Neonatal-6-hydroxydopamine treatment: Model of susceptibility for self-mutilation in the lesch-nyhan syndrome
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[1] K. Shannak,et al. Biochemical evidence of dysfunction of brain neurotransmitters in the Lesch-Nyhan syndrome. , 1981, The New England journal of medicine.
[2] R. Watts,et al. Clinical and biochemical studies on treatment of Lesch-Nyhan syndrome , 1974, Archives of disease in childhood.
[3] B. Cooper,et al. BIOCHEMICAL AND BEHAVIOURAL ALTERATIONS FOLLOWING 6-HYDROXYDOPAMINE ADMINISTRATION INTO BRAIN. , 1974, Biochemical pharmacology.
[4] B. Cooper,et al. Growth and behavioral changes in developing rats treated intracisternally with 6-hydroxydopamine: evidence for involvement of brain dopamine. , 1973, The Journal of pharmacology and experimental therapeutics.
[5] B. Cooper,et al. Alterations in consummatory behavior following intracisternal injection of 6-hydroxydopamine. , 1973, Pharmacology, biochemistry, and behavior.
[6] O. Hornykiewicz. Parkinson's disease: from brain homogenate to treatment. , 1973, Federation proceedings.
[7] G. Breese,et al. Developmental characteristics of brain catecholamines and tyrosine hydroxylase in the rat: effects of 6‐hydroxydopamine , 1972, British journal of pharmacology.
[8] G. Breese,et al. Effect of 6-hydroxydopamine on brain norepinephrine and dopamine evidence for selective degeneration of catecholamine neurons. , 1970, The Journal of pharmacology and experimental therapeutics.
[9] J. Seegmiller,et al. Enzyme Defect Associated with a Sex-Linked Human Neurological Disorder and Excessive Purine Synthesis , 1967, Science.
[10] W. Nyhan,et al. A FAMILIAL DISORDER OF URIC ACID METABOLISM AND CENTRAL NERVOUS SYSTEM FUNCTION. , 1964, The American journal of medicine.