Pirt, a Phosphoinositide-Binding Protein, Functions as a Regulatory Subunit of TRPV1
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[1] B. Nilius,et al. Modulation of TRPs by PIPs , 2007, The Journal of physiology.
[2] P. Várnai,et al. Dual Regulation of TRPV1 by Phosphoinositides , 2007, The Journal of Neuroscience.
[3] A. Patapoutian,et al. TRPM8 Is Required for Cold Sensation in Mice , 2007, Neuron.
[4] M. D'Andrea,et al. Attenuated Cold Sensitivity in TRPM8 Null Mice , 2007, Neuron.
[5] M. Schaefer,et al. TRPA1 Is Differentially Modulated by the Amphipathic Molecules Trinitrophenol and Chlorpromazine* , 2007, Journal of Biological Chemistry.
[6] C. Montell,et al. Integration of phosphoinositide- and calmodulin-mediated regulation of TRPC6. , 2007, Molecular cell.
[7] R. Hardie. TRP channels and lipids: from Drosophila to mammalian physiology , 2007, The Journal of physiology.
[8] P. McNaughton,et al. Modulation of temperature-sensitive TRP channels. , 2006, Seminars in cell & developmental biology.
[9] A. Stein,et al. Phosphoinositide 3-Kinase Binds to TRPV1 and Mediates NGF-stimulated TRPV1 Trafficking to the Plasma Membrane , 2006, The Journal of general physiology.
[10] Clifford J. Woolf,et al. TRPA1 Contributes to Cold, Mechanical, and Chemical Nociception but Is Not Essential for Hair-Cell Transduction , 2006, Neuron.
[11] David Julius,et al. TRPA1 Mediates the Inflammatory Actions of Environmental Irritants and Proalgesic Agents , 2006, Cell.
[12] Beiying Liu,et al. Functional Recovery from Desensitization of Vanilloid Receptor TRPV1 Requires Resynthesis of Phosphatidylinositol 4,5-Bisphosphate , 2005, The Journal of Neuroscience.
[13] I. Michailidis,et al. PI(4,5)P2 regulates the activation and desensitization of TRPM8 channels through the TRP domain , 2005, Nature Neuroscience.
[14] Craig Montell,et al. The TRP Superfamily of Cation Channels , 2005, Science's STKE.
[15] D. Julius,et al. A Modular PIP2 Binding Site as a Determinant of Capsaicin Receptor Sensitivity , 2003, Science.
[16] Makoto Tominaga,et al. Heat-Evoked Activation of the Ion Channel, TRPV4 , 2002, The Journal of Neuroscience.
[17] David J. Anderson,et al. Comparison of the generic neuronal differentiation and neuron subtype specification functions of mammalian achaete-scute and atonal homologs in cultured neural progenitor cells. , 2002, Development.
[18] David J. Anderson,et al. A Diverse Family of GPCRs Expressed in Specific Subsets of Nociceptive Sensory Neurons , 2001, Cell.
[19] A. Basbaum,et al. Bradykinin and nerve growth factor release the capsaicin receptor from PtdIns(4,5)P2-mediated inhibition , 2001, Nature.
[20] S. McLaughlin,et al. The Effector Domain of Myristoylated Alanine-rich C Kinase Substrate Binds Strongly to Phosphatidylinositol 4,5-Bisphosphate* , 2001, The Journal of Biological Chemistry.
[21] S. Bingham,et al. Vanilloid receptor-1 is essential for inflammatory thermal hyperalgesia , 2000, Nature.
[22] A I Basbaum,et al. Impaired nociception and pain sensation in mice lacking the capsaicin receptor. , 2000, Science.
[23] D. Julius,et al. Sense and specificity: a molecular identity for nociceptors , 1999, Current Opinion in Neurobiology.
[24] A. Basbaum,et al. The Cloned Capsaicin Receptor Integrates Multiple Pain-Producing Stimuli , 1998, Neuron.
[25] R. Tsai,et al. Using a eukaryotic GST fusion vector for proteins difficult to express in E. coli. , 1997, BioTechniques.
[26] D. Julius,et al. The capsaicin receptor: a heat-activated ion channel in the pain pathway , 1997, Nature.
[27] Timothy J. Brennan,et al. Characterization of a rat model of incisional pain , 1996, Pain.
[28] S. Bevan,et al. Nerve growth factor (NGF) differentially regulates the chemosensitivity of adult rat cultured sensory neurons , 1995, The Journal of neuroscience : the official journal of the Society for Neuroscience.
[29] T. Yaksh,et al. Quantitative assessment of tactile allodynia in the rat paw , 1994, Journal of Neuroscience Methods.
[30] C. Hodge. Sensory Neurons: Diversity, Development, and Plasticity. , 1994 .
[31] S. Hunskaar,et al. The formalin test: an evaluation of the method , 1992, Pain.