Increased adhesive properties of eosinophils in sickle cell disease.

[1]  D. Granger,et al.  Endothelial cell P-selectin mediates a proinflammatory and prothrombogenic phenotype in cerebral venules of sickle cell transgenic mice. , 2004, American journal of physiology. Heart and circulatory physiology.

[2]  B. Coller,et al.  Intravenous immune globulin prevents venular vaso-occlusion in sickle cell mice by inhibiting leukocyte adhesion and the interactions between sickle erythrocytes and adherent leukocytes. , 2004, Blood.

[3]  G. Vercellotti,et al.  Anti-inflammatory therapy ameliorates leukocyte adhesion and microvascular flow abnormalities in transgenic sickle mice. , 2004, American journal of physiology. Heart and circulatory physiology.

[4]  R. Hebbel,et al.  Sickle blood contains tissue factor-positive microparticles derived from endothelial cells and monocytes. , 2003, Blood.

[5]  P. Dri,et al.  Evidence that TNF‐induced respiratory burst of adherent PMN is mediated by integrin αLβ2 , 2002, Journal of Leukocyte Biology.

[6]  J. Goldman,et al.  Relationship between the clinical manifestations of sickle cell disease and the expression of adhesion molecules on white blood cells , 2002, European journal of haematology.

[7]  N. Hogg,et al.  Mechanisms contributing to the activity of integrins on leukocytes , 2002, Immunological reviews.

[8]  J. Hamilton,et al.  GM-CSF in inflammation and autoimmunity. , 2002, Trends in immunology.

[9]  J. Zangrilli.  Regulation of eosinophil viability by cytokines. , 2002, American journal of respiratory cell and molecular biology.

[10]  B. Coller,et al.  Primary role for adherent leukocytes in sickle cell vascular occlusion: A new paradigm , 2002, Proceedings of the National Academy of Sciences of the United States of America.

[11]  M. Gonçalves,et al.  Interleukin 8 as a vaso-occlusive marker in Brazilian patients with sickle cell disease. , 2001, Brazilian journal of medical and biological research = Revista brasileira de pesquisas medicas e biologicas.

[12]  J. Eckman,et al.  Thrombogenesis in sickle cell disease. , 2001, The Journal of laboratory and clinical medicine.

[13]  J. Lammers,et al.  IL-8 Induces a Transient Arrest of Rolling Eosinophils on Human Endothelial Cells1 , 2001, The Journal of Immunology.

[14]  G. Vercellotti,et al.  Activated monocytes in sickle cell disease: potential role in the activation of vascular endothelium and vaso-occlusion. , 2000, Blood.

[15]  W. Busse,et al.  The effect of transendothelial migration on eosinophil function. , 2000, American journal of respiratory cell and molecular biology.

[16]  Scott T. Miller,et al.  Prediction of adverse outcomes in children with sickle cell disease. , 2000, The New England journal of medicine.

[17]  Rochon,et al.  Analysis of integrin (CD11b/CD18) movement during neutrophil adhesion and migration on endothelial cells , 2000, Journal of microscopy.

[18]  F. Graziano,et al.  Cytokines, chemokines, RANTES, and eotaxin. , 1999, Allergy and asthma proceedings.

[19]  E. Wayner,et al.  Circulating activated endothelial cells in sickle cell anemia. , 1997, The New England journal of medicine.

[20]  S. Taylor,et al.  Type 2 cytokine serum levels in healthy sickle cell disease patients. , 1997, Journal of the National Medical Association.

[21]  H. Bunn.  Pathogenesis and treatment of sickle cell disease. , 1997, The New England journal of medicine.

[22]  K. Sung,et al.  Granulocyte-macrophage colony-stimulating factor regulates the functional adhesive state of very late antigen-4 expressed by eosinophils. , 1997, Journal of immunology.

[23]  A. Duits,et al.  Enhanced levels of soluble VCAM-1 in sickle cell patients and their specific increment during vasoocclusive crisis. , 1996, Clinical immunology and immunopathology.

[24]  E. Plow,et al.  Overlapping, but Not Identical, Sites Are Involved in the Recognition of C3bi, Neutrophil Inhibitory Factor, and Adhesive Ligands by the αMβ2 Integrin* , 1996, The Journal of Biological Chemistry.

[25]  F. Redegeld,et al.  LFA-1, and not Mac-1, is crucial for the development of hyperreactivity in a murine model of nonallergic asthma. , 1996, American journal of respiratory and critical care medicine.

[26]  D. Golan,et al.  Adhesion of sickle neutrophils and erythrocytes to fibronectin. , 1996, Blood.

[27]  W. Busse,et al.  Eosinophil adhesion to vascular cell adhesion molecule-1 activates superoxide anion generation. , 1995, Journal of immunology.

[28]  P. Brandtzaeg,et al.  Eosinophil infiltration is related to increased expression of vascular cell adhesion molecule-1 in nasal polyps. , 1995, American journal of respiratory cell and molecular biology.

[29]  A. Nakao,et al.  Induction of T cell tolerance by pretreatment with anti-ICAM-1 and anti-lymphocyte function-associated antigen-1 antibodies prevents antigen-induced eosinophil recruitment into the mouse airways. , 1994, Journal of immunology.

[30]  G. Rodgers,et al.  Mortality in sickle cell disease. , 1994 .

[31]  T. Carlos,et al.  Leukocyte-endothelial adhesion molecules. , 1994, Blood.

[32]  H. Croizat.  Circulating cytokines in sickle cell patients during steady state , 1994, British journal of haematology.

[33]  O. Platt,et al.  Mortality in sickle cell disease. Life expectancy and risk factors for early death. , 1994, The New England journal of medicine.

[34]  H. Kita,et al.  CD11b/CD18 (Mac-1) is required for degranulation of human eosinophils induced by human recombinant granulocyte-macrophage colony-stimulating factor and platelet-activating factor. , 1994, Journal of immunology.

[35]  E. Wayner,et al.  Integrin alpha 4 beta 1 and glycoprotein IV (CD36) are expressed on circulating reticulocytes in sickle cell anemia , 1993 .

[36]  A. Kumar,et al.  Alpha 4 beta 1-integrin expression on sickle reticulocytes: vascular cell adhesion molecule-1-dependent binding to endothelium , 1993 .

[37]  K. Blaser,et al.  An improved immunomagnetic procedure for the isolation of highly purified human blood eosinophils. , 1991, Journal of immunological methods.

[38]  M. Arnaout,et al.  Structure and function of the leukocyte adhesion molecules CD11/CD18 , 1990 .

[39]  S. Davies,et al.  The presentation, management and prophylaxis of sickle cell disease. , 1989, Blood reviews.

[40]  D. Boggs,et al.  Search for eosinopenia in hospitalized patients with normal blood leukocyte concentration , 1987, American journal of hematology.

[41]  Graham R Serjeant,et al.  Sickle-cell disease , 1984, The Lancet.

[42]  J. Innes.  Examination of the blood. , 1949, The Practitioner.

[43]  T. Pearson,et al.  Blood polymorphonuclear leukocytes from the majority of sickle cell patients in the crisis phase of the disease show enhanced adhesion to vascular endothelium and increased expression of CD64. , 1998, Blood.

[44]  J. J. Schwartz,et al.  Perspectives series: cell adhesion in vascular biology. Adhesive interactions of sickle erythrocytes with endothelium. , 1997, The Journal of clinical investigation.

[45]  T. Fukuda,et al.  Eosinophils and allergy in asthma. , 1995, Allergy proceedings : the official journal of regional and state allergy societies.

[46]  T. Arends,et al.  Levels of Tumor Necrosis Factor α/Cachectin (TNFα) in Sera from Patients with Sickle Cell Disease , 1993 .

[47]  Ekeke Gi.  Sickle cell disease: some haematological changes in steady state and crisis. , 1987 .

[48]  C. Srodes,et al.  An unusual pattern of neutrophil kinetics in sickle cell anemia. , 1973, Blood.