Preliminary observations in systemic oxygen consumption during targeted temperature management after cardiac arrest.
暂无分享,去创建一个
[1] C. Callaway,et al. Characterization of mitochondrial injury after cardiac arrest (COMICA). , 2017, Resuscitation.
[2] F. Sterz,et al. Resting energy expenditure and substrate oxidation rates correlate to temperature and outcome after cardiac arrest - a prospective observational cohort study , 2015, Critical Care.
[3] T. Oshima,et al. Fulfilling caloric demands according to indirect calorimetry may be beneficial for post cardiac arrest patients under therapeutic hypothermia. , 2015, Resuscitation.
[4] R. Neumar,et al. Post-cardiac arrest syndrome: epidemiology, pathophysiology, treatment, and prognostication. A Scientific Statement from the International Liaison Committee on Resuscitation; the American Heart Association Emergency Cardiovascular Care Committee; the Council on Cardiovascular Surgery and Anesthesia; , 2008, Resuscitation.
[5] R. Gazmuri,et al. Targeting mitochondria for resuscitation from cardiac arrest , 2008, Critical care medicine.
[6] S. Javadov,et al. Mitochondrial permeability transition pore opening during myocardial reperfusion--a target for cardioprotection. , 2004, Cardiovascular research.
[7] T. Walsh,et al. Clinical evaluation of a continuous oxygen consumption monitor in mechanically ventilated patients , 2003, Anaesthesia.
[8] M. Fink. Bench-to-bedside review: Cytopathic hypoxia , 2002, Critical care.
[9] T. Walsh,et al. Comparison between the Datex-Ohmeda M-COVX metabolic monitor and the Deltatrac II in mechanically ventilated patients , 2002, Intensive Care Medicine.
[10] M. Carraway,et al. Oxidative metabolism in rat hepatocytes and mitochondria during sepsis. , 1997, Archives of biochemistry and biophysics.
[11] M. Singer,et al. Oxygen tension in the bladder epithelium rises in both high and low cardiac output endotoxemic sepsis. , 1995, Journal of applied physiology.
[12] M. Fink,et al. Endotoxemia causes ileal mucosal acidosis in the absence of mucosal hypoxia in a normodynamic porcine model of septic shock. , 1995, Critical care medicine.
[13] C. Hinds,et al. Elevation of systemic oxygen delivery in the treatment of critically ill patients. , 1994, The New England journal of medicine.
[14] C. Hinds,et al. Clinical Investigations In Critical CareResponse of Critically III Patients to Treatment Aimed at Achieving Supranormal Oxygen Delivery and Consumption: Relationship to Outcome , 1993 .
[15] H. Smithline,et al. Venous hyperoxia after cardiac arrest. Characterization of a defect in systemic oxygen utilization. , 1992, Chest.
[16] M. Weil,et al. Early impairment of oxidative metabolism and energy production in severe sepsis. , 1988, Circulatory shock.
[17] E. Ravussin,et al. Respiratory quotients lower than 0.70 in ketogenic diets. , 1980, The American journal of clinical nutrition.
[18] C. Koch,et al. Cyanide insensitive respiration in mammalian cells: an artifact of mycoplasmal contamination. , 1983, Advances in experimental medicine and biology.