Molecular pathway for (-)-epigallocatechin-3-gallate-induced cell cycle arrest and apoptosis of human prostate carcinoma cells.

[1]  D. Malkin.  The Role of P53 in Human Cancer , 2001, Journal of Neuro-Oncology.

[2]  Massimo Loda,et al.  Role of p27 in Prostate Carcinogenesis , 2004, Cancer and Metastasis Reviews.

[3]  Christine E. Canman,et al.  P53, cell cycle control and apoptosis: Implications for cancer , 1995, Cancer and Metastasis Reviews.

[4]  G. Giaccone,et al.  Apoptosis: target of cancer therapy. , 2002, Clinical cancer research : an official journal of the American Association for Cancer Research.

[5]  Luciana Dini,et al.  Cell death: apoptosis versus necrosis (review). , 2002, International journal of oncology.

[6]  M. Barbacid,et al.  Cyclin D-dependent kinases, INK4 inhibitors and cancer. , 2002, Biochimica et biophysica acta.

[7]  S. De Flora,et al.  ‘Angioprevention’: angiogenesis is a common and key target for cancer chemopreventive agents , 2002, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

[8]  S. Gupta,et al.  Green tea constituent (--)-epigallocatechin-3-gallate inhibits topoisomerase I activity in human colon carcinoma cells. , 2001, Biochemical and biophysical research communications.

[9]  D. Fisher.  Pathways of apoptosis and the modulation of cell death in cancer. , 2001, Hematology/oncology clinics of North America.

[10]  T. Owa,et al.  Cell cycle regulation in the G1 phase: a promising target for the development of new chemotherapeutic anticancer agents. , 2001, Current medicinal chemistry.

[11]  Z. Dong,et al.  Mechanisms of inhibition of the Ras‐MAP kinase signaling pathway in 30.7b Ras 12 cells by tea polyphenols (‐)‐epigallocatechin‐3‐gallate and theaflavin‐3,3′‐digallate1 , 2001, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

[12]  J. Lewin,et al.  Inhibition of prostate carcinogenesis in TRAMP mice by oral infusion of green tea polyphenols , 2001, Proceedings of the National Academy of Sciences of the United States of America.

[13]  D. Heber,et al.  Green Tea and Its Catechins Inhibit Breast Cancer Xenografts , 2001, Nutrition and cancer.

[14]  J. Pietenpol,et al.  p53 Signaling and cell cycle checkpoints. , 2001, Chemical research in toxicology.

[15]  Steven J M Jones,et al.  Connecting signaling and cell cycle progression in growth factor-stimulated cells , 2000, Oncogene.

[16]  M. Molinari.  Cell cycle checkpoints and their inactivation in human cancer , 2000, Cell proliferation.

[17]  K. Kiura,et al.  (–)-Epigallocatechin gallate can prevent cisplatin-induced lung tumorigenesis in A/J mice , 2000 .

[18]  W. El-Deiry,et al.  Cell cycle control as a basis for cancer drug development (Review). , 2000, International journal of oncology.

[19]  S. Gupta,et al.  Green tea polyphenol epigallocatechin-3-gallate differentially modulates nuclear factor kappaB in cancer cells versus normal cells. , 2000, Archives of biochemistry and biophysics.

[20]  S. Gupta,et al.  Growth inhibition, cell-cycle dysregulation, and induction of apoptosis by green tea constituent (-)-epigallocatechin-3-gallate in androgen-sensitive and androgen-insensitive human prostate carcinoma cells. , 2000, Toxicology and applied pharmacology.

[21]  J. Slingerland,et al.  Deregulation of the cell cycle in cancer. , 2000, Cancer detection and prevention.

[22]  N. Pavletich.  Mechanisms of cyclin-dependent kinase regulation: structures of Cdks, their cyclin activators, and Cip and INK4 inhibitors. , 1999, Journal of molecular biology.

[23]  J. Cheville,et al.  p27kip1: a multifunctional cyclin-dependent kinase inhibitor with prognostic significance in human cancers. , 1999, The American journal of pathology.

[24]  F. Kadlubar,et al.  Chemopreventive effects of tea extracts and various components on human pancreatic and prostate tumor cells in vitro. , 1999, Nutrition and cancer.

[25]  M. Suganuma,et al.  Growth inhibition of leukemic cells by (-)-epigallocatechin gallate, the main constituent of green tea. , 1998, Life sciences.

[26]  A. Khafif,et al.  Green tea regulates cell cycle progression in oral leukoplakia , 1998, Head & neck.

[27]  C. Young,et al.  Induction of apoptosis in prostate cancer cell lines by the green tea component, (-)-epigallocatechin-3-gallate. , 1998, Cancer letters.

[28]  Chi-Tang Ho,et al.  Green tea epigallocatechin gallate shows a pronounced growth inhibitory effect on cancerous cells but not on their normal counterparts. , 1998, Cancer letters.

[29]  G. Yang,et al.  Inhibition of growth and induction of apoptosis in human cancer cell lines by tea polyphenols. , 1998, Carcinogenesis.

[30]  H. Hibasami,et al.  Induction of apoptosis in human stomach cancer cells by green tea catechins. , 1998, Oncology reports.

[31]  M L Agarwal,et al.  The p53 Network* , 1998, The Journal of Biological Chemistry.

[32]  V. Steele,et al.  Effect of Tea Extracts, Polyphenols, and Epigallocatechin Gallate on Azoxymethane-induced Colon Cancer , 1998, Proceedings of the Society for Experimental Biology and Medicine. Society for Experimental Biology and Medicine.

[33]  R. Agarwal,et al.  Green tea constituent epigallocatechin-3-gallate and induction of apoptosis and cell cycle arrest in human carcinoma cells. , 1997, Journal of the National Cancer Institute.

[34]  Weiya Ma,et al.  Inhibition of tumor promoter-induced activator protein 1 activation and cell transformation by tea polyphenols, (-)-epigallocatechin gallate, and theaflavins. , 1997, Cancer research.

[35]  A. Porter,et al.  Target to apoptosis: A hopeful weapon for prostate cancer , 1997, The Prostate.

[36]  R. Mohan,et al.  Protection against induction of mouse skin papillomas with low and high risk of conversion to malignancy by green tea polyphenols. , 1997, Carcinogenesis.

[37]  M. Hirose,et al.  Effects of green tea catechins on the progression or late promotion stage of mammary gland carcinogenesis in female Sprague-Dawley rats pretreated with 7,12-dimethylbenz(a)anthracene. , 1997, Cancer letters.

[38]  J. Bartek,et al.  Aberrations of the G1- and G1/S-regulating genes in human cancer. , 1997, Progress in cell cycle research.

[39]  C. Sherr.  Cancer Cell Cycles , 1996, Science.

[40]  A. Gartel,et al.  p21—Negative Regulator of the Cell Cycle , 1996, Proceedings of the Society for Experimental Biology and Medicine. Society for Experimental Biology and Medicine.

[41]  R. Hiipakka,et al.  Growth inhibition and regression of human prostate and breast tumors in athymic mice by tea epigallocatechin gallate. , 1995, Cancer letters.

[42]  J. Bartek,et al.  Cyclin D1 is dispensable for G1 control in retinoblastoma gene-deficient cells independently of cdk4 activity , 1995, Molecular and cellular biology.

[43]  R. Müller,et al.  Cell cycle-regulated transcription in mammalian cells. , 1995, Progress in cell cycle research.