Wanchun Tang,et al.  Epinephrine increases the severity of postresuscitation myocardial dysfunction. , 1995, Circulation.

[15]  M. Arita,et al.  Pharmacological evidence for the persistent activation of ATP-sensitive K+ channels in early phase of reperfusion and its protective role against myocardial stunning. , 1995, Circulation.

[16]  G. Gross,et al.  Bimakalim, an ATP-sensitive potassium channel opener, mimics the effects of ischemic preconditioning to reduce infarct size, adenosine release, and neutrophil function in dogs. , 1995, Circulation.

[17]  H. Malone,et al.  KATP-channel openers protect against increased cytosolic calcium during ischaemia and reperfusion. , 1995, Journal of molecular and cellular cardiology.

[18]  G. Gross,et al.  Evidence for the involvement of the ATP-sensitive potassium channel in a novel model of hypoxic preconditioning in dogs. , 1995, Cardiovascular research.

[19]  S. Mochizuki,et al.  Protective effect of pacing on reperfusion-induced ventricular arrhythmias in isolated rat hearts. , 1995, The Canadian journal of cardiology.

[20]  T. Ehring,et al.  Stunned myocardium and the attenuation of stunning by calcium antagonists. , 1995, The American journal of cardiology.

[21]  Wanchun Tang,et al.  Mechanisms of myocardial hypercarbic acidosis during cardiac arrest , 1995 .

[22]  J. Papp,et al.  Pronounced antiarrhythmic effects of ischaemic preconditioning--are there possibilities for pharmacological exploitation? , 1995, Pharmacological research.

[23]  J. Papp,et al.  Attenuation of the antiarrhythmic effects of ischaemic preconditioning by blockade of bradykinin B2 receptors , 1994, British journal of pharmacology.

[24]  G. Lombardi,et al.  Outcome of Out-of-Hospital Cardiac Arrest in New York City: The Pre-Hospital Arrest Survival Evaluation (PHASE) Study , 1994 .

[25]  R. Bolli,et al.  Demonstration of free radical generation in the "stunned" myocardium in the conscious dog and identification of major differences between conscious and open-chest dogs. , 1993, The Journal of clinical investigation.

[26]  M. Weil,et al.  Progressive myocardial dysfunction after cardiac resuscitation , 1993, Critical care medicine.

[27]  T. Uegaito,et al.  Dobutamine prevents both myocardial stunning and phosphocreatine overshoot without affecting ATP level. , 1993, Journal of molecular and cellular cardiology.

[28]  Wanchun Tang,et al.  EPINEPHRINE INCREASES THE SEVERITY OF POST RESUSCITATION MYOCARDIAL DYSFUNCTION , 1993 .

[29]  J. Downey,et al.  Pretreatment with pertussis toxin blocks the protective effects of preconditioning: evidence for a G-protein mechanism. , 1993, Journal of molecular and cellular cardiology.

[30]  D. Chamberlain,et al.  Out-of-hospital resuscitation in East Sussex: 1981 to 1989. , 1992, British heart journal.

[31]  A. Schwartz.  Molecular and cellular aspects of calcium channel antagonism. , 1992, The American journal of cardiology.

[32]  G. Grover,et al.  Blockade of ischaemic preconditioning in dogs by the novel ATP dependent potassium channel antagonist sodium 5-hydroxydecanoate. , 1992, Cardiovascular research.

[33]  P. Pepe,et al.  A comparison of standard-dose and high-dose epinephrine in cardiac arrest outside the hospital. The Multicenter High-Dose Epinephrine Study Group. , 1992, The New England journal of medicine.

[34]  I. Stiell,et al.  High-dose epinephrine in adult cardiac arrest. , 1992, The New England journal of medicine.

[35]  D. Yellon,et al.  Ischaemic preconditioning limits infarct size in the rat heart. , 1992, Cardiovascular research.

[36]  T. Ehring,et al.  The Calcium Antagonist Nisoldipine Improves the Functional Recovery of Reperfused Myocardium Only When Given Before Ischemia , 1992, Journal of cardiovascular pharmacology.

[37]  L. Opie,et al.  Modulation of severity of reperfusion stunning in the isolated rat heart by agents altering calcium flux at onset of reperfusion. , 1992, Circulation research.

[38]  R. Kloner,et al.  The transient nature of the effect of ischemic preconditioning on myocardial infarct size and ventricular arrhythmia. , 1992, American heart journal.

[39]  G. Gross,et al.  Blockade of ATP-sensitive potassium channels prevents myocardial preconditioning in dogs. , 1992, Circulation research.

[40]  R. Capone,et al.  Unsuccessful emergency medical resuscitation--are continued efforts in the emergency department justified? , 1991, The New England journal of medicine.

[41]  Andrew M. Johanos.  A randomized clinical study of a calcium-entry blocker (lidoflazine) in the treatment of comatose survivors of cardiac arrest. , 1991, The New England journal of medicine.

[42]  J. Downey,et al.  Preconditioning Causes Improved Wall Motion as Well as Smaller Infarcts After Transient Coronary Occlusion in Rabbits , 1991, Circulation.

[43]  J. Downey,et al.  Protection Against Infarction Afforded by Preconditioning is Mediated by A1 Adenosine Receptors in Rabbit Heart , 1991, Circulation.

[44]  R. London,et al.  Amiloride delays the ischemia-induced rise in cytosolic free calcium. , 1991, Circulation research.

[45]  J Barrett,et al.  Outcome of CPR in a large metropolitan area--where are the survivors? , 1991, Annals of emergency medicine.

[46]  T. Miura,et al.  Myocardial Infarct Size–Limiting Effect of Ischemic Preconditioning Was Not Attenuated by Oxygen Free‐Radical Scavengers in the Rabbit , 1991, Circulation.

[47]  E. Marbán,et al.  Myocardial stunning and hibernation. The physiology behind the colloquialisms. , 1991, Circulation.

[48]  S. Kaul,et al.  Reversible Myocardial Depression in Survivors of Cardiac Arrest , 1990, Pacing and clinical electrophysiology : PACE.

[49]  W. Schaper,et al.  Ischemic preconditioning reduces infarct size in swine myocardium. , 1990, Circulation research.

[50]  R. London,et al.  Correlation between cytosolic free calcium, contracture, ATP, and irreversible ischemic injury in perfused rat heart. , 1990, Circulation research.

[51]  D. Hearse,et al.  Preconditioning of ischemic myocardium: reperfusion-induced arrhythmias. , 1987, The American journal of physiology.

[52]  M. Weisfeldt,et al.  Pathophysiology and pathogenesis of stunned myocardium. Depressed Ca2+ activation of contraction as a consequence of reperfusion-induced cellular calcium overload in ferret hearts. , 1987, The Journal of clinical investigation.

[53]  R. Raker,et al.  Randomized clinical study of thiopental loading in comatose survivors of cardiac arrest: Brain Resuscitation clinical Trial I Study Group. N Eng J Med 314:397–403, (February), 1986 , 1987 .

[54]  R. Jennings,et al.  Four brief periods of myocardial ischemia cause no cumulative ATP loss or necrosis. , 1986, The American journal of physiology.

[55]  R. Jennings,et al.  Preconditioning with ischemia: a delay of lethal cell injury in ischemic myocardium. , 1986, Circulation.

[56]  N. Abramson,et al.  Randomized clinical study of thiopental loading in comatose survivors of cardiac arrest. Brain resuscitation clinical trial I study group , 1986 .

[57]  J J Heger,et al.  Sudden cardiac death. , 1998, Circulation.

[58]  E. Nagel,et al.  Prehospital ventricular defibrillation. Prognosis and follow-up course. , 1974, The New England journal of medicine.