Mitochondrial respiratory chain activity in idiopathic dilated cardiomyopathy.

[1]  A. Khaghani,et al.  Energetics and function of the failing human heart with dilated or hypertrophic cardiomyopathy , 1999, European journal of clinical investigation.

[2]  R. Kapsa,et al.  The polymerase chain reaction in the study of mitochondrial genetics. , 1997, Journal of biochemical and biophysical methods.

[3]  H. Mizusawa,et al.  Histological determination of nitric oxide synthase (NOS) and NADPH-diaphorase in ragged-red fibers from patients with mitochondrial encephalomyopathies , 1997, Journal of the Neurological Sciences.

[4]  D. Wallace,et al.  A mouse model for mitochondrial myopathy and cardiomyopathy resulting from a deficiency in the heart/muscle isoform of the adenine nucleotide translocator , 1997, Nature Genetics.

[5]  J. Shoffner,et al.  Maternal inheritance and the evaluation of oxidative phosphorylation diseases , 1996, The Lancet.

[6]  S. Dimauro,et al.  Multiple mitochondria1 DNA deletions associated with autosomal recessive ophthalmoplegia and severe cardiomyopathy , 1996, Neurology.

[7]  P. Tsao,et al.  Expression of inducible nitric oxide synthase in human heart failure. , 1996, Circulation.

[8]  C. Epstein,et al.  Dilated cardiomyopathy and neonatal lethality in mutant mice lacking manganese superoxide dismutase , 1995, Nature Genetics.

[9]  N. Yanagisawa,et al.  Multiple mitochondrial DNA deletions in a patient with mitochondrial myopathy and cardiomyopathy but no ophthalmoplegia , 1995, Muscle & nerve.

[10]  J. Marín-García,et al.  Impaired mitochondrial function in idiopathic dilated cardiomyopathy: biochemical and molecular analysis. , 1995, Journal of cardiac failure.

[11]  M. Hayakawa,et al.  Genotype and phenotype of severe mitochondrial cardiomyopathy: a recipient of heart transplantation and the genetic control. , 1995, Biochemical and biophysical research communications.

[12]  M. Miyata,et al.  Cardiac involvement in mitochondrial diseases. A study on 17 patients with documented mitochondrial DNA defects. , 1995, Circulation.

[13]  P. Macdonald,et al.  Increased nitric oxide production in heart failure , 1994, The Lancet.

[14]  S. Moncada,et al.  Reversible inhibition of cytochrome c oxidase, the terminal enzyme of the mitochondrial respiratory chain, by nitric oxide , 1994, FEBS letters.

[15]  S. Dimauro,et al.  A new mtDNA mutation in the tRNALeu(UUR) gene associated with maternally inherited cardiomyopathy , 1994, Human mutation.

[16]  S. Zierz,et al.  Myocardial respiratory chain enzyme activities in idiopathic dilated cardiomyopathy, and comparison with those in atherosclerotic coronary artery disease and valvular aortic stenosis. , 1993, The American journal of cardiology.

[17]  W. Abelmann,et al.  Prevalence and etiology of idiopathic dilated cardiomyopathy (summary of a National Heart, Lung, and Blood Institute workshop. , 1992, The American journal of cardiology.

[18]  D. Thompson.  Textbook and Colour Atlas of the Cardiovascular System , 1992 .

[19]  V. Tiranti,et al.  Maternally inherited myopathy and cardiomyopathy: association with mutation in mitochondrial DNA tRNALeu(UUR) , 1991, The Lancet.

[20]  A. Friedl,et al.  Impairment of the myocardial ultrastructure and changes of the cytoskeleton in dilated cardiomyopathy. , 1991, Circulation.

[21]  H. Figulla,et al.  Enzyme histochemistry of endomyocardial biopsies in idiopathic dilated cardiomyopathy. , 1991, Cardiology.

[22]  D. Wallace,et al.  Evaluation of procedures for assaying oxidative phosphorylation enzyme activities in mitochondrial myopathy muscle biopsies. , 1990, Biochimica et biophysica acta.

[23]  H. Figulla,et al.  Alterations of the mitochondrial respiratory chain in human dilated cardiomyopathy. , 1990, European heart journal.

[24]  M. Billingham,et al.  Ultrastructural features of familial cardiomyopathy. , 1988, The American journal of cardiology.

[25]  T. Bashore,et al.  Histologic and biochemical correlates of left ventricular chamber dynamics in man. , 1987, Journal of the American College of Cardiology.

[26]  M. Wilson,et al.  Mitochondria: a practical approach. , 1987 .

[27]  D. Wilson,et al.  Energy relationships between cytosolic metabolism and mitochondrial respiration in rat heart. , 1978, The American journal of physiology.

[28]  M. Entman,et al.  Abnormal biochemistry in myocardial failure. , 1973, The American journal of cardiology.

[29]  A. Schwartz,et al.  Some biochemical studies on subcellular systems isolated from fresh recipient human cardiac tissue obtained during transplantation. , 1971, The American journal of cardiology.

[30]  L. Sordahl,et al.  Reevaluation of Oxidative Phosphorylation in Cardiac Mitochondria from Normal Animals and Animals in Heart Failure , 1968, Circulation research.

[31]  E. Braunwald,et al.  Normal Oxidative Phosphorylation in Mitochondria from the Failing Heart , 1967 .

[32]  C. Chidsey,et al.  Biochemical studies of energy production in the failing human heart. , 1966, The Journal of clinical investigation.

[33]  M. Feinstein,et al.  Effects of Experimental Congestive Heart Failure, Ouabain, and Asphyxia on the High‐Energy Phosphate and Creatine Content of the Guinea Pig Heart , 1962, Circulation research.

[34]  K. S. Lee,et al.  Study of Heart Mitochondria and Glycolytic Metabolism in Experimentally Induced Cardiac Failure , 1962, Circulation research.

[35]  E. Rideal,et al.  On the action of plant growth regulators. , 1952, Biochimica et biophysica acta.

[36]  O. H. Lowry,et al.  Protein measurement with the Folin phenol reagent. , 1951, The Journal of biological chemistry.