Different repair of O6-methylguanine occurring in DNA modified by MMS in vivo or in vitro.
暂无分享,去创建一个
[1] M. Dolan,et al. Increased killing of prostate, breast, colon, and lung tumor cells by the combination of inactivators of O6-alkylguanine-DNA alkyltransferase and N,N'-bis(2-chloroethyl)-N-nitrosourea. , 1995, Biochemical pharmacology.
[2] E. Sledziewska-Gojska. Inactivation of O6-methylguanine-DNA methyltransferase in vivo by SN2 alkylating agents. , 1995, Mutation research.
[3] S. Hughes,et al. Response of repair-competent and repair-deficient Escherichia coli to three O6-substituted guanines and involvement of methyl-directed mismatch repair in the processing of O6-methylguanine residues. , 1994, Biochemistry.
[4] E. Sledziewska-Gojska. The level of GC-->AT transitions induced by MMS is not affected by the adaptive response in Escherichia coli K12. , 1993, Mutation research.
[5] E. Winer,et al. Modulation of O6-alkylguanine-DNA alkyltransferase-mediated carmustine resistance using streptozotocin: a phase I trial. , 1992, Cancer research.
[6] L. Samson. The suicidal DNA repalr methyltransferases of microbes , 1992, Molecular microbiology.
[7] E. Grzesiuk,et al. Mutagenesis of Escherichia coli: a method for determining mutagenic specificity by analysis of tRNA suppressors. , 1992, Mutagenesis.
[8] M. Dolan,et al. Depletion of mammalian O6-alkylguanine-DNA alkyltransferase activity by O6-benzylguanine provides a means to evaluate the role of this protein in protection against carcinogenic and therapeutic alkylating agents. , 1990, Proceedings of the National Academy of Sciences of the United States of America.
[9] S. Gerson. Modulation of human lymphocyte O6-alkylguanine-DNA alkyltransferase by streptozotocin in vivo. , 1989, Cancer research.
[10] M. Dolan,et al. Cell specific differences in O6-methylguanine-DNA methyltransferase activity and removal of O6-methylguanine in rat pulmonary cells. , 1988, Carcinogenesis.
[11] K. Sakumi,et al. Activation of Ada protein as a transcriptional regulator by direct alkylation with methylating agents. , 1988, The Journal of biological chemistry.
[12] A. A. Zeeland. Molecular dosimetry of alkylating agents: quantitative comparison of genetic effects on the basis of DNA adduct formation. , 1988 .
[13] Y. Nakabeppu,et al. Regulation and expression of the adaptive response to alkylating agents. , 1988, Annual review of biochemistry.
[14] B W Glickman,et al. Influence of neighbouring base sequence on N-methyl-N'-nitro-N-nitrosoguanidine mutagenesis in the lacI gene of Escherichia coli. , 1987, Journal of molecular biology.
[15] D. Scudiero,et al. The Role of O6-Methylguanine in Human Cell Killing, Sister Chromatid Exchange Induction and Mutagenesis: A Review , 1987, Journal of Cell Science.
[16] N. Berger,et al. Comparison of O6-alkylguanine-DNA alkyltransferase activity based on cellular DNA content in human, rat and mouse tissues. , 1986, Carcinogenesis.
[17] T. Brent. Inactivation of purified human O6-alkylguanine-DNA alkyltransferase by alkylating agents or alkylated DNA. , 1986, Cancer research.
[18] R. Chambers,et al. uvrA and recA mutations inhibit a site-specific transition produced by a single O6-methylguanine in gene G of bacteriophage phi X174. , 1985, Proceedings of the National Academy of Sciences of the United States of America.
[19] S. Williams,et al. The cytotoxic and mutagenic effects of alkylating agents on human lymphoid cells are caused by different DNA lesions. , 1985, Carcinogenesis.
[20] J. Essigmann,et al. In vivo mutagenesis by O6-methylguanine built into a unique site in a viral genome. , 1984, Proceedings of the National Academy of Sciences of the United States of America.
[21] L. Erickson,et al. Pretreatment of human colon tumor cells with DNA methylating agents inhibits their ability to repair chloroethyl monoadducts. , 1984, Carcinogenesis.
[22] D. Grunberger,et al. Molecular Biology of Mutagens and Carcinogens , 1983, Springer US.
[23] P. Karran,et al. Death of an enzyme: suicide repair of DNA , 1983 .
[24] J. Messing. [2] New M13 vectors for cloning , 1983 .
[25] T. Lindahl,et al. Suicide inactivation of the E. coli O6‐methylguanine‐DNA methyltransferase. , 1982, The EMBO journal.
[26] J. Swenberg,et al. Cell-specific differences in O6-alkylguanine DNA repair activity during continuous exposure to carcinogen. , 1982, Proceedings of the National Academy of Sciences of the United States of America.
[27] B. Singer. Mutagenesis from a chemical perspective: nucleic acid reactions, repair, translation, and transcription. , 1982, Basic life sciences.
[28] J. Cairns. Efficiency of the adaptive response of Escherichia coli to alkylating agents , 1980, Nature.
[29] J. Miller,et al. Genetic studies of the lac repressor. IV. Mutagenic specificity in the lacI gene of Escherichia coli. , 1977, Journal of molecular biology.
[30] P. D. Lawley. Some chemical aspects of dose-response relationships in alkylation mutagenesis. , 1974, Mutation research.
[31] Jeffrey H. Miller. Experiments in molecular genetics , 1972 .
[32] P. D. Lawley,et al. Methylation of deoxyribonucleic acid in cultured mammalian cells by N-methyl-N'-nitro-N-nitrosoguanidine. The influence of cellular thiol concentrations on the extent of methylation and the 6-oxygen atom of guanine as a site of methylation. , 1970, The Biochemical journal.