APOA2, dietary fat, and body mass index: replication of a gene-diet interaction in 3 independent populations.
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D. Corella | L. Cupples | G. Peloso | D. Arnett | J. Ordovás | K. Tucker | Yu-Chi Lee | L. Parnell | S. Demissie | Chao-Qiang Lai | Ó. Coltell
[1] S. Grundy,et al. Dietary fat consumption and health. , 2009, Nutrition reviews.
[2] K. Resch. [Dietary Intervention Randomized Controlled Trial (DIRECT) group: weight loss with a low-carbohydrate, Mediterranean, or low-fat diet]. , 2008, Forschende Komplementarmedizin.
[3] Steven Wiltshire,et al. The apolipoprotein AII rs5082 variant is associated with reduced risk of coronary artery disease in an Australian male population. , 2008, Atherosclerosis.
[4] M. Stampfer,et al. Weight loss with a low-carbohydrate, Mediterranean, or low-fat diet. , 2008, The New England journal of medicine.
[5] A. Lusis,et al. Apolipoprotein AII Is a Regulator of Very Low Density Lipoprotein Metabolism and Insulin Resistance* , 2008, Journal of Biological Chemistry.
[6] X. Adiconis,et al. PPARGC1A Variation Associated With DNA Damage, Diabetes, and Cardiovascular Diseases , 2008, Diabetes.
[7] Thomas A. Pearson,et al. How to interpret a genome-wide association study. , 2008, JAMA.
[8] Siobhan M. Dolan,et al. Assessment of cumulative evidence on genetic associations: interim guidelines. , 2008, International journal of epidemiology.
[9] O. Receveur,et al. Could the Quality of Dietary Fat, and Not Just Its Quantity, Be Related to Risk of Obesity? , 2008, Obesity.
[10] P. Donnelly,et al. Replicating genotype–phenotype associations , 2007, Nature.
[11] I. Borecki,et al. The -256T>C polymorphism in the apolipoprotein A-II gene promoter is associated with body mass index and food intake in the genetics of lipid lowering drugs and diet network study. , 2007, Clinical chemistry.
[12] H. Kraemer,et al. Comparison of the Atkins, Zone, Ornish, and LEARN diets for change in weight and related risk factors among overweight premenopausal women: the A TO Z Weight Loss Study: a randomized trial. , 2007, JAMA.
[13] A. Guttmacher,et al. Science & society: Educating health-care professionals about genetics and genomics , 2007, Nature Reviews Genetics.
[14] D. Corella,et al. APOA5 gene variation modulates the effects of dietary fat intake on body mass index and obesity risk in the Framingham Heart Study , 2007, Journal of Molecular Medicine.
[15] L. Afman,et al. Nutrigenomics: from molecular nutrition to prevention of disease. , 2006, Journal of the American Dietetic Association.
[16] Dolores Corella,et al. Integration of environment and disease into 'omics' analysis. , 2005, Current opinion in molecular therapeutics.
[17] M. Pencina,et al. Unique dietary patterns and chronic disease risk profiles of adult men: the Framingham nutrition studies. , 2005, Journal of the American Dietetic Association.
[18] D. Corella,et al. SINGLE NUCLEOTIDE POLYMORPHISMS THAT INFLUENCE LIPID METABOLISM: Interaction with Dietary Factors. , 2005, Annual review of nutrition.
[19] T. Rankinen,et al. Gene-diet interactions on body weight changes. , 2005, Journal of the American Dietetic Association.
[20] J. Lovejoy,et al. Apolipoprotein A-II polymorphism and visceral adiposity in African-American and white women. , 2005, Obesity research.
[21] P. Tso,et al. Apolipoprotein A-IV, food intake, and obesity , 2004, Physiology & Behavior.
[22] P. Wilson,et al. Genetic variation at the scavenger receptor class B type I gene locus determines plasma lipoprotein concentrations and particle size and interacts with type 2 diabetes: the framingham study. , 2003, The Journal of clinical endocrinology and metabolism.
[23] J. Chambaz,et al. Apolipoprotein A-II: beyond genetic associations with lipid disorders and insulin resistance , 2003, Current opinion in lipidology.
[24] M. Emi,et al. Interaction between the LDL-receptor gene bearing a novel mutation and a variant in the apolipoprotein A-II promoter: molecular study in a 1135-member familial hypercholesterolemia kindred , 2002, Journal of Human Genetics.
[25] D. Midthune,et al. Comparative validation of the Block, Willett, and National Cancer Institute food frequency questionnaires : the Eating at America's Table Study. , 2001, American journal of epidemiology.
[26] F. Blanco-Vaca,et al. Role of apoA-II in lipid metabolism and atherosclerosis: advances in the study of an enigmatic protein. , 2001, Journal of lipid research.
[27] N E Day,et al. Sample size determination for studies of gene-environment interaction. , 2001, International journal of epidemiology.
[28] A. Hamsten,et al. Human Evidence That the Apolipoprotein A-II Gene Is Implicated in Visceral Fat Accumulation and Metabolism of Triglyceride-Rich Lipoproteins , 2001, Circulation.
[29] A. Lusis,et al. Studies with apolipoprotein A-II transgenic mice indicate a role for HDLs in adiposity and insulin resistance. , 2001, Diabetes.
[30] Hannes Oberkofler,et al. Leptin, Peroxisome Proliferator-Activated Receptor-γ, and CCAAT/Enhancer Binding Protein-α mRNA Expression in Adipose Tissue of Humans and Their Relation to Cardiovascular Risk Factors , 2000 .
[31] R. Paffenbarger,et al. Physical activity and stroke incidence: the Harvard Alumni Health Study. , 1998, Stroke.
[32] K. Tucker,et al. Adaptation of a food frequency questionnaire to assess diets of Puerto Rican and non-Hispanic adults. , 1998, American journal of epidemiology.
[33] J. Ordóñez‐Llanos,et al. Human apolipoprotein A-II is a pro-atherogenic molecule when it is expressed in transgenic mice at a level similar to that in humans: evidence of a potentially relevant species-specific interaction with diet. , 1998, Journal of lipid research.
[34] P. Wolf,et al. Physical activity and stroke risk: the Framingham Study. , 1994, American journal of epidemiology.
[35] G A Colditz,et al. Reproducibility and validity of an expanded self-administered semiquantitative food frequency questionnaire among male health professionals. , 1992, American journal of epidemiology.
[36] W. Kannel,et al. The Framingham Offspring Study. Design and preliminary data. , 1975, Preventive medicine.
[37] E. Burchard,et al. Population admixture associated with disease prevalence in the Boston Puerto Rican health study , 2008, Human Genetics.
[38] M. Jarvelin,et al. References and Notes Supporting Online Material Materials and Methods Som Text Figs. S1 to S7 References a Common Variant in the Fto Gene Is Associated with Body Mass Index and Predisposes to Childhood and Adult Obesity , 2022 .